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Human Monoclonal TGFb Primary Antibody for - ABIN577154
Nishiyama, McDonough, Bruns, Burgeson: Type XII and XIV collagens mediate interactions between banded collagen fibers in vitro and may modulate extracellular matrix deformability. in The Journal of biological chemistry 1994
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Human Polyclonal TGFb Primary Antibody for IHC (p), ELISA - ABIN2476753
Goldraich, Ramos, Goldraich: Urography versus DMSA scan in children with vesicoureteric reflux. in Pediatric nephrology (Berlin, Germany) 1990
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Cow (Bovine) Monoclonal TGFb Primary Antibody for ELISA, FACS - ABIN2476751
Mahan, Ballal, Nanda: Mitral valve tear complicating percutaneous valvuloplasty: diagnosis by transesophageal Doppler color flow mapping. in American heart journal 1991
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BMP/TGF-beta signaling pathway regulates X-inactive specific transcript (XIST), and whose down-regulation reactivated the methyl CpG binding protein 2 (MeCP2) reporter on the inactive X chromosome (Xi).
Expression of TGF-beta1 (show TGFB1 Antibodies) also did not associate with clinical outcome in either of groups. Our results suggest that changes in TGF-beta1 (show TGFB1 Antibodies) are associated with the progression of oral carcinogenesis
No association was found between target lesion revascularization after percutaneous coronary intervention and TGFB1 (show TGFB1 Antibodies) genotype.
real-time polymerase chain reaction showed higher expression levels of TGF-beta (show TGFB1 Antibodies) and CTGF (show CTGF Antibodies) in desmoid fibromatosis compared with normal skin. The high constitutive expression of beta-catenin (show CTNNB1 Antibodies) downstream effectors; TGF-beta (show TGFB1 Antibodies), CTGF (show CTGF Antibodies) has the potential for enabling targeted therapy
miR (show MLXIP Antibodies)-9 was up-regulated and SOX7 (show SOX7 Antibodies) was down-regulated in human non-small-cell lung cancer tissues and cell lines.
For the TGF-beta1 (show TGFB1 Antibodies) polymorphism rs1982073, the results indicted that the C allele was associated with decreased risk of COPD (show ARCN1 Antibodies) in Caucasians (odds ratio (OR) =0.79, 95% confidence interval (CI): 0.64-0.99, P=0.038) but not in Asians (OR =0.95, 95% CI: 0.71-1.28, P=0.741).
Studied serum Levels of interleukins 6 and 8 (IL-6 (show IL6 Antibodies) and IL-8 (show IL8 Antibodies)), tumor necrosis factor(TNF-alpha (show TNF Antibodies)), and transforming growth factor beta (TGF-beta) in association with chronic hepatitis B patients with depression or anxiety.
Heme oxygenase-1 (show HMOX1 Antibodies)/CO axis inhibits TGF-beta1 (show TGFB1 Antibodies)-induced growth inhibition in human hepatocellular carcinoma cell lines.
KC can co-exist with GCD. The missense mutation (c.370G > A) in the TGFBI gene and insert mutation (c.1456-1457ins GAT) in the KRT12 gene were identified in a 23-year-old male patient with concurrent KC and GCD.
we found p53 (show TP53 Antibodies) to play a crucial role as a Smad (show SMAD1 Antibodies) partner in TGF-beta (show TGFB1 Antibodies)-mediated tumor suppression, and the functional switching of TGF-beta (show TGFB1 Antibodies) is partially caused by p53 (show TP53 Antibodies) loss or its mutation during tumor development.
TGF-beta1 (show TGFB1 Antibodies) stimulated lubricin (show PRG4 Antibodies) secretion by superficial zone chondrocytes at all densities with twice-a-week TGF-beta (show TGFB1 Antibodies) treatment. It is noteworthy that the daily treatment of TGF-beta1 (show TGFB1 Antibodies) increased lubricin (show PRG4 Antibodies) much higher compared with twice-a-week treatment.
hypoxia increased the expression of platelet-derived growth factor (PDGF (show PDGFA Antibodies)) and transforming growth factor-beta1 (TGF-beta1 (show TGFB1 Antibodies)) and decreased the expression of neprilysin (NEP (show MME Antibodies)), which contributed to the hypoxia-induced Endothelial-to-mesenchymal transition of pulmonary artery endothelial cells.
