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anti-Mouse (Murine) Renin Antibodies:
anti-Rat (Rattus) Renin Antibodies:
anti-Human Renin Antibodies:
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Human Polyclonal Renin Primary Antibody for IF (p), IHC (p) - ABIN715436
Wanka, Staar, Lutze, Peters, Hildebrandt, Beck, Bäumgen, Albers, Krieg, Zimmermann, Sczodrok, Schäfer, Hoffmann, Peters: Anti-necrotic and cardioprotective effects of a cytosolic renin isoform under ischemia-related conditions. in Journal of molecular medicine (Berlin, Germany) 2015
Show all 3 Pubmed References
Human Monoclonal Renin Primary Antibody for ELISA, WB - ABIN562625
Salhan, Husain, Subrati, Goyal, Singh, Rai, Malhotra, Singhal: HIV-induced kidney cell injury: role of ROS-induced downregulated vitamin D receptor. in American journal of physiology. Renal physiology 2012
Cow (Bovine) Polyclonal Renin Primary Antibody for WB - ABIN2776795
Lavoie, Liu, Bianco, Beltz, Johnson, Sigmund: Evidence supporting a functional role for intracellular renin in the brain. in Hypertension 2006
Show all 2 Pubmed References
Results show negative regulation of HDAC1 (show HDAC1 Antibodies) by a Cul3 (show CUL3 Antibodies)-REN (show REN1 Antibodies) E3 ubiquitin ligase (show MUL1 Antibodies) complex.
REN (show REN1 Antibodies) is upregulated by neurogenic signals (retinoic acid, EGF (show EGF Antibodies), and NGF (show NGFB Antibodies)) in embryonal stem (ES) cells and neural progenitor cell lines in association with neurotypic differentiation.
REN (show REN1 Antibodies) protein enhances caspase-3 (show CASP3 Antibodies) activation and antagonizes the Shh (show SHH Antibodies) pathway suggesting that this gene may represent a restraint of Shh (show SHH Antibodies) signaling.
The current study provides evidence for a possible association of renin gene polymorphisms with essential hypertension in a Han population of northern China.
Plasma renin activity is associated with cardiovascular mortality or heart failure readmissions.
In patients with established atherosclerotic disease undergoing carotid endarterectomy, plasma renin and aldosterone concentrations were not associated with atherosclerotic plaque characteristics. Plasma renin concentration was positively associated with the occurrence of major secondary vascular events.
These observations demonstrate that activation of the renin-angiotensin system (RAS) promotes colitis in a blood pressure independent manner. Angiotensin II appears to drive colonic mucosal inflammation by promoting intestinal epithelial cell apoptosis and mucosal TH17 responses in colitis development
The genotypes of REN, AT1R (show AGTR1 Antibodies) and AT2R (show AGTR2 Antibodies) were not associated with the development of preeclampsia in South African Black women.
Urinary angiotensinogen (show AGT Antibodies) and renin excretion are elevated in CKD patients. Both parameters are negatively associated with eGFR (show EGFR Antibodies) and these associations are independent of urinary albumin (show ALB Antibodies) excretion
Autosomal dominant polycystic kidney disease (ADPKD), uniquely increases urinary angiotensinogen (show AGT Antibodies) and renin excretion despite their circulating levels being comparable with those in non-ADPKD chronic kidney disease.
Prospective study of consecutive cardiac disease patients referred for cardiac catheterization has revealed distinct cardiac disease condition-associated differences in the frequencies of elevations in plasma renin, plasma aldosterone concentration, and the aldosterone-renin ratio
Renin-angiotensin system transgenic mouse model suggests that renal injury in preeclampsia may be mediated through local VEGF (show VEGFA Antibodies).
Data suggest mild primary aldosteronism (PA), where plasma renin activity is not as suppressed, is susceptible to dietary sodium influences on renin and ARR (show SAG Antibodies) (aldosterone to renin ratio). Optimal screening for PA should occur under conditions of high dietary sodium intake (rather than condition of low dietary sodium intake as frequently prescribed for patients with hypertension, which may be a symptom of PA).
Renin catalyzes the first step in the activation pathway of angiotensinogen--a cascade that can result in aldosterone release,vasoconstriction, and increase in blood pressure. Renin, an aspartyl protease, cleaves angiotensinogen to form angiotensin I, which is converted to angiotensin II by angiotensin I converting enzyme, an important regulator of blood pressure and electrolyte balance. Transcript variants that encode different protein isoforms and that arise from alternative splicing and the use of alternative promoters have been described, but their full-length nature has not been determined. Mutations in this gene have been shown to cause familial hyperproreninemia.
, BTB/POZ domain-containing protein KCTD11
, retinoic acid, EGF, and NGF upregulated
, renin 1 structural
, angiotensin-forming enzyme
, renin precursor, renal