Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Human FOXM1 Antibodies:
anti-Rat (Rattus) FOXM1 Antibodies:
anti-Mouse (Murine) FOXM1 Antibodies:
Go to our pre-filtered search.
Human Polyclonal FOXM1 Primary Antibody for ChIP, ICC - ABIN441006
Zhao, Siu, Jiang, Tam, Ngan, Le, Wong, Wong, Gomes, Bella, Khongkow, Lam, Cheung: Overexpression of forkhead box protein M1 (FOXM1) in ovarian cancer correlates with poor patient survival and contributes to paclitaxel resistance. in PLoS ONE 2014
Show all 2 Pubmed References
Human Polyclonal FOXM1 Primary Antibody for IF (p), IHC (p) - ABIN749138
Liu, Zhang, Mao, Zhang, Zhang: Over-expression of FoxM1 is associated with adverse prognosis and FLT3-ITD in acute myeloid leukemia. in Biochemical and biophysical research communications 2014
Dog (Canine) Polyclonal FOXM1 Primary Antibody for WB - ABIN2781166
Laoukili, Alvarez, Meijer, Stahl, Mohammed, Kleij, Heck, Medema: Activation of FoxM1 during G2 requires cyclin A/Cdk-dependent relief of autorepression by the FoxM1 N-terminal domain. in Molecular and cellular biology 2008
Show all 2 Pubmed References
Study found that FOXM1 is a target of miR (show MLXIP Antibodies)-149. miR (show MLXIP Antibodies)-149 inhibited FOXM1 mRNA and protein expression levels by binding to its 3'-UTR (show UTS2R Antibodies) in non-small cell lung cancer (NSCLC) cells. Moreover, patients with low expression levels of miR (show MLXIP Antibodies)-149 exerted high FOXM1 mRNA levels.
we found that FOXM1 inhibitor attenuated tumorigenesis and radioresistance of glioblastoma (GBM) both in vitro and in vivo. Altogether, BUB1B (show BUB1B Antibodies) promotes tumor proliferation and induces radioresistance in GBM, indicating that BUB1B (show BUB1B Antibodies) could be a potential therapeutic target for GBM.
MYBL2 (show MYBL2 Antibodies) is a key downstream factor of Akt (show AKT1 Antibodies)/FoxM1 signaling to promote progression of human glioma, and could be a new candidate gene for molecular targeting therapy and biomarker for radiotherapy of glioma.
FoxM1 may enhance the invasion and migration of cancer cells, and thus promotes their Epithelialmesenchymal transition, in a mechanism that may involve the regulation of Snai1 (show SNAI1 Antibodies).
FoxM1 promotes glioma progression by enhancing UBE2C (show UBE2C Antibodies) transcription
We found that, compared with the control, the proliferative, migratory and invasive abilities of PC-3 (show PCSK1 Antibodies) cells were decreased after incubation with different concentrations of TMP . The expression of FOXM1 was decreased in TMP-treated PC-3 (show PCSK1 Antibodies) cells
FOXM1 expression could be suppressed by miR (show MLXIP Antibodies)-216b via direct binding to FOXM1 3'-UTR (show UTS2R Antibodies) and miR (show MLXIP Antibodies)-216b could inhibit cell proliferation by regulating FOXM1 related Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) signal pathway. MiR (show MLXIP Antibodies)-216b level is related to prognosis in cervical cancer patients and may serve as a potential prognostic marker.
Results revealed that FoxM1 protein was highly expressed in HSCC tissues and that its high expression was closely associated with HSCC tumor differentiation (P=0.004), tumor size (P=0.002), clinical stage (P=0.001), lymph node metastasis (P=0.002), treatment (P=0.045) and expression of the proliferation marker Ki-67 (show MKI67 Antibodies) (P<0.001).
Findings suggest that FoxM1 promotes the EMT (show ITK Antibodies), invasion and migration of TSCC cells, and cross-talks with c-Met/AKT (show AKT1 Antibodies) signaling to form a positive feedback loop to promote TSCC development.
The suppressive activity of miR216b in glioma, which is largely ascribed to downregulation of FoxM1.
Upregulated ROS (show ROS1 Antibodies) induced by FABP4 (show FABP4 Antibodies) was of significance in activating FoxM1 leading to airway inflammation and epithelial barrier dysfunction.
Interactions between the Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) and the Kras/ERK (show EPHB2 Antibodies)/Foxm1 pathways are essential to restrict SOX9 (show SOX9 Antibodies) expression in basal cells during pulmonary branching morphogenesis
YAP (show YAP1 Antibodies) cooperates with FOXM1 to contribute to chromosome instability in hepatocellular carcinoma.
RCM-1 (show TNNI3 Antibodies) blocked the nuclear localization and increased the proteasomal degradation of Forkhead box M1 (FOXM1), a transcription factor critical for the differentiation of goblet cells from airway progenitor cells.
These data implicate the insulin (show INS Antibodies)-FoxM1/PLK1 (show PLK1 Antibodies)/CENP-A (show CENPA Antibodies) pathway-regulated mitotic cell-cycle progression as an essential component in the beta cell adaptation to delay and/or prevent progression to diabetes.
EGF (show EGF Antibodies) promotes FoxM1 expression through the ERK (show EPHB2 Antibodies) signal pathway
FoxM1 induction in the pulmonary vasculature was inhibited by a p110gamma (show PIK3CG Antibodies)-selective inhibitor and in Pik3cg (show PIK3CG Antibodies)(-/-) mice after LPS (show TLR4 Antibodies) challenge. Defective vascular repair in Pik3cg (show PIK3CG Antibodies)-/- mice results from impaired FoxM1 expression
we suggest that proper regional decidualization and polyploidy development requires FoxM1 signaling downstream of Hoxa10 (show HOXA10 Antibodies) and cyclin D3 (show CCND3 Antibodies).
FOXM1 and CENPF (show CENPF Antibodies) are master regulators of prostate cancer malignancy, and can serve as drug response markers for antineoplastic drugs efficiency.
Both gain-of-function and loss-of-function TP53 (show TP53 Antibodies) mutations contribute to overexpression of FoxM1 in high-grade serous ovarian cancer.
the sequence and expression pattern of FoxM1 (fork head box M1) transcription factor in Xenopus laevis embryos are described
Results suggest that FoxM1 functions to link cell division and neuronal differentiation in early Xenopus embryos.
The protein encoded by this gene is a transcriptional activator involved in cell proliferation. The encoded protein is phosphorylated in M phase and regulates the expression of several cell cycle genes, such as cyclin B1 and cyclin D1. Several transcript variants encoding different isoforms have been found for this gene.
Forkhead, drosophila, homolog-like 16
, HNF-3/fork-head homolog 11
, M-phase phosphoprotein 2
, MPM-2 reactive phosphoprotein 2
, forkhead box protein M1
, forkhead-related protein FKHL16
, hepatocyte nuclear factor 3 forkhead homolog 11
, transcription factor Trident
, winged-helix factor from INS-1 cells
, INS-1 winged helix
, forkhead box M1
, forkhead box protein M1-like
, forkhead homolog 16
, winged-helix transcription factor Trident