TGF-beta1 (show TGFB1 Antibodies) modulates the expression of syndecan-4 (show SDC4 Antibodies) in cultured vascular endothelial cells in a biphasic manner.
Taken together, Staphylococcus aureus induces TGF-beta1 (show TGFB1 Antibodies) and bFGF (show FGF2 Antibodies) expression through the activation of AP-1 (show JUN Antibodies) and NF-kappaB (show NFKB1 Antibodies) in bovine mammary gland fibroblasts.
The results identify TGFB1 (show TGFB1 Antibodies) and ESRRA (show ESRRA Antibodies) as likely transcriptional regulators of rumen epithelial development and energy metabolism, respectively, and provide targets for modulation of rumen development and function in the growing calf.
the combined treatment with TGF-beta1 (show TGFB1 Antibodies) and BMP-7 (show BMP7 Antibodies) or treatment first with TGF-beta1 (show TGFB1 Antibodies) followed by BMP-7 (show BMP7 Antibodies) was more effective than other treatment groups in both chondrogenic differentiation and SZP (show PRG4 Antibodies) secretion.
Tenascin-X (show TNXB Antibodies) promotes activation of latent TGF-beta1 (show TGFB1 Antibodies) and subsequent epithelial to mesenchymal transition in mammary epithelial cells.
a detailed computational model for TGF-beta (show TGFB1 Antibodies) signalling that incorporates elements of previous models together with crosstalking between Smad1 (show SMAD1 Antibodies)/5/8 and Smad2 (show SMAD2 Antibodies)/3 channels through a negative feedback loop dependent on Smad7 (show SMAD7 Antibodies).
Endogenous TGF-beta1 (show TGFB1 Antibodies) became more bioactive following activation of the transgene protein product in chondrocytes.
A novel peptide, P2K, regulating TGF-beta1 (show TGFB1 Antibodies) signaling had an anabolic effect on bovine intervertebral disc cells and rabbit degenerated discs.
Data indicate that cardiac contractility modulation (CCM) therapy exerted protective effects against myocardial fibrosis potentially by inhibiting TGF-beta1 (show TGFB1 Antibodies)/Smad3 (show SMAD3 Antibodies) signaling pathway in chronic heart failure .
study suggested that TGF-beta1/Smad3/smad7 is a major pathway which plays an important role in the regulation of the IUA and specific inhibitor of Smad3 (SIS3) may provide a new therapeutic strategy for IUA.
cell therapy promoted TGF-beta1 (show TGFB1 Antibodies) expression in nucleus pulposus, leading to anti-inflammatory effects via the inhibition of NF-kappaB (show NFKB1 Antibodies), and the amelioration of disc degradation.
that prenatal tracheal occlusion increases TGF-beta (show TGFB1 Antibodies)/Rho kinase (show ROCK1 Antibodies) pathway, myofibroblast differentiation, and matrix deposition in neonatal rabbit and human congenital diaphragmatic hernia lungs
observation. Based on the above results, we conclude that TGF-beta1 (show TGFB1 Antibodies)-immobilized PLGA-gelatin scaffold seeded with ASCs considerably enhances the quality of the tissue-engineered cartilage, therefore, advancing the field of cartilage tissue engineering
Smad7 (show SMAD7 Antibodies) plays a crucial role in antagonizing epithelial-mesenchymal transition induced by TGFbeta (show TGFB1 Antibodies) signaling and is a Notch (show NOTCH1 Antibodies) signaling target in limbal epithelial stem cells.
the role of IL-10 in inhibited allograft rejection may be associated with CD4+CD25+ regulatory T cells and IL-10, and may be independent of TGF-b
After NOTCH1 (show NOTCH1 Antibodies) knockdown and TGFB1 (show TGFB1 Antibodies) stimulation, rabbit mesenchymal stem cells expressed higher levels of proteoglycan (show Vcan Antibodies) and collagen II.
