V-Maf Musculoaponeurotic Fibrosarcoma Oncogene Family, Protein K (Avian) (MAFK) ELISA Kits

Since they lack a putative transactivation domain, the small Mafs behave as transcriptional repressors when they dimerize among themselves. Additionally we are shipping MAFK Antibodies (38) and MAFK Proteins (6) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
MAFK 7975 O60675
MAFK 17135 Q61827
MAFK    
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Top MAFK ELISA Kits at antibodies-online.com

Showing 3 out of 5 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Delivery Price Details
Human < 0.188 ng/mL 0.313 ng/mL - 20 ng/mL   96 Tests 11 to 18 Days
$838.60
Details
Mouse < 0.094 ng/mL 0.156 ng/mL - 10 ng/mL   96 Tests 11 to 18 Days
$838.60
Details
Chicken
  96 Tests 15 to 18 Days
$1,095.60
Details

More ELISA Kits for MAFK Interaction Partners

Human V-Maf Musculoaponeurotic Fibrosarcoma Oncogene Family, Protein K (Avian) (MAFK) interaction partners

  1. MAFK genetic polymorphisms are significantly associated with susceptibility to ulcerative colitis development in Japan.

  2. Findings suggest that MAFK and its target gene GPNMB play important roles in the malignant progression of triple-negative breast cancer (TNBC) cells, offering potentially new therapeutic targets for TNBC patients.

  3. Results suggested that Wnt1-induced MAFK expression promoted cell proliferation in MG63 cells, and that the role of MAFK in osteosarcoma might be closely linked to the Wnt signaling pathway.

  4. MafK mediated p65 acetylation by CBP upon LPS stimulation, thereby facilitating recruitment of p65 to NF-kappaB promoters such as IL-8 and TNFalpha.

  5. Results suggest that JDP2 is an integral component of the Nrf2-MafK complex and that it modulates antioxidant and detoxification programs by acting via the ARE.

  6. Data indicate that transcription factors MafK and Bach1 regulate expression of heme oxygenase-1 (HO-1).

  7. These findings demonstrate that a combination of p45NF-E2, Maf G, and Maf K is a key determinant of both megakaryopoiesis and thrombopoiesis.

Mouse (Murine) V-Maf Musculoaponeurotic Fibrosarcoma Oncogene Family, Protein K (Avian) (MAFK) interaction partners

  1. beta-Cell-Specific Mafk Overexpression Impairs Pancreatic Endocrine Cell Development

  2. These data identify Mafg and Mafk as new cataract-associated candidates and define their function in regulating largely non-crystallin genes linked to human cataract.

  3. KAP-1 may contribute to the repression of Ey and beta-major globin gene transcription through recruitment to the promoters of these two genes, mediated by the interaction of KAP-1 with either Zfp445 or MafK, respectively

  4. These findings demonstrate that a combination of p45NF-E2, Maf G, and Maf K is a key determinant of both megakaryopoiesis and thrombopoiesis.

  5. MafF, MafG, and MafK triple-knockout mice developed normally until embryonic day 9.5 (E9.5). Thereafter, however, the triple-knockout embryos showed severe growth retardation and liver hypoplasia, and the embryos died around E13.5.

  6. Thus compound mafG::mafK mutants develop age- and maf gene dosage-dependent cell-autonomous neuronal deficiencies that lead to profound neurological defects.

  7. regulation of dynamic exchange with Bach1

  8. mafG::mafK::mafF triple-mutant fibroblasts that completely lack small Mafs are highly susceptible to oxidative stress. (mafK)

  9. DNA-binding activity of endogenous MafA was significantly increased in the MafK transgenic mice

  10. The involvement of both HNF4alpha and NF-E2 in Abcc6 gene regulation suggests that Abcc6 might be involved in a detoxification processes related to hemoglobin or heme.

  11. MafK/NF-E2 p18 recruitment was involved in the physical proximity of LCR and active beta-globin genes upon beta-globin gene transcriptional activation.

  12. Histological analysis revealed that embryonic development of beta cells in the MafA(-/-)MafK(+) mice was significantly suppressed and the reduced number of beta cells was responsible for the early onset of diabetes.

MAFK Antigen Profile

Antigen Summary

The developmentally regulated expression of the globin genes depends on upstream regulatory elements termed locus control regions (LCRs). LCRs are associated with powerful enhancer activity that is mediated by the transcription factor NFE2 (nuclear factor erythroid-2). NFE2 recognition sites are also present in the gene promoters of 2 heme biosynthetic enzymes, porphobilinogen deaminase (PBGD\; MIM 609806) and ferrochelatase (FECH\; MIM 612386). NFE2 DNA-binding activity consists of a heterodimer containing an 18-kD Maf protein (MafF, MafG (MIM 602020), or MafK) and p45 (MIM 601490). Both subunits are members of the activator protein-1 superfamily of basic leucine zipper (bZIP) proteins (see MIM 165160). Maf homodimers suppress transcription at NFE2 sites.

Gene names and symbols associated with MAFK

  • MAF bZIP transcription factor K (MAFK) antibody
  • v-maf musculoaponeurotic fibrosarcoma oncogene family, protein K (avian) (Mafk) antibody
  • MAF bZIP transcription factor K (Mafk) antibody
  • AW061068 antibody
  • NF-E2 antibody
  • Nfe2u antibody
  • P18 antibody

Protein level used designations for MAFK

basic-leucine zipper transcription factor MafK , erythroid transcription factor NF-E2 p18 subunit , nuclear factor erythroid-2, ubiquitous (p18) , transcription factor MafK , v-maf avian musculoaponeurotic fibrosarcoma oncogene family, protein K , v-maf musculoaponeurotic fibrosarcoma oncogene homolog K , nuclear factor, erythroid derived 2, ubiquitous , p18 NF-E2

GENE ID SPECIES
7975 Homo sapiens
17135 Mus musculus
395518 Gallus gallus
100735555 Cavia porcellus
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