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Human PPARG ELISA Kit for Sandwich ELISA - ABIN366589
Mirzaei, Hossein-Nezhad, Keshavarz, Koohdani, Saboor-Yaraghi, Hosseini, Eshraghian, Djalali: Crosstalk between circulating peroxisome proliferator-activated receptor gamma, adipokines and metabolic syndrome in obese subjects. in Diabetology & metabolic syndrome 2013
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Mouse (Murine) PPARG ELISA Kit for Sandwich ELISA - ABIN367893
Feng, Hai-ning, Xiao-man, Zun-chen, Sheng-rong, Das: Effect of yellow capsicum extract on proliferation and differentiation of 3T3-L1 preadipocytes. in Nutrition (Burbank, Los Angeles County, Calif.) 2014
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Human PPARG ELISA Kit for Sandwich ELISA - ABIN414461
Szalardy, Zadori, Tanczos, Simu, Bencsik, Vecsei, Klivenyi: Elevated levels of PPAR-gamma in the cerebrospinal fluid of patients with multiple sclerosis. in Neuroscience letters 2013
Mouse (Murine) PPARG ELISA Kit for Sandwich ELISA - ABIN415421
Rohm, Holik, Kretschy, Somoza, Ley, Widder, Krammer, Marko, Somoza: Nonivamide enhances miRNA let-7d expression and decreases adipogenesis PPAR? expression in 3T3-L1 cells. in Journal of cellular biochemistry 2015
Rat (Rattus) PPARG ELISA Kit for Sandwich ELISA - ABIN416144
Chandru, Vishwanath, Devegowda, Ramasamudra, Prashant, Hathur: Evaluation of Protein Kinase Cβ and PPARγ Activity in Diabetic Rats Supplemented with Momordica charantia. in Journal of clinical and diagnostic research : JCDR 2016
Rabbit PPARG ELISA Kit for Sandwich ELISA - ABIN431338
Li, Zhang, Chen, Zhang, Ma, Xia: Establishment of a rabbit model to study the influence of advanced glycation end products accumulation on osteoarthritis and the protective effect of pioglitazone. in Osteoarthritis and cartilage / OARS, Osteoarthritis Research Society 2015
Rat (Rattus) PPARG ELISA Kit for Sandwich ELISA - ABIN368258
Sangeetha, ShriShri Mal, Atmaja, Sali, Vasanthi: PPAR's and Diosgenin a chemico biological insight in NIDDM. in Chemico-biological interactions 2013
Time course analysis demonstrated that the adipogenic 'hub', sampled by PPARgamma and Lpin1 (show LPIN1 ELISA Kits), undergoes orchestrated reorganization during adipogenesis.
Aleglitazar protects cardiomyocytes against hyperglycaemia-induced apoptosis by combined activation of both peroxisome proliferator-activated receptor-alpha (show PPARA ELISA Kits) and peroxisome proliferator-activated receptor-gamma.
IRF6 (show IRF6 ELISA Kits) suppresses PPARgamma through binding IRF (show TRIM63 ELISA Kits) recognition sites located upstream of the PPARgamma coding region. Taken together, the results suggest that an IRF6 (show IRF6 ELISA Kits)/PPARgamma regulatory axis suppresses anti-inflammatory responses in bone marrow-derived macrophages and provides references for future study addressing dysregulated metabolic and immunologic homeostasis of obese adipose tissue.
NFIA (show NFIA ELISA Kits) activates the cell-type-specific enhancers and facilitates the binding of PPARgamma to control the brown fat gene program.
adipogenic miR (show MLXIP ELISA Kits)-27a in adipose tissue upregulates macrophage activation via inhibiting PPARgamma of insulin (show INS ELISA Kits) resistance induced by high-fat diet-associated obesity
Report shows the identification of a novel Pparg splicing variant, Pparc1sv, in mice that is synergistically upregulated with Pparc2 during adipocyte differentiation of 3T3-L1 cells and mouse primary cultured preadipocytes. Both promotors are activated by C/EBPbeta (show CEBPB ELISA Kits) and C/EBPdelta (show CEBPD ELISA Kits).
High-fat diet-induced obesity reduces vascular PPAR-gamma activity. Reduced PPAR-gamma exaggerates oxidative stress and inflammation in response to hypoxia.
Low expression of PPARG is associated with squamous cell lung carcinoma.
Since the R6/2 mice represent a 'truncated' huntingtin (Htt (show HTT ELISA Kits)) mouse model of Huntington's disease (HD), we tested the efficacy of bezafibrate in a 'full-length' Htt (show HTT ELISA Kits) mouse model, the BACHD mice. Bezafibrate treatment restored the impaired PPARg, PPARd (show PPARD ELISA Kits), PGC (show PGC ELISA Kits)-1a signaling pathway, enhanced mitochondrial biogenesis and improved antioxidant defense in the striatum of BACHD mice
Tfe3 (show TFE3 ELISA Kits) and Tfeb are required for the induced expression of Ppargamma2 and subsequently for adipogenic genes.