TGF-beta1 (show TGFB1 Antibodies) gene transcription significantly correlates with the surgical vaginal and dermal wound closure rate.
TGF beta (show TGFB1 Antibodies) is involved in the pathogenesis of tympanosclerosis.
There was significantly higher expression of TGF-beta1 (show TGFB1 Antibodies) and MMP-9 (show MMP9 Antibodies) in nasal mucosa of experimental allergic rhinitis guinea pigs than in controls.
TGF-beta1 (show TGFB1 Antibodies) regulated pAKT (show AKT1 Antibodies) and IFNgamma expressions were associated with epithelial cell survival in rhesus macaque colon explants and suggest a potential role of mucosal TGF-beta1 (show TGFB1 Antibodies) in regulating intestinal homeostasis and EC integrity.
SIV infection of rhesus macaques results in the emergence of IL-17 (show IL17A Antibodies)-expressing cells during the acute phase. This subpopulation appears at day 14 postinfection concomitantly with an increase in TGF-beta (show TGFB1 Antibodies) and IL-18 (show IL18 Antibodies) expression.
SIV-infected macaques exhibiting progression to AIDS displayed greater expression of TGF-beta (show TGFB1 Antibodies) and indoleamine 2,3 dioxygenase in CD8 (show CD8A Antibodies)+ T cells from mesentric lymph nodes.
Activated TGF-beta (show TGFB1 Antibodies) signaling rescued miR (show MYLIP Antibodies)-143-reduced FSHR (show FSHR Antibodies) and intracellular signaling molecules, and miR (show MYLIP Antibodies)-143-induced porcine granulosa cell apoptosis.
TGF-beta1 (show TGFB1 Antibodies)-induced epithelial-myofibroblast plasticity is FAK (show PTK2 Antibodies)- dependent, whereas TGF-beta1 (show TGFB1 Antibodies)-induced apoptosis is favored when FAK (show PTK2 Antibodies) signaling is inhibited.
this study shows that TGF-beta1 (show TGFB1 Antibodies) protects intestinal integrity and influences Smad (show SMAD1 Antibodies) and MAPK (show MAPK1 Antibodies) signal pathways in intestinal epithelium cells after TNF-alpha (show TNF Antibodies) challenge
this study shows that anemonin may ameliorate LPS (show IRF6 Antibodies)-induced intestinal injury and improve restoration by regulating the TGF-b1 and EGFR (show EGFR Antibodies) signaling pathways
The results indicated that TGF-beta-1 (show TGFB1 Antibodies) was associated with the restoration of intestinal morphology and barrier function following weaning stress.
Data (including data from in vitro and in vivo experiments) suggest that day 14 elongated conceptus secretes proteins that up-regulate TGFbeta1 (show TGFB1 Antibodies) mRNA and TGFbeta1 (show TGFB1 Antibodies) expression in endometrium; TGFbeta1 (show TGFB1 Antibodies) may be important during pregnancy maintenance.
The effects of semen, spermatozoa in extender, or extender alone on the expression of TGF-beta1 (show TGFB1 Antibodies), IL-10 (show IL10 Antibodies), and IL-6 (show IL6 Antibodies) in ovarian follicles are reported.
Dietary (1,3/1,6)-beta-D-glucan reduced the mRNA expression of transforming growth factor (TGF) beta2 (show TGFB2 Antibodies) and tended to reduce the mRNA expression of TGF-beta1 (show TGFB1 Antibodies) in lung tissue of neonatal piglet.
TGF-beta1 (show TGFB1 Antibodies), via TGF-beta1 (show TGFB1 Antibodies) receptor I and p38 MAPK (show MAPK14 Antibodies) signaling, reduces CFTR (show CFTR Antibodies) expression to impair CFTR (show CFTR Antibodies)-mediated anion secretion, which would likely compound the effects associated with mild CFTR (show CFTR Antibodies) mutations and ultimately would compromise male fertility.
