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Human IFNB1 ELISA Kit for Sandwich ELISA - ABIN414734
Li, Li, Qian, Zhang, Chen, Shi: Impaired TLR3/IFN-beta signaling in monocyte-derived dendritic cells from patients with acute-on-chronic hepatitis B liver failure: relevance to the severity of liver damage. in Biochemical and biophysical research communications 2009
Show all 10 Pubmed References
Horse (Equine) IFNB1 ELISA Kit for Sandwich ELISA - ABIN416691
Waldschmidt, Pirottin, Art, Audigié, Bureau, Tosi, El Abbas, Farnir, Richard, Dupuis: Experimental model of equine alveolar macrophage stimulation with TLR ligands. in Veterinary immunology and immunopathology 2013
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Rhesus Monkey IFNB1 ELISA Kit for Sandwich ELISA - ABIN415337
Bosinger, Johnson, Folkner, Patel, Hashempour, Jochems, Del Rio Estrada, Paiardini, Lin, Vanderford, Hiscott, Silvestri: Intact type I Interferon production and IRF7 function in sooty mangabeys. in PLoS pathogens 2013
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Mouse (Murine) IFNB1 ELISA Kit for Sandwich ELISA - ABIN415550
Kang, Kim, Choi, Kim, Lee: Scoparone attenuates D-galactosamine/lipopolysaccharide-induced fulminant hepatic failure through inhibition of toll-like receptor 4 signaling in mice. in Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association 2013
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Chicken IFNB1 ELISA Kit for Sandwich ELISA - ABIN414096
Feng, Dai, Xie, Li, Shi, Zhang: Innate Immune Responses in ALV-J Infected Chicks and Chickens with Hemangioma In Vivo. in Frontiers in microbiology 2016
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Rat (Rattus) IFNB1 ELISA Kit for Sandwich ELISA - ABIN416262
Kang, Koh, Lee: Melatonin protects liver against ischemia and reperfusion injury through inhibition of toll-like receptor signaling pathway. in Journal of pineal research 2011
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Cow (Bovine) IFNB1 ELISA Kit for Competition ELISA - ABIN577538
Zhang, Fu, Deng, Xie, Xu, Liu, Hu, Chen, Guo: Attenuation of bovine herpesvirus type 1 by deletion of its glycoprotein G and tk genes and protection against virulent viral challenge. in Vaccine 2011
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Dog (Canine) IFNB1 ELISA Kit for Competition ELISA - ABIN993158
Han, Park, Ahn, Park, Jung, Seo, Ra, Kang, Lee, Youn: Pro-apoptotic and Growth-inhibitory Effect of IFN-?-Overexpressing Canine Adipose Tissue-derived Mesenchymal Stem Cells Against Melanoma Cells. in Anticancer research 2015
Mouse (Murine) IFNB1 ELISA Kit for Sandwich ELISA - ABIN366343
Yin, Xu, Sun, Wei, Tian: Interleukin-15 suppresses hepatitis B virus replication via IFN-? production in a C57BL/6 mouse model. in Liver international : official journal of the International Association for the Study of the Liver 2012
Pig (Porcine) IFNB1 ELISA Kit for Sandwich ELISA - ABIN365282
Cruz, Becares, Sola, Oliveros, Enjuanes, Zúñiga: Alphacoronavirus protein 7 modulates host innate immune response. in Journal of virology 2013
either JC1 or DS1 C/EBP site is sufficient to mediate IFN beta-induced down-regulation of SIV long terminal repeat activity and virus replication in macrophages
Hepatitis A virus protein 2B suppresses beta interferon (show IFNA ELISA Kits) (IFN) gene transcription by interfering with IFN regulatory factor 3 activation.
BVDV2-E significantly increased IFN-beta activity compared to BVDV2-wt.
The data confirm the involvement of EHMT2 (show EHMT2 ELISA Kits) in the epigenetic regulation of IFN-b and demonstrate the activation of a general antiviral state after EHMT2 (show EHMT2 ELISA Kits) inhibition.
The authors provide evidence that ICP27 protein encoded by bovine herpesvirus type 1, a viral early protein that shuttles between the nucleus and cytoplasm inhibits transcriptional activity of two bovine IFN-beta gene promoters (IFN-beta1 and IFN-beta3).
