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anti-Human KIT Ligand Antibodies:
anti-Mouse (Murine) KIT Ligand Antibodies:
anti-Rat (Rattus) KIT Ligand Antibodies:
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Human Polyclonal KIT Ligand Primary Antibody for ELISA, ICC - ABIN4352192
Reber, Da Silva, Frossard: Stem cell factor and its receptor c-Kit as targets for inflammatory diseases. in European journal of pharmacology 2006
Show all 3 Pubmed References
Human Polyclonal KIT Ligand Primary Antibody for IF (p), IHC (fro) - ABIN669936
Goldstein, Goss, Hatzistergos, Rangel, Seidler, Saur, Hare: Adult c-Kit(+) progenitor cells are necessary for maintenance and regeneration of olfactory neurons. in The Journal of comparative neurology 2014
Show all 3 Pubmed References
Human Polyclonal KIT Ligand Primary Antibody for Func, IHC (p) - ABIN2476604
Sakhaee, Alpern, Poindexter, Pak: Citraturic response to oral citric acid load. in The Journal of urology 1992
Show all 2 Pubmed References
Mouse (Murine) Polyclonal KIT Ligand Primary Antibody for ELISA, WB - ABIN2476606
Chou, Flygare, Lodish: Fetal hepatic progenitors support long-term expansion of hematopoietic stem cells. in Experimental hematology 2013
PAR2 (show F2RL1 Antibodies) plays a direct role in melanogenesis by increasing SCF secretion from keratinocytes.
We did not find any KITLG mutation in two affected individuals with familial progressive hyperpigmentation and hypopigmentation.
results suggest that SCF can accelerate cell homing and the maturation of the pulp-dentin complex in human immature teeth.
SCF is a critical regulatory factor for conceptus development and implantation during pregnancy in pigs.
the critical physiological role of the KIT-ET3 (show EDN3 Antibodies)-NO pathway in fulfilling high demand (exceeding basal level) of endothelium-dependent NO generation for coping with atherosclerosis, pregnancy, and aging, is reported.
results revealed that the Th17-mediated inflammatory environment promotes mast cell accumulation through keratinocyte-derived SCF
Bone marrow adipocytes synthesize SCF, promoting hematopoietic stem cell proliferation/regeneration.
Hypoxia microenvironment in hepatocellular carcinoma up-regulates HIF2 (show EPAS1 Antibodies)/SCF expression, which in turn promotes angiogenesis and HCC (show FAM126A Antibodies) metastasis.
Insights into the transmembrane helix associations of Kit ligand have been reported.
There were no significant differences in CD334 (show FGFR4 Antibodies)-SCF double-positive FB and CD31 (show HBA1 Antibodies)-SCF double-positive EC levels between the two periapical tissue groups (P > 0.05). CD14 (show NDUFA2 Antibodies)-SCF double-positive MP density was considerably higher in periapical granulomas than in cysts (P < 0.01).
In vitro, SCF induced the phosphorylation of p38 MAPK (show MAPK14 Antibodies) and cofilin (show CFL1 Antibodies), leading to the migration of cardiac stem cells.
MAPK3 (show MAPK3 Antibodies)/1 participates in primordial follicle activation through mTORC1-KITL signaling.
MicroRNA-205 maintains T cell development following stress by regulating Foxn1 (show FOXN2 Antibodies) and its two regulated targets, stem cell factor and ccl25 (show CCL25 Antibodies), following stress.
These findings indicate the SCF/Kit signaling insufficiency may contribute to the underdevelopment of ICCs and intestinal motility dysfunction upon hypoxia exposure.
SCF/c-kit (show KIT Antibodies) signaling may potentiate chronic hypoxia-induced vascular remodeling by modulating ERK (show EPHB2 Antibodies) activation. Inhibition of c-kit (show KIT Antibodies) activity may be a potential strategy to alleviate PH
Kit ligand regulates the subcellular localization of FOXO3 (show FOXO3 Antibodies) in the neonatal mouse ovary.
The results of this study indicate that c-kit (show KIT Antibodies)/KITL signalling also occurs in the human ovary, as established in various animal models, and may involve previously unknown autocrine signalling
study suggests extracellular HMGB1 (show HMGB1 Antibodies) participates in NLRP3 (show NLRP3 Antibodies) inflammasome activity and regulates IL-1beta (show IL1B Antibodies) associated sterile inflammation induced by MWCNT
SCF/c-kit (show KIT Antibodies) signaling was required for TPA (show PLAT Antibodies)-induced migration and differentiation of hair follicle melanocytes.
Oocyte-derived signals interact locally to mediate granulosa and theca cell function. SCF has a role in modulating this local interaction.
little evidence was found for the KITLG gene being linked to variation in colour
FGF7 (show FGF7 Antibodies) may be an important regulator for oocyte growth and its action is mediated via the KIT/KITLG signaling pathway.
Results suggest that the KITLG gene is a strong candidate gene affecting litter size in goats.
This gene encodes the ligand of the tyrosine-kinase receptor encoded by the KIT locus. This ligand is a pleiotropic factor that acts in utero in germ cell and neural cell development, and hematopoiesis, all believed to reflect a role in cell migration. In adults, it functions pleiotropically, while mostly noted for its continued requirement in hematopoiesis. Two transcript variants encoding different isoforms have been found for this gene.
, c-Kit ligand
, familial progressive hyperpigmentation 2
, kit ligand
, mast cell growth factor
, steel factor
, stem cell factor
, C-kit ligand
, Steel factor
, cloud gray
, hematopoietic growth factor KL
, steel factor/kit ligand
, stem cell factor KL-1
, KIT ligand precursor form 1
, KIT ligand precursor form 4
, c-kit ligand
, stem cell factor, CSF
, mast cell growth factor (white heifer disease)
, Mast cell growth factor
, stem cell factor homolog