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anti-Human IL17D Antibodies:
anti-Mouse (Murine) IL17D Antibodies:
anti-Rat (Rattus) IL17D Antibodies:
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Mouse (Murine) Monoclonal IL17D Primary Antibody for CyTOF, FACS - ABIN4899881
Seelige, Saddawi-Konefka, Adams, Picarda, Sun, Benedict, Bui: Interleukin-17D and Nrf2 mediate initial innate immune cell recruitment and restrict MCMV infection. in Scientific reports 2018
Nrf2 Induces IL-17D to mediate tumor and virus surveillance.
Study of the expression of IL-17 provides theoretical support for the clinical treatment and prognosis prediction of ovarian epithelial tumor.
Utilizing gene microarray analysis, authors found the cytokine interleukin 17D (IL-17D) was highly expressed in certain unedited tumors.
These experiments were designed to investigate the effects of IL-17 upon the phenotype and function of human Natural Killer (NK) cells.
IL-27 priming results in enhanced lipopolysaccharide-induced IL-6, tumor necrosis factor (TNF)-alpha, macrophage inflammatory protein (MIP)-1alpha, and MIP-1beta expression in human primary monocytes.
IL-17D stimulates production of cytokines in endothelial cells in vitro and suppresses proliferation of myeloid progenitors in colony formation assays.
Mice deficient in IL-17D or the transcription factor Nuclear factor (erythroid-derived 2)-like 2 (Nrf2), an inducer of IL-17D, featured an early decreased number of innate immune cells at the point of viral entry and were more susceptible to mouse cytomegalovirus infection.
a new role for nuclear factor erythroid-derived 2-like 2 (Nrf2) in regulating early anti-cancer immune responses by inducing IL-17D and recruiting natural killer (NK) cells.
Data indicate that IL-17A, up-regulated by 218-fold during early inflammation, suggesting involvement of T helper 17 cells in the pathogenesis of the soybean meal (SBM)-induced inflammatory response.
The protein encoded by this gene is a cytokine that shares the sequence similarity with IL17. The treatment of endothelial cells with this cytokine has been shown to stimulate the production of other cytokines including IL6, IL8 and CSF2/ GM-CSF. The increased expression of IL8 induced by this cytokine was found to be NF-kappa B-dependent.
, interleukin 27A
, interleukin 17D
, Interleukin 17-4 (il17-4)
, interleukin 17D-like
, interleukin 17d