High yield isolation of BMP-2 (show BMP2 Antibodies) from bone and in vivo activity of a combination of BMP-2 (show BMP2 Antibodies)/TGF-beta1 (show TGFB1 Antibodies).
Study shows that expression of Gadd45 (show GADD45A Antibodies) family members is adjusted in response to KCl/depolarization, BDNF (show BDNF Antibodies)/TRKB (show NTRK2 Antibodies), and/or TGFB (show TGFB1 Antibodies)-signaling. Additionally, results indicate decreased Gadd45 (show GADD45A Antibodies) expression in a mouse model of depression, and that Gadd45b (show GADD45B Antibodies) expression and DNA demethylation activate Arc (show NOL3 Antibodies) transcription.
Bioinformatics analysis provides positive supportive correlation between somatic mutations for VD-related genes and the TGF-beta (show TGFB1 Antibodies) pathway
Taken together, these results indicate that eIF6 (show EIF6 Antibodies) may be involved in external mechanical force-mediated murine dermal fibroblast function at least partly through the TGF-beta1 (show TGFB1 Antibodies)/TGFBR1 (show TGFBR1 Antibodies)/TGFBR2 (show TGFBR2 Antibodies) pathway.
Osteopontin (show SPP1 Antibodies) is highly induced in carbon nanotube-exposed lungs and plays critical roles in TGF-beta1 (show TGFB1 Antibodies) signaling activation and myofibroblast differentiation to promote fibrosis development.
These data suggest that the interplay between cell-matrix adhesion and intercellular adhesion is an important determinant for some aspects of TGFbeta1 (show TGFB1 Antibodies)-induced epithelial-mesenchymal transition via alphaSMA (show ACTA2 Antibodies) expression induction.
Excessive activation of TGFbeta (show TGFB1 Antibodies) by spinal instability causes vertebral endplate sclerosis and intervertebral disk degeneration.
study reveals a critical mechanism by which TGFbeta (show TGFB1 Antibodies) controls TH17 cell differentiation and uncovers the SKI (show SKI Antibodies)-SMAD4 (show SMAD4 Antibodies) axis as a potential therapeutic target for treating TH17-related diseases
A Smad3 (show SMAD3 Antibodies)-PTEN regulatory loop controls proliferation and apoptotic responses to TGF-beta (show TGFB1 Antibodies) in mouse endometrium.
TGFss through the Alk1 (show ACVRL1 Antibodies)/TgfssR2 receptor axis is acting on endothelial cells to produce hematopoiesis.
The adoptive transfer of NK1.1(-) CD4 (show CD4 Antibodies)(+) NKG2D (show KLRK1 Antibodies)(+) cells suppressed DSS (show PMP22 Antibodies)-induced colitis largely dependent on TGF-beta (show TGFB1 Antibodies). Thus, NK1.1(-) CD4 (show CD4 Antibodies)(+) NKG2D (show KLRK1 Antibodies)(+) cells exhibited immune regulatory functions, and this T cell subset could be developed to suppress inflammation in clinics.
This gene encodes a member of the transforming growth factor beta (TGFB) family of cytokines, which are multifunctional peptides that regulate proliferation, differentiation, adhesion, migration, and other functions in many cell types. Many cells have TGFB receptors, and the protein positively and negatively regulates many other growth factors. The secreted protein is cleaved into a latency-associated peptide (LAP) and a mature TGFB1 peptide, and is found in either a latent form composed of a TGFB1 homodimer, a LAP homodimer, and a latent TGFB1-binding protein, or in an active form composed of a TGFB1 homodimer. The mature peptide may also form heterodimers with other TGFB family members. This gene is frequently upregulated in tumor cells, and mutations in this gene result in Camurati-Engelmann disease.
TGF-beta 1 protein
, latency-associated peptide
, transforming growth factor beta-1
, transforming growth factor, beta 1 (Camurati-Engelmann disease)
, transforming growth factor-beta 1
, transforming growth factor beta 1
, transforming growth factor-beta
, transforming growth factor beta1
, transforming growth factor-beta-1
, TGF-beta 1
, regulatory protein
, transforming growth factor, beta-1