COX-2 (show COX2 ELISA Kits) is a potential marker for identifying high-risk sebaceous gland carcinoma (SGC (show SGCB ELISA Kits)) patients. Expression of PPAR-gamma in eyelid SGC (show SGCB ELISA Kits) cases reflects terminal sebaceous differentiation.
PAK4 (show PAK4 ELISA Kits) downregulation decreased PPARgamma-mediated Nox1 (show NOX1 ELISA Kits) expression and suppressed EMT (show ITK ELISA Kits) in IR-treated glioma cells.
a positive association of placental PPARgamma mRNA levels and placental DHA levels with baby weight
we found that PPARdelta (show PPARD ELISA Kits) directly regulated neutral amino acid transporter (show SLC6A19 ELISA Kits) SLC1 (show MCHR1 ELISA Kits)-A5 (solute carrier family 1 member 5 (show SLC1A5 ELISA Kits)) and glucose transporter-1 (Glut1 (show SLC2A1 ELISA Kits)) gene transcription, leading to uptake of glucose and amino acid, activation of mTOR (show FRAP1 ELISA Kits) signaling, and tumor progression. In contrast, silence of PPARdelta (show PPARD ELISA Kits) or its antagonist inhibited this event.
The Atgl (show PNPLA2 ELISA Kits) is down-regulated by the basal transcription factor Sp1 (show SP1 ELISA Kits) in preadipocytes and that the magnitude of down-regulation depends on interactions between Sp1 (show PSG1 ELISA Kits) and peroxisome proliferator-activated receptor gamma (PPARgamma).
Altogether, the authors establish the first in vivo human angiomyolipoma model, which provides evidence that angiomyolipoma may originate in a PPARG-activated renal mesenchymal stem cell lineage that is skewed toward adipocytes and smooth muscle and away from osteoblasts, and uncover PPARG as a regulator of angiomyolipoma growth, which could serve as an attractive therapeutic target.
Analysis of the PPARG gene in families with familial partial lipodystrophy
These data demonstrate that complex of PPARgamma with GW1929 is a negative regulator involved in the control of ApoA-I (show APOA1 ELISA Kits) expression and secretion in human hepatocyte- and enterocyte-like cells
The Adiponectin (show ADIPOQ ELISA Kits) increases AQP3 (show AQP3 ELISA Kits) expression through PPARalpha (show PPARA ELISA Kits)-mediated signaling inhepatic stellate cells.
The results suggest the higher expression of miR (show MYLIP ELISA Kits)-130a, which targeting peroxisome proliferator-activated receptor gamma, may be the reason for less fat deposition in intramuscular adipose tissue than subcutaneous adipose tissue.
The immunoprecipitation results also showed that high AA concentrations significantly increased the interaction of mTOR (show FRAP1 ELISA Kits) and PPARg. In summary, PPARg plays an important role in the regulation of IGF-1 (show IGF1 ELISA Kits) secretion and gene expression in response to dietary protein.
The regulatory role of microRNA miR (show MYLIP ELISA Kits)-27b-3p on peroxisome proliferator-activated receptor-gamma (PPARgamma) was confirmed by their inversed expression patterns in oocytes: [miR (show MYLIP ELISA Kits)-27b-3p]
an Enhancer box and a binding site for a cooperative co-activator of MyoD (show MYOD1 ELISA Kits) are present in the promoter region of porcine PPARgamma.
The data suggest that there is local cooperation between resistin (show RETN ELISA Kits) and PPARgamma expression in the porcine ovary. Resistin (show RETN ELISA Kits) significantly increased the expression of PPARgamma, whereas PPARgamma decreased resistin (show RETN ELISA Kits) expression; thus, PPARgamma is a new key regulator of resistin (show RETN ELISA Kits) expression and function.
Therefore, this study demonstrated that the different regulatory adipogenic roles of MSTN (show MSTN ELISA Kits) in ADSCs and MSCs act by differentially regulating PPARgamma and MyoD (show MYOD1 ELISA Kits) expression.
PGRN (show GRN ELISA Kits) inhibits adipogenesis in porcine preadipocytes partially through ERK (show MAPK1 ELISA Kits) activation mediated PPARgamma phosphorylation.
Resveratrol activated sirtuin 1 (Sirt1 (show SIRT1 ELISA Kits)) gene expression and increased adipose triglyceride lipase (ATGL (show PNPLA2 ELISA Kits)) gene expression and glycerol release. Furthermore, this study found the opposite Sirt1 (show SIRT1 ELISA Kits) regulation pattern for PPARgamma to that of ATGL (show PNPLA2 ELISA Kits) in adipocytes.
The results indicate that the endometrial expression of PPARgamma genes fluctuates during the estrous cycle and pregnancy.
Increasing dietary DE linearly enhanced the expression of PPARgamma in adipose tissues but not skeletal muscle of Rongchang piglets.
PPARgamma is a positive regulator of milk fat synthesis in dairy cow mammary epithelial cells.
