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The Khc73 stalk/14-3-3 (show YWHAZ Antibodies)/NudE pathway defines a physical connection that coordinates the activities of multiple motor proteins to precisely position the spindle.
role of 14-3-3epsilon in germ cell migration
14-3-3epsilon acts as a biochemical control point for axon guidance in Drosophila, silencing Plexin A repulsive axon guidance and regulating a semaphorin repulsion/integrin adhesion switch.
We conclude that 14-3-3epsilon is required for Rab11 (show RAB11A Antibodies)-positive vesicle function, which in turn enables antimicrobial peptide (show cAMP Antibodies) secretion during an innate immune response.
Hpo (show GFER Antibodies) signaling inhibited Yki (show YAP1 Antibodies) nuclear localization and activity by phosphorylating Yki (show YAP1 Antibodies) and both isoforms of 14-3-3 (show YWHAZ Antibodies), 14-3-3varepsilon and 14-3-3zeta (show YWHAZ Antibodies), regulate Yki (show YAP1 Antibodies) activity through modulating its subcellular localization.
Drosophila 14-3-3/PAR-5 (show YWHAZ Antibodies) is an essential mediator of PAR-1 (show F2R Antibodies) function in axis formation.
PAR-1 (show F2R Antibodies) phosphorylates Bazooka/PAR-3 (show PARD3 Antibodies) on two conserved serines to generate 14-3-3 (show YWHAZ Antibodies) binding sites. This inhibits formation of the Bazooka (show PARD3 Antibodies)/PAR-6 (show PARD6A Antibodies)/aPKC complex by blocking Bazooka (show PARD3 Antibodies) oligomerization and binding to aPKC.
Serves as a central modulator of forkhead transcription factor FoxO (show FOXO Antibodies) activity in the regulation of growth, cell death and longevity in vivo.
The expression of epsilon isoforms of 14-3-3 protein (show YWHAE Antibodies) was identified at substantial levels in the first instar larva, upregulated in the second instar larva, and the highest levels were maintained in the late stage of larva, the pupa, and the adult.
Positively regulates Ras-mediated pathways. Acts downstream or parallel to Raf, but upstream of nuclear factors in Ras signaling. Three mutants have been isolated, that suppress the rough eye phenotype caused by mutated Ras1 (sev-Ras1 v12). Inhibits yki activity by restricting its nuclear localization.
, suppressor of Ras85D 3-9
, 14-3-3 protein
, 14-3-3 protein epsilon
, 14-3-3 epsilon
, 14-3-3 epsilon protein
, tyrosine 3-monooxygenase/tryptophan 5-monooxygenase activation protein epsilon polypeptide