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Interleukin 17A (IL17A)is a proinflammatory cytokine secreted by activated T-lymphocytes. Additionally we are shipping IL17RA Antibodies (215) and IL17RA Kits (12) and many more products for this protein.
Showing 10 out of 36 products:
Human IL17RA Protein expressed in Human Cells - ABIN2003563
Schwandner, Yamaguchi, Cao: Requirement of tumor necrosis factor receptor-associated factor (TRAF)6 in interleukin 17 signal transduction. in The Journal of experimental medicine 2000
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Rat (Rattus) IL17RA Protein expressed in Human Cells - ABIN2009276
Takahashi, Vanlaere, de Rycke, Cauwels, Joosten, Lubberts, van den Berg, Libert: IL-17 produced by Paneth cells drives TNF-induced shock. in The Journal of experimental medicine 2008
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Rhesus Monkey IL17RA Protein expressed in Human Cells - ABIN2009892
Toy, Kugler, Wolfson, Vanden Bos, Gurgel, Derry, Tocker, Peschon: Cutting edge: interleukin 17 signals through a heteromeric receptor complex. in Journal of immunology (Baltimore, Md. : 1950) 2006
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Human IL17RA Protein expressed in HEK-293 Cells - ABIN2181343
Puel, Cypowyj, Bustamante, Wright, Liu, Lim, Migaud, Israel, Chrabieh, Audry, Gumbleton, Toulon, Bodemer, El-Baghdadi, Whitters, Paradis, Brooks, Collins, Wolfman, Al-Muhsen, Galicchio, Abel, Picard et al.: Chronic mucocutaneous candidiasis in humans with inborn errors of interleukin-17 immunity. ... in Science (New York, N.Y.) 2011
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It has been shown that IL-17 (show IL17A Proteins) can be produced by enteroendocrine chromogranin A (show CHGA Proteins)-positive cells as well as by goblet cells in the human colon.
Results suggest that IL-17 (show IL17A Proteins)-IL-17R interaction in GSCs induces an autocrine/paracrine cytokine feedback loop, which may provide an important signaling component for maintenance/self-renewal of GSCs.
High IL-17RA expression is associated with Intrahepatic Cholangiocarcinoma.
Identified the IL17RA rs4819554 SNP as a risk factor for psoriasis.
IL-17 receptor A and adenosine deaminase 2 (show CECR1 Proteins) deficiency due to deletion mutations was found in siblings with chronic mucocutaneous candidiasis and chronic systemic inflammation.
In vitro, CSE stimulation significantly increased IL-17F and IL-17R in 16HBE (2.5%) and A549 (5%) while IL-17A and IL-17F in PBMC (10%). IL-17A and CSE stimulation, rather than CSE or rhIL-17A alone, increased proliferation in 16HBE and apoptosis in A549
The data indicate that the genetic variants of rs2275913 in IL-17A, rs763780 in IL-17F, and rs4819554 in IL-17RA may not play a role in the pathogenesis of preeclampsia in Chinese Han women.
IL17F (show IL17F Proteins) and IL17RA polymorphisms modulate susceptibility to cerebral malaria(CM) and provide evidence that IL-17F (show IL17F Proteins) protects against CM.
Increased expression of IL-17RA plays an important role in gastric cancer progression, migration and prognosis.
Both genetic variants of IL-17R (rs882643 and rs2241049) were associated with PGD (show PGD Proteins). This confirms a genetic predisposition toward PGD (show PGD Proteins) and suggests a role of IL-17 (show IL17A Proteins) in driving neutrophilia in PGD (show PGD Proteins).
These data show that IL-17R signaling in the lung epithelium plays a critical role in establishing chemokine (show CCL1 Proteins) gradients that are essential for mucosal immunity against pulmonary bacterial pathogens.
Thus, oral epithelial cells dominantly control IL-17R-dependent responses to oropharyngeal candidiasis through regulation of BD3 (show DEFB103A Proteins) expression.
this study showsthat the nature of the protective memory response depends closely on the route of infection and highlights the role of IFN-gamma (show IFNG Proteins)-and IL-17RA-mediated responses in the control of mucosal infection by Brucella
IL-17RA contributes to experimental glomerulonephritis, with IL-17RA expression on both leukocytes and stromal cells being required for the full expression of nephritogenic humoral immunity.
IL-17RA in non hematopoietic cells regulates recruitment of neutrophils to the lung during the adaptive phase of the immune response.
Bronchial epithelial TLR2/IL17R signaling crosstalk is necessary for lung IL17 (show IL17A Proteins) homeostasis and is compromised by morphine.
IL-17 (show IL17A Proteins) signaling through IL-17RA is an important driver of lung inflammation and airway hyperresponsiveness.
results suggest that the p38alpha (show MAPK14 Proteins)-MKP-1 (show DUSP1 Proteins) signaling axis links IL-17R signaling in tissue-resident cells to autoimmune inflammation dependent on infiltrating T(H)17 cells.
There were no significant differences in clinical scores and numbers of infiltrating eosinophils among IL-25KO, TSLPR (show CRLF2 Proteins) KO, and wild-type mice.
IL-17RA signaling is critical for the development of renal pathology in autoimmune glomerulonephritis.IL-17 enhanced the production of proinflammatory cytokines and chemokines from tubular epithelial cells.
Interleukin 17A (IL17A)is a proinflammatory cytokine secreted by activated T-lymphocytes. It is a potent inducer of the maturation of CD34-positive hematopoietic precursors into neutrophils. The protein encoded by this gene (interleukin 17A receptor\; IL17RA) is a ubiquitous type I membrane glycoprotein that binds with low affinity to interleukin 17A. Interleukin 17A and its receptor play a pathogenic role in many inflammatory and autoimmune diseases such as rheumatoid arthritis. Like other cytokine receptors, this receptor likely has a multimeric structure.
IL-17 receptor A
, interleukin-17 receptor A