The authors demonstrate that bovine herpesvirus 1 bICP0 effectively inhibits bovine IFN-beta promoter activity and induces IRF3 (show IRF3 ELISA Kits) degradation.
These studies provide evidence that virus infection differentially stimulates expression of the three bovine IFN-beta genes.
recombinant bovine respiratory syncytial virus lacking the NS proteins, and those lacking NS2 in particular, are strong inducers of IFN-alpha (show IFNA ELISA Kits)/beta in bovine nasal fibroblasts and bronchoalveolar macrophages.
The effect of topical TREX1 (show TREX1 ELISA Kits) knockdown and local interferon (show IFNA ELISA Kits) production on HIV transmission in human cervicovaginal explants and humanized mice, is reported.
The TBK1 (show TBK1 ELISA Kits) Y179A mutant failed to rescue type I IFN production by virally infected RAW264.7 macrophages deficient in TBK1 (show TBK1 ELISA Kits).
this study shows that poly I:C treated PAR-1 (show MARK2 ELISA Kits)-/- mice given the thrombin (show F2 ELISA Kits) inhibitor dabigatran etexilate exhibited less IFNbeta and CXCL10 (show CXCL10 ELISA Kits) expression in the spleen and plasma
The data demonstrate that an atypical TLR7 (show TLR7 ELISA Kits) signaling pathway contributes to type interferon-beta expression during Y. pestis infection and suggest that the TLR7 (show TLR7 ELISA Kits)-driven type I IFN response plays an important role in determining the outcome of plague.
Long-term exposure to atmospheric particulates, PM2.5 up-regulated H3K4 and H3K9 methylation in IL-6 (show IL6 ELISA Kits) and IFN-beta promoter regions through down-regulating Kdm6a (show KDM6A ELISA Kits) expression. The results suggest that short-term exposure to PM2.5 significantly enhances the survival rate of influenza A-contaminated mice, while long-term PM2.5 inhalation lowers the capacity of pulmonary macrophages to secrete IL-6 (show IL6 ELISA Kits) and IFN-beta.
Extracellular ATP reduces the replication of VSV, Newcastle disease virus, murine leukemia virus and HSV in vivo and in vitro through the P2X7 receptor (show P2RX7 ELISA Kits); ATP increases IFN-beta expression. Mechanistically, ATP facilitates IFN-beta secretion through P38 (show CRK ELISA Kits)/JNK (show MAPK8 ELISA Kits)/ATF-2 (show ATF2 ELISA Kits) signaling pathways, which are crucial in promoting antiviral immunity.
study reports that mitochondrial antiviral signaling protein (MAVS (show MAVS ELISA Kits)), -mediated IFN-alpha (show IFNA ELISA Kits)/beta production and IFNAR (show IFNAR1 ELISA Kits) signaling are required for alloimmune responses to the human K1 Ag in a murine model of inflammation-induced alloimmunization
study identifies a novel role for RIPK1 (show RIPK1 ELISA Kits) and RIPK3 (show RIPK3 ELISA Kits), a pair of homologous serine/threonine kinases previously implicated in the regulation of necroptosis and pathologic tissue injury, in directing IFN-beta production in macrophages
results suggest that, in addition to its well-known signaling activity, IFN-beta may be directly antimicrobial and be part of a growing family of cytokines and chemokines, called kinocidins, that also have antimicrobial properties.
results show that resistance to HSV-1 in the trigeminal ganglia during acute infection is conferred in part by STING and IFN-alpha (show IFNA ELISA Kits)/beta signaling in both bone marrow-derived and -resident cells, which coalesce to support a robust HSV-1-specific CD8 (show CD8A ELISA Kits)(+) T cell response
this study shows that the IFN-beta/STAT1 (show STAT1 ELISA Kits) pathway is dysregulated in inflammatory bowel disease
results demonstrate that Sphingosine 1-phosphate lyase (SPL (show SGPL1 ELISA Kits)) is a host factor that augments type I IFN responses during influenza A virus infection; study delineates the relationship between IKKepsilon (show IKBKE ELISA Kits) and SPL (show SGPL1 ELISA Kits), which provides a mechanistic understanding of the pro-IFN activity of SPL (show SGPL1 ELISA Kits)
This study demonstrates a novel pathway for elevated IFNbeta signaling in SLE that is not dependent on stimulation by immune complexes but rather is cell intrinsic and critically mediated by IFNbeta and MAVS (show MAVS ELISA Kits).
