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Results identified a novel pH-dependent mechanism by which epigenetic reader, the PHD (show PDC Antibodies) fingers of DIDO, recognize the histone mark H3K4me3. The pH-sensing ability might be necessary for normal biological processes and those characterized by altered cellular pH.
evaluated the Death Inducer-Obliterator (DIDO) (variants DIDO 1, 2 and 3) levels in CML (show BCR Antibodies), PV, ET and PMF (show PRB1 Antibodies) patients. Our data reported the DIDO 1, 2 and 3 differential expressions in Myeloproliferative Neoplasms.
Dido1 induces the expression of Integrin alphaV, thereby promoting the attachment, migration, invasion and apoptosis resistance of melanoma cells.
Data show that CSE1L (show CSE1L Antibodies), DIDO1 and RBM39 (show RBM39 Antibodies) mRNA expression levels correlated with chromosome 20q DNA copy number status.
Gambogic acid induces the apoptosis of Raji cells through DIO-1 upregulation, nuclear translocation, Bcl-xL (show BCL2L1 Antibodies) downregulation and caspase 3 (show CASP3 Antibodies) activation.
Novel transcription factors identified in human CD34 antigen (show CD34 Antibodies)-positive hematopoietic stem cells.
Dido3, the largest splice variant of the Dido gene, is a centrosome-associated protein (show CEP250 Antibodies) whose disruption leads to supernumerary centrosomes, failure to maintain cellular mitotic arrest, and early degradation of the mitotic checkpoint (show BUB3 Antibodies) protein BubR1 (show BUB1B Antibodies).
Gambogic acid induces DIDO1-mediated apoptosis in Jurkat T cells.
Embryonic stem cells mainly express DIDO3 and their differentiation after leukemia inhibitory factor (show LIF Antibodies) withdrawal requires DIDO1 expression. DIDO3 regulates DIDO1 expression.
Death inducer obliterator (Dido3)-dependent targeting of histone deacetylase 6 (HDAC6 (show HDAC6 Antibodies)) is a key determinant of cilium size in growth-arrested cells
Dido1 is able to regulate self-renewal of mouse embryonic stem cells.
Dido3 PHD (show PDC Antibodies) interacts with histone H3K4me3 and regulates expression of stemness genes in embryonic stem cells.
loss of Dido3 expression compromises differentiation of embryonic stem cells in vitro and of epiblast cells in vivo, resulting in early embryonic death at around day 8.5 of gestation
Dido3, the largest splice variant of the Dido gene, is a centrosome-associated protein (show BLOC1S2 Antibodies) whose disruption leads to supernumerary centrosomes, failure to maintain cellular mitotic arrest, and early degradation of the mitotic checkpoint (show BUB3 Antibodies) protein BubR1 (show BUB1B Antibodies).
results indicate that histone H3 (show HIST3H3 Antibodies) lysine 4 demethylation modulates DIDO3 localization in meiosis and suggest epigenetic regulation of the synaptonemal complex
Apoptosis, a major form of cell death, is an efficient mechanism for eliminating unwanted cells and is of central importance for development and homeostasis in metazoan animals. In mice, the death inducer-obliterator-1 gene is upregulated by apoptotic signals and encodes a cytoplasmic protein that translocates to the nucleus upon apoptotic signal activation. When overexpressed, the mouse protein induced apoptosis in cell lines growing in vitro. This gene is similar to the mouse gene and therefore is thought to be involved in apoptosis. Alternatively spliced transcripts have been found for this gene, encoding multiple isoforms.
death-associated transcription factor 1
, death-inducer obliterator 1
, death inducer-obliterator-2
, death inducer-obliterator-3
, death associated transcription factor 1
, death inducer-obliterator 1
, death-inducer obliterator 1-like