An Asp7Gly substitution in PPARG is associated with adiposity.
upregulation of PPARgamma was observed in the backfat tissue of Lilu cattle with increasing age
Co-culture of adipocytes and myoblasts elicited an increase in C/EBPbeta (show CEBPB ELISA Kits) and PPAR-gamma gene expression in differentiated myoblasts and an increase in GPR43 (show FFAR2 ELISA Kits) gene expression in adipocytes.
A potential association of an single nucleotide polymorphism (72472 GT in exon7) of the bovine PPAR-gamma gene with carcass and meat quality traits, was evaluated.
study demonstrates the co-expression of DLX3 (show DLX3 ELISA Kits), PPARG and SP1 (show SP1 ELISA Kits) in trophoblast binucleated cell(BNC)nuclei; this suggests a possible role of these transcription factors through BNC specific genes at the time of pre-placental differentiation
Single nucleotide polymorphisms in coding region of the PPARgamma gene, were examined.
oxidative stress attenuates PPAR gamma expression and activity in vascular endothelial cells
The effects of dietary fat components on the expression of PPAR-gamma AND PPAR-gamma coactivator 1 (show PPARGC1A ELISA Kits) in cultured bovine preadipocytes are reported.
Intramammary infection elicited a strong transcriptomic response, leading to potent activation of pro-inflammatory pathways that were associated with a marked inhibition of lipid synthesis, stress-activated kinase signaling cascades, and PPAR (show PPARA ELISA Kits) signaling.
study found PPAR-gamma expression was prominent in the subthalamic nucleus, oculomotor nucleus, ventral tegmental nucleus, and to a lesser extent, in the putamen; 3 or 12 months after MPTP (show PTPN2 ELISA Kits), only the lesioned putamen had increased PPAR-gamma
Aleglitazar, a dual PPARalpha (show PPARA ELISA Kits)/gamma agonist, has beneficial effects on both lipid and glucose parameters in a primate model of the metabolic syndrome.
siRNA targeting PPARgamma gene can inhibit adipogenic differentiation of BMSCs and prevent steroid-induced osteonecrosis in rabbit.
vitamin E supplementation affords protection by decreasing MMP-1 (show MMP1 ELISA Kits) and increasing PPARg, GSTa (show GSTa2 ELISA Kits), and ABCA1 (show ABCA1 ELISA Kits) levels in aortae of rabbits fed a cholesterol-rich diet
Telmisartan improves microvascular dysfunction during myocardial ischemia/reperfusion injury via the PPARgamma pathway.
PPARgamma plays a luteotropic role in pseudopregnant rabbits, through PTGS2 (show PTGS2 ELISA Kits) down-regulation and 3beta-HSD (show HAL ELISA Kits) up-regulation, with a consequent PGF2alpha decrease and progesterone increase.
In an animal model of atherosclerosis, the expression of PPAR-gamma is upregulated following atorvastatin administration.
Tongxinluo can inhibit the expression of MMP-3 (show MMP3 ELISA Kits) and 9 and increase the expression of PPARgamma in atherosclerotic rabbits.
Niacin Reduces serum level and adipose mRNA expression of leptin (show LEP ELISA Kits) and up-regulates PPARgamma and CD36 (show CD36 ELISA Kits) mRNA expression in hypercholesterolemic rabbits.
Antidiabetic drug pioglitazone protects the heart via activation of PPAR-gamma receptors, PI3-kinase (show PIK3CA ELISA Kits), Akt (show AKT1 ELISA Kits), and eNOS (show NOS3 ELISA Kits) pathway in a rabbit model of myocardial infarction.
This gene encodes a member of the peroxisome proliferator-activated receptor (PPAR) subfamily of nuclear receptors. PPARs form heterodimers with retinoid X receptors (RXRs) and these heterodimers regulate transcription of various genes. Three subtypes of PPARs are known: PPAR-alpha, PPAR-delta, and PPAR-gamma. The protein encoded by this gene is PPAR-gamma and is a regulator of adipocyte differentiation. Additionally, PPAR-gamma has been implicated in the pathology of numerous diseases including obesity, diabetes, atherosclerosis and cancer. Alternatively spliced transcript variants that encode different isoforms have been described.
peroxisome proliferator activated receptor gamma
, peroxisome proliferative activated receptor gamma
, peroxisome proliferator-activated receptor gamma
, peroxisome proliferator activator receptor gamma
, nuclear receptor subfamily 1 group C member 3
, peroxisome proliferator activated receptor gamma 2
, peroxisome proliferator activated receptor gamma 4
, Nuclear receptor subfamily 1 group C member 3
, PPAR gamma
, peroxisome proliferator-activated nuclear receptor gamma variant 1
, peroxisome proliferator-activated receptor gamma 1
, peroxisome proliferator activator receptor, gamma
, PPAR gamma 2
, xPPAR gamma
, peroxisome proliferative activated receptor, gamma
, peroxisome proliferator-activated receptor gamma 2
, peroxisome proliferator-activated receptor gamma 1-a
, peroxisome proliferator-activated receptor gamma 1-b
, PPAR gamma 1
, peroxisome proliferator activated receptor gamma-1
, peroxisome proliferator-activated receptor-gamma