G45R mutation of NS1 (show PTPN11 ELISA Kits) slightly decreased NS1 (show PTPN11 ELISA Kits) binding to dsRNA but did not interfere with its suppression of RIG-I (show DDX58 ELISA Kits)-mediated type I IFN production.
Upon influenza virus infection, DPF2 dysregulated IFN-beta induction and expression of cytokines/chemokines and antiviral proteins. This study provides evidence that influenza virus utilizes DPF2 to escape host innate immunity.
Overexpression of PKV VP3 blocked IFN-beta-induced activation of the STAT1 (show STAT1 ELISA Kits)/STAT2 (show STAT2 ELISA Kits)/IRF9 (show IRF9 ELISA Kits) promoter in a dose-dependent manner.
Data suggest that EV71 infection in enterocytes does not inhibit phosphorylation of STAT1 (show STAT1 ELISA Kits)/2 induced by IFN-beta, but p-STAT1 (show STAT1 ELISA Kits)/2 transport into the nucleus is significantly blocked; EV71 infection in enterocytes down-regulates expression of KPNA1 (show KPNA1 ELISA Kits) and induces degradation of cellular KPNA1 (show KPNA1 ELISA Kits) via caspase-3 (show CASP3 ELISA Kits). [EV17 = Enterovirus 71]
This study reveals that decreased induction of both PKR (show PKLR ELISA Kits) and p300 (show EP300 ELISA Kits) proteins contribute to impaired induction of IFN-beta in Chronic obstructive pulmonary disease primary bronchial epithelial cells upon influenza infection.
Nuclear export of NSP1alpha of porcine reproductive and respiratory syndrome virus was necessary for its ability for IFN beta inhibition.
Porcine deltacoronavirus nsp5, the 3C-like protease, inhibits interferon-beta production through the cleavage of NEMO (show IKBKG ELISA Kits).
Highly pathogenic Porcine reproductive and respiratory syndrome virus modulates Interferon-beta expression mainly through attenuating IRF-3 (show IRF3 ELISA Kits) phosphorylation.
Porcine epidemic diarrhea virus nsp1 inhibited the IFN-beta and IRF3 (show IRF3 ELISA Kits) promoter activities.
poIFIT3 plays a significant role in the clearance of swine influenza virus in pigs and potentiates IFN-beta production.
DDX41 (show DDX41 ELISA Kits) is involved in the dsDNA- and dsDNA-virus-mediated type IFN-beta signaling pathway in porcine kidney cells.
VIral NS4 (show SOS1 ELISA Kits) protein antagonizes beta interferon (show IFNA ELISA Kits) expression by targeting the NF-kappaB (show NFKB1 ELISA Kits) essential modulator.
Swine IFN-beta promotes genetic mutation of porcine reproductive and respiratory syndrome virus.
Expression of LSm14A (show LSM14A ELISA Kits) in HEK293 or Marc (show CCL7 ELISA Kits)-145 cells enhanced activities of IFN-beta and NF-kappaB (show NFKB1 ELISA Kits) promoters, induced IFN-beta transcription, and potentiated IFN-beta promoter activation, indicating that LSm14A (show LSM14A ELISA Kits) is a potential signal molecule (show WNT4 ELISA Kits) in the IFN-beta pathway.
Overall, data provide evidence for the possible role of PI3K in the activation of the transcription of IFN-alpha (show IFNA ELISA Kits)/beta by PRRSV; study concludes that PRRSV inhibits the induction of IFN-alpha (show IFNA ELISA Kits) in monocyte-derived dendritic cells by as yet undefined post-transcriptional mechanisms.
suppresses the growth of rat glioma cells
, fibroblast interferon
, interferon beta
, interferon, beta 1
, interferon beta-1
, interferon beta 1
, interferon type B
, interferon, beta 1, fibroblast
, IFN-alpha/beta receptor 2
, interferon alpha/beta receptor 2
, interferon, beta; receptor
, interferon-alpha/beta receptor 2
, interferon-alpha/beta receptor beta chain
, type I interferon receptor 2