Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Mouse (Murine) IL-3 Antibodies:
anti-Rat (Rattus) IL-3 Antibodies:
anti-Human IL-3 Antibodies:
Go to our pre-filtered search.
Mouse (Murine) Monoclonal IL-3 Primary Antibody for Inhibition, Neut - ABIN2689758
Abrams: Immunoenzymetric assay of mouse and human cytokines using NIP-labeled anti-cytokine antibodies. in Current protocols in immunology / edited by John E. Coligan ... [et al.] 2008
Show all 5 Pubmed References
Mouse (Murine) Monoclonal IL-3 Primary Antibody for FACS, Neut - ABIN1177293
Weinstein, Ihle, Lavu, Reddy: Truncation of the c-myb gene by a retroviral integration in an interleukin 3-dependent myeloid leukemia cell line. in Proceedings of the National Academy of Sciences of the United States of America 1986
Show all 4 Pubmed References
Mouse (Murine) Monoclonal IL-3 Primary Antibody for ELISA, WB - ABIN2689759
Abrams, Pearce: Development of rat anti-mouse interleukin 3 monoclonal antibodies which neutralize bioactivity in vitro. in Journal of immunology (Baltimore, Md. : 1950) 1988
Show all 4 Pubmed References
Human Monoclonal IL-3 Primary Antibody for Neut, ELISA - ABIN2689757
Abrams, Roncarolo, Yssel, Andersson, Gleich, Silver: Strategies of anti-cytokine monoclonal antibody development: immunoassay of IL-10 and IL-5 in clinical samples. in Immunological reviews 1992
Show all 2 Pubmed References
Human Polyclonal IL-3 Primary Antibody for ELISA, WB - ABIN1043802
Davila, Froeling, Tan, Bonnard, Boland, Snippe, Hibberd, Seielstad: New genetic associations detected in a host response study to hepatitis B vaccine. in Genes and immunity 2010
findings indicate that STAT5 (show STAT5A Antibodies) contributes to the remarkable IL-3-mediated inhibition of RANKL (show TNFSF11 Antibodies)-induced osteoclastogenesis by activating Id genes and their associated pathways.
c-Kit (show KIT Antibodies)(+) Adipose tissue-derived mesenchymal stem cells (ASCs)may promote breast cancer growth and angiogenesis by a synergistic effect of c-Kit (show KIT Antibodies) and IL-3. Our findings suggest that c-Kit (show KIT Antibodies)(+) subpopulations of ASCs should be eliminated in fat grafts for breast reconstruction of cancer patients following mastectomy.
These impaired macrophage functions in leukemic mice were significantly corrected by IL-3 and GM-CSF (show CSF2 Antibodies) treatment indicating the therapeutic benefit of these two cytokines in leukemia.
IL-3 signaling does not contribute to Jak2 (show JAK2 Antibodies) V617F myeloproliferative neoplasm pathogenesis.
Thus, IL-3 plays an important role in the pathogenesis of SLE and the progression of lupus nephritis.
Cytoplasmic granule containing HERMES (show CD44 Antibodies), NonO (show NONO Antibodies), PSF, and G3BP1 (show G3BP1 Antibodies) is a neuronal RNA-protein granule that is transported in neurites during retinal differentiation.
Stem cell factor (SCF (show KITLG Antibodies)), but not interleukin-3 (IL-3), is a major effector of HSC (show FUT1 Antibodies) maturation during embryonic day E9-E10.
study reports IL3 potentiates inflammation in sepsis; in model of abdominal sepsis, findings show innate response activator B cells produce IL3, which induces myelopoiesis of monocytes and neutrophils and fuels cytokine storm; IL3 deficiency protects against sepsis
these findings reveal a role for CSF-1 (show CSF1 Antibodies) in mediating the IL-3 hematopoietic pathway through monopoiesis, which regulates expansion of CD11c (show ITGAX Antibodies)+ macrophages.
Altered expression of CD30L (show TNFSF8 Antibodies) and IL-3 may be potential biomarkers for hepatotoxicity induced by D. bulbifera.
The present study demonstrates for the first time that IL-3 has an important role in enhancing the migration of human MSCs through regulation of the CXCR4 (show CXCR4 Antibodies)/SDF-1alpha axis. These findings suggest a potential role of IL-3 in improving the efficacy of MSCs in regenerative cell therapy.
These results indicate that IL-3 regulates endothelial cells-extracellular vesicles release, cargo and IL-3 angiogenic paracrine action via STAT5 (show STAT5A Antibodies).
T-GM-CSF (show CSF2 Antibodies) and -IL-3 significantly, and reciprocally, blunted receptor binding and myeloid progenitor cell proliferation activity of both FL-GM-CSF (show CSF2 Antibodies) and -IL-3 in vitro and in vivo
Results show that IL-3 induces several signaling pathways associated with increased cell survival under oxidative stress. This activity correlates with previous fi ndings indicating glucose uptake stimulation by IL-3, which together contribute to an emerging picture of a broader mechanism promoting cell survival.
The IL-3/ GM-CSF (show CSF2 Antibodies) effected on the myofibroblastic differentiation of human adipose derive stromal cells (hASCs) as well as it did on human dermal fibroblasts (HDFs).
Genetically engineered mesenchymal stromal cells produce IL-3 and TPO (show THPO Antibodies) to further improve human scaffold-based xenograft models
IL-3 hardly down-regulates the alpha-chain (show FCGRT Antibodies) of its receptor without depleting the common beta-chain (show CSF2RB Antibodies), which enables extraordinarily sustained signaling events, predominantly the activation of Stat5 (show STAT5A Antibodies).
genetic polymorphisms in the immune genes IL-3 rs181781 and CTLA4 (show CTLA4 Antibodies) rs4553808 may influence the TAC (show IL2RA Antibodies) dose-adjusted concentrations
Our results indicated that the IL-3 and IL-13 (show IL13 Antibodies) polymorphisms were not associated with rheumatoid arthritis (RA). stratification analyses suggested that the IL-13 (show IL13 Antibodies) rs1800925 CT and CT/CC genotypes increased the risk of RA in patients with erythrocyte sedimentation rate (ESR (show ESR1 Antibodies)) <25.00. these findings suggest that the IL-13 (show IL13 Antibodies) rs1800925 C/T polymorphism may be associated with increased risk of RA in ESR (show ESR1 Antibodies)
results suggest that IL-3 and IL-12p40 could be considered as molecular predictors for recurrent wheezing due to RSV infection
cytokine that functions as a multi-lineage haemopoietic growth regulator
, hematopoietic growth factor
, mast cell growth factor
, multipotential colony-stimulating factor
, P-cell stimulating factor
, mast-cell growth factor
, multilineage-colony-stimulating factor
, colony-stimulating factor, multiple
, Hematopoietic growth factor
, Mast cell growth factor
, Multipotential colony-stimulating factor