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Human Polyclonal SOX11 Primary Antibody for IHC, IHC (p) - ABIN4355327
Ek, Dictor, Jerkeman, Jirström, Borrebaeck: Nuclear expression of the non B-cell lineage Sox11 transcription factor identifies mantle cell lymphoma. in Blood 2008
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Guinea Pig Polyclonal SOX11 Primary Antibody for WB - ABIN2777865
Branas, Elliott, Richmond, Culhane, Wiebe: Alcohol consumption, alcohol outlets, and the risk of being assaulted with a gun. in Alcoholism, clinical and experimental research 2009
Show all 3 Pubmed References
Guinea Pig Polyclonal SOX11 Primary Antibody for WB - ABIN2777866
Shim, Kwan, Li, Lefebvre, Sestan: Cis-regulatory control of corticospinal system development and evolution. in Nature 2012
Show all 2 Pubmed References
Both SOX11 and TFE3 (show TFE3 Antibodies) were overexpressed in solid-pseudopapillary neoplasms (SPNs) and may be involved in the pathogenesis.
Studied the association between miR (show MLXIP Antibodies)-211-5p and SOX11 in regards to proliferation, viability, and invasion of human thyroid cancer (TC) cells. Found miR (show MLXIP Antibodies)-211-5p was upregulated and SOX11 was downregulated in TC tissues and cell lines, and found By inhibiting SOX11, miR (show MLXIP Antibodies)-211- 5p suppressed the proliferation and invasion of the TC cells.
REVIEW: addresses the implication of SOX11 overexpression and frequent genetic lesions, cooperating with cyclin D1 (show CCND1 Antibodies) underlying the pathogenesis of mantle cell lymphoma
our work casts new light on the biology of mantle cell lymphoma (MCL (show FH Antibodies)), revealing the role of SOX11 exerting a functional effect through the repression of BCL6 (show BCL6 Antibodies) transcription in MCL (show FH Antibodies) cells
Study showed for the first time that HIG-2 (show HILPDA Antibodies) and SOX11 mutually co-regulate each other, and that HIG-2 (show HILPDA Antibodies) and SOX11 knock-down promote increased proliferation in a non-synergistic manner in primary mantle cell lymphoma cells.
Solid pseudopapillary neoplasms (SPNs) showed positive staining for SRY (show SRY Antibodies)-related high-mobility group box (show SSRP1 Antibodies) 11 (SOX-11), transcription factor E3 (TFE3 (show TFE3 Antibodies)) and beta-catenin (show CTNNB1 Antibodies) on cell blocks.
SOX11 (sex determining region Y (show SRY Antibodies)-box 11) was inversely expressed with miR (show MLXIP Antibodies)-223-3p in ovarian cancer (OC) cell lines and tissue specimens. miR (show MLXIP Antibodies)-223-3p mimic decreased SOX11 expression. Overexpressing SOX11 inhibited ovarian cancer cell proliferation and invasion, which indicated that miR (show MLXIP Antibodies)-223-3p regulated OC cell proliferation and invasion through targeting SOX11 expression.
Our studies demonstrate that SOX11 is a critical regulator of multiple basal-like breast cancer phenotypes, including growth, migration, invasion, and expression of signature basal-like breast cancer genes
findings suggest that SOX11 is a potential biomarker for ductal carcinoma in situ lesions containing cells harbouring distinct biological features that are likely to progress to invasive breast cancer.
SOX11 antigen can be reliably detected in decalcified tissue bone marrow tissue from mantle cell lymphoma patients.
SOX11 represents a useful prognostic marker in mantle cell lymphoma.
Sox11 can induce substantial axon regeneration. Transcriptome profiling indicated that Sox11 activates genes involved in cytoskeletal remodeling and axon growth. Remarkably, alpha-RGCs, which preferentially regenerate following treatments such as Pten deletion, were killed by Sox11 overexpression. Thus, Sox11 promotes regeneration of non-alpha-RGCs, which are refractory to Pten deletion-induced regeneration.
These findings indicate that suppression of dendritic morphogenesis by Sox11 during radial migration is crucial for the formation of the cerebral cortex.
Following elevation of the palatal shelves (E14.5), Sox2 (show SOX2 Antibodies), Sox11 and Sox21 (show SOX21 Antibodies) expression was present in the midline epithelial seam. We thus identify dynamic spatio-temporal expression of Sox (show QSOX1 Antibodies) gene family during the process of palatogenesis
Sox11 directly regulates the expression of Fgf9; ablation of the Sox11 gene results in clefting of the secondary palate resembling the Pierre Robin sequence.
SOXC proteins act cell- and non-cell-autonomously in perichondrocytes and chondrocytes to establish noncanonical WNT (show WNT2 Antibodies) signaling crosstalk essential for growth plate induction and control
Sox11 and Sox4 are not redundant in promoting neuronal differentiation in the cortex, but rather act in overlapping and discrete populations of neurons
Fetal Sox11 disruption lead to hypoplastic lungs, ventricular septation defects, abdominal defects, skeletal malformations, decreased birth weight and postnatal lethality.
These data identify Sox11 as a key transcription factor that can confer an elevated innate regenerative capacity to CNS neurons.
Sox11 expression was high in fetal.
Binds to the DNA sequence 5'-AACAAT-3'. Plays a role together with nlk in neural induction during early embryogenesis (By similarity).
SRY (sex-determining region Y)-box 11
, SRY-related HMG-box gene 11
, transcription factor SOX-11
, SRY-box 11
, Sox11 transcription factor
, transcription factor Sox-11
, SRY (sex determining region Y)-box 11
, SRY (sex determining region Y)-box 11 b
, Transcription factor Sox-13
, transcription factor Sox-11-B
, SRY-box containing gene 11
, LOW QUALITY PROTEIN: transcription factor SOX-11