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IFNa (show IFNA ELISA Kits) and IFNc show similar antiviral activities and ability to induce antiviral genes in lymphoid tissues.
Data indicate that the combination of AAV2.IL-4 (show IL4 ELISA Kits) and AAV2.IFN-alpha (show IFNA ELISA Kits) was not significantly different as compared to AAV2.IL-4 (show IL4 ELISA Kits) alone.
Infecting virus and the target cell type dictate the extent and signature of inducible IFN-alpha2 gene expression.
Mutation of the IFNAR-1 (show IFNAR1 ELISA Kits) receptor binding site of human IFN-alpha2 generates type I IFN competitive antagonists.
produced in vitro and in vivo in response to noncytopathic bovine viral diarrhea virus in lymph nodes cells expressing the myeloid markers CD14 (show CD14 ELISA Kits), CD11b (show ITGAM ELISA Kits), and CD172a (show SIRPA ELISA Kits)
Prolonged treatment with IFN-alpha (12-48 h) resulted in increased expression of STAT1 (show STAT1 ELISA Kits) and, to a lesser extent, STAT2 (show STAT2 ELISA Kits).
All the tested TLRAs induced greater infant IFN-alpha2 production compared to newborn and adult blood. In contrast, CpG induced greater IFN-gamma (show IFNG ELISA Kits), IL-1beta (show IL1B ELISA Kits), IL-4 (show IL4 ELISA Kits), IL-12p40, IL-10 (show IL10 ELISA Kits) and CXCL8 (show IL8 ELISA Kits) in newborn than in infant and adult blood.
Interferon-alpha (show IFNA ELISA Kits)-induced CD100 (show SEMA4D ELISA Kits) expression on naive CD8 (show CD8A ELISA Kits)(+) T cells enhances antiviral responses to hepatitis C infection through CD72 (show CD72 ELISA Kits) signal transduction.
Tumor-associated M2 macrophages in mycosis fungoides acquire immunomodulatory function on exposure to IFN-alpha2a and IFN-gamma (show IFNG ELISA Kits).
Increase in serum IFNA2 is associated with adenoma.
Data suggest IFNA2 binding to extracellular domain of IFN receptors IFNAR1 (show IFNAR1 ELISA Kits) or IFNAR2 (show IFNAR2 ELISA Kits) promotes proximity between intracellular domains; signaling depends on duration of activation and affinity of binding rather than specific conformational changes.
Report biased expression of human interferon alpha-2b and Escherichia coli methionine amino peptidase genes under control of single promoter in E. coli.
interferon alpha-2b gene mutations were identified among brain tumor patients; highest percentage of frameshift mutations was identified; patients were found to be under environmental stress from contaminated drinking water and from local gamma radiations
PPARalpha (show PPARA ELISA Kits) activation by an agonist WY-14643 could potentiate IFN-alpha (show IFNA ELISA Kits) responses, reverse IFN-alpha (show IFNA ELISA Kits) refractoriness, and enhance viral eradication in hepatocytes.
The chronic use of low dose of radiations by occupational workers has a significant correlation with mutational effects on interferon alpha 2b gene, further evident by depressed interferon alpha (show IFNA ELISA Kits) levels in serum.
IFN-beta (show IFNB1 ELISA Kits) was significantly more effective than IFNA2 in protecting human head and neck squamous cell carcinoma lines from oncolysis by vesicular stomatitis.
This gene is a member of the alpha interferon gene cluster on chromosome 9. The encoded protein is a cytokine produced in response to viral infection. Use of the recombinant form of this protein has been shown to be effective in reducing the symptoms and duration of the common cold.
, interferon alpha 2
, interferon alpha family, gene 2
, interferon alpha A
, IFN-alpha 2
, interferon-alpha 2
, alpha-2a interferon
, interferon alpha 2a
, interferon alpha 2b
, leIF A