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Human IL7 ELISA Kit for Sandwich ELISA - ABIN625028
Meltzer, Standley: Modeled repetitive motion strain and indirect osteopathic manipulative techniques in regulation of human fibroblast proliferation and interleukin secretion. in The Journal of the American Osteopathic Association 2008
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Mouse (Murine) IL7 ELISA Kit for Sandwich ELISA - ABIN625152
Jung, Han, Kim, Kim, Lee, Seo, Youn, Lee: Stimulatory effect of HGF-overexpressing adipose tissue-derived mesenchymal stem cells on thymus regeneration in a rat thymus involution model. in Cell biology international 2014
Rat (Rattus) IL7 ELISA Kit for Sandwich ELISA - ABIN1115680
Dai, Wu, Li, Chen, Chen, Hu, Shen: Effects and mechanisms of Geniposide on rats with adjuvant arthritis. in International immunopharmacology 2014
increased IL-7 protein production in situ (lung tissue) from non-human primates who received rBCG vaccination, which was associated with increased survival.
we demonstrated that Imatinib mesylate (IM)impaired T cell survival through the inhibition of IL-7 and STAT5 (show STAT5A ELISA Kits)-p but not TCR signaling which remained unaffected during IM therapy. Thus, off-target inhibitory effects of IM on IL-7 and STAT5 (show STAT5A ELISA Kits)-p explain how T cell lymphopenia occurs in patients treated with IM.
In this study, authors compared the concentrations of IL-15 (show IL15 ELISA Kits) and IL-7 in the plasma of MDS (show PAFAH1B1 ELISA Kits) patients (n = 20) compared with that in the plasma of healthy controls (n = 20).
Data strongly implicate IL-7 in the thymus-independent long-term survival of functional naive-Tregs.
the critical role played by IL-7 and IL-15 in T cell homeostasis and how these cytokines could be used to improve immune reconstitution after allogeneic SCT.
IL-7/IL-7R signaling pathway plays a possible role in recurrent pregnancy loss by upregulating Th17 immunity while downregulating Treg immunity.
IL-7 inhibits the osteogenic differentiation of Periodontal ligament stem cells probably via inactivation of mitogen-activated protein kinase (show MAPK1 ELISA Kits) (MAPK (show MAPK1 ELISA Kits)) signaling pathway.
this study shows that IL-7 restores T Lymphocyte immunometabolic failure in septic shock patients through mTOR (show FRAP1 ELISA Kits) activation
In CRC (show CALR ELISA Kits), IL-7 was higher in patients with lymph node and distant metastases and with tumors located in right colon. In adenomas, IL-7 elevation was associated exclusively with villous growth pattern, while in IBD, circulating IL-7 reflected clinical activity of Crohn's disease and ulcerative colitis.
Tuberculosis patients had lower soluble IL-7R and higher IL-7 plasma concentrations as compared to healthy contacts.
CD56 (show NCAM1 ELISA Kits)(bright) NK IL-7Ralpha (show IL7R ELISA Kits) expression negatively associates with HCV level, and IL-7-induced NK function is impaired during HCV and HIV infections
the in vivo biological role of m(6)A modification in T-cell-mediated pathogenesis and reveals a novel mechanism of T cell homeostasis and Il-7 signal-dependent induction of mRNA degradation
Our new compelling findings have established the critical roles of both IL-7 and IL-15 (show IL15 ELISA Kits) in the maintenance of memory Th17 cells, and the previously undescribed therapeutic advantages of targeting IL-7 and IL-15 (show IL15 ELISA Kits) further support a promising clinical translation in Th17 cell-mediated autoimmune and inflammatory disorders.
Taken together, these data suggest that CCR7 (show CCR7 ELISA Kits) biases memory CD8 (show CD8A ELISA Kits) T cells toward IL-7-dependent niches over IL-15 (show IL15 ELISA Kits)-dependent niches, which provides insight into the homeostatic regulation of different memory T-cell subsets.
this study shows that IL-7 homeostasis is achieved through consumption by multiple subsets of innate and adaptive immune cells
lymphatic vessel expansion occurs in two distinct phases; the first wave of expansion is dependent on IL-7; the second phase, responsible for leukocyte exit from the glands, is regulated by lymphotoxin (show LTB ELISA Kits) (LT)betaR signaling
DNM2 (show DNM2 ELISA Kits) mutations cooperate with Lmo2 (show LMO2 ELISA Kits) T-cell oncogenes by enhancing IL-7 signalling.
IL-7 responsiveness in RTE is designed to maximize survival at the expense of reduced proliferation, consistent with RTE serving as a subpopulation of T cells rich in diversity but not in frequency.
Expression of IL-7 is beneficial for induction of potent and long-lasting humoral immune responses.
findings support a redundant role for adaptive Th17 cell- and innate gammadeltaT17 cell-derived IL17 (show IL17A ELISA Kits) in bacteria-induced colon carcinogenesis, stressing the importance of therapeutically targeting the cytokine itself rather than its cellular sources
The protein encoded by this gene is a cytokine important for B and T cell development. This cytokine and the hepatocyte growth factor (HGF) form a heterodimer that functions as a pre-pro-B cell growth-stimulating factor. This cytokine is found to be a cofactor for V(D)J rearrangement of the T cell receptor beta (TCRB) during early T cell development. This cytokine can be produced locally by intestinal epithelial and epithelial goblet cells, and may serve as a regulatory factor for intestinal mucosal lymphocytes. Knockout studies in mice suggested that this cytokine plays an essential role in lymphoid cell survival. Alternative splicing results in multiple transcript variants encoding distinct isoforms. Additional splice variants have been described but their presence in normal tissues has not been confirmed.