ADAM Metallopeptidase with thrombospondin Type 1 Motif, 9 (ADAMTS9) ELISA Kits

ADAMTS9 encodes a member of the ADAMTS (a disintegrin and metalloproteinase with thrombospondin motifs) protein family. Additionally we are shipping ADAMTS9 Antibodies (31) and and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
ADAMTS9 101401  
ADAMTS9 56999 Q9P2N4
ADAMTS9 312566  
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Top ADAMTS9 ELISA Kits at antibodies-online.com

Showing 10 out of 17 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Delivery Price Details
Human 0.116 ng/mL 0.31 ng/mL - 20 ng/mL 96 Tests 13 to 16 Days
$736.84
Details
Rabbit 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15 to 18 Days
$707.14
Details
Guinea Pig 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15 to 18 Days
$707.14
Details
Rat 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15 to 18 Days
$707.14
Details
Mouse 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15 to 18 Days
$707.14
Details
Monkey 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15 to 18 Days
$707.14
Details
Dog 0.1 ng/mL 0.5-10 ng/mL   96 Tests 15 to 18 Days
$707.14
Details
Human < 0.116 ng/mL 0.312 ng/mL - 20 ng/mL   96 Tests 11 to 18 Days
$902.56
Details
Pig
  96 Tests 15 to 18 Days
$707.14
Details
Sheep
  96 Tests 15 to 18 Days
$707.14
Details

Top referenced ADAMTS9 ELISA Kits

  1. Human ADAMTS9 ELISA Kit for Sandwich ELISA - ABIN1118181 : Tajima, Sekimoto, Yamaguchi, Taniguchi, Kurogi, Maruyama, Chosa: Hemoglobin stimulates the expression of ADAMTS-5 and ADAMTS-9 by synovial cells: a possible cause of articular cartilage damage after intra-articular hemorrhage. in BMC musculoskeletal disorders 2017 (PubMed)

More ELISA Kits for ADAMTS9 Interaction Partners

Mouse (Murine) ADAM Metallopeptidase with thrombospondin Type 1 Motif, 9 (ADAMTS9) interaction partners

  1. ADAMTS9 Regulates Skeletal Muscle Insulin Sensitivity Through Extracellular Matrix Alterations.

  2. ADAMTS9 or ADAMTS20 mutagenesis impaired neural and yolk sac ciliogenesis, leading to morphogenetic anomalies resulting from impaired hedgehog signaling, which is transduced by primary cilia.

  3. there is a melanocyte cell-autonomous requirement for Adamts9.

  4. Extracellular matrix dynamics is a major influence on umbilical vascular SMC fate, with ADAMTS9 acting as its principal mediator.

  5. The present study reveals ADAMT-9 expression by mast cells(MCs) and that MC activation regulates the expression of the protease, thus implicating the ADAMT-9 of protease in MC function.

  6. Western blot analyses indicated that aggrecanase-generated proteoglycan fragments are produced after SCI.

  7. These findings support a model in which cooperative versican proteolysis by ADAMTS9 in vascular endothelium and by ADAMTS20 in palate mesenchyme drives palatal shelf sculpting and extension.

  8. These data identify ADAMTS9 as a novel, constitutive, endogenous angiogenesis inhibitor that operates cell-autonomously in endothelial cells.

  9. ancillary domains of ADAMTS-9 are required both for specific extracellular localization and for its versicanase and aggrecanase activities

  10. Adamts9 is widely expressed during mouse embryo development

  11. show that combinatorial mouse alleles for the secreted metalloproteases Adamts5, Adamts20 (bt), and Adamts9 result in fully penetrant soft-tissue syndactyly.

Human ADAM Metallopeptidase with thrombospondin Type 1 Motif, 9 (ADAMTS9) interaction partners

  1. findings suggest that the identified mutations in ADAMTS9 cause nephronophthisis-related ciliopathies and that ADAMTS9 is required for the formation and function of primary cilia

  2. ADAMTS9, a secreted metalloprotease, is required for myometrial activation during late gestation and for parturition. Through knockdown of ADAMTS9 in uterine SMC, and manipulation of pericellular versican via knockdown or proteolysis, regulated pericellular matrix dynamics is essential for focal adhesion maintenance.

  3. ADAMTS9 Regulates Skeletal Muscle Insulin Sensitivity Through Extracellular Matrix Alterations.

  4. Low ADAMTS9 expression is associated with non-alcoholic fatty liver disease.

  5. ADAMTS9 expression and methylation was analysed in breast cancer cell lines and tissue samples.

  6. CRISPR-Cas9 inactivation of ADAMTS9 impaired ciliogenesis in RPE-1 cells, which was restored by catalytically active ADAMTS9 or ADAMTS20 acting in trans, but not by their proteolytically inactive mutants.

  7. Data indicate that miR-32 accelerates progression in HCC by targeting ADAMTS9, and the abnormal expression of miR-32 is correlated with prognosis.

  8. ADAMTS9 gene cytosine-adenine repeat polymorphism may be used to determine the prognosis for knee osteoarthritis radiologic progression.

  9. expression by synovial cells induced by hemoglobin at low doses, suggesting a possible role for hemoglobin in cartilage damage after intra-articular hemorrhage

  10. By inhibiting ADAMTS-9, miR-338-5p suppressed the proliferation and metastasis of rheumatoid arthritis synovial fibroblasts.

  11. Exogenous expression of ADAMTS9 in colorectal cancer cell lines inhibited cell proliferation and migration through the regulation of cell cycle and apoptosis.

  12. Our data showed that four SNPs (rs73832338, rs9985304, rs4317088, and rs9831846) in the ADAMTS9 gene were significantly associated with cognitive aging among the subjects. Additionally, we found that interactions between the ADAMTS9 rs9985304 and ADAMTS9 rs76346246 SNPs influenced cognitive aging.

  13. we identified the top 10 highly differentiated SNPs in Brazilian Amerindian ancestry compared to Asian, European, and African Genomes.SNPs within or proximal to CIITA (rs6498115), SMC6 (rs1834619), and KLHL29 (rs2288697) were most differentiated in the Amerindian-specific branch. SNPs in ADAMTS9 (rs7631391), DOCK2 (rs77594147), SLC28A1 (rs28649017), ARHGAP5 (rs7151991), and CIITA (rs45601437) in the Asian comparison.

  14. study identified a suggestive genome-wide significant association of ADAMTS9 with asthma in Spanish subjects

  15. NF-kappaBp65 bound to elements located at -1177 and -1335 in the ADAMTS9 promoter region, in contrast to the untreated samples. The results of the present study suggested that NF-kappaB may be involved in IL-1beta-induced activation of ADAMTS9 in human chondrocytes

  16. A deletion at ADAMTS9-MAGI1 locus is associated with psoriatic arthritis risk.

  17. LncRNA ADAMTS9-AS2 is regulated by DNMT1 and inhibits migration of glioma cells.

  18. The present study reveals ADAMT-9 expression by mast cells(MCs) and that MC activation regulates the expression of the protease, thus implicating the ADAMT-9 of protease in MC function.

  19. The pathways MAPK and NF-kappaB were responsible pathways for the induction of ADAMTS9 gene.

  20. PPARG2 and ADAMTS9 variants are both associated with type 2 diabetes mellitus and with insulin resistance, whereas only ADAMTS9 may be related to beta cell function.

ADAMTS9 Antigen Profile

Antigen Summary

This gene encodes a member of the ADAMTS (a disintegrin and metalloproteinase with thrombospondin motifs) protein family. Members of the family share several distinct protein modules, including a propeptide region, a metalloproteinase domain, a disintegrin-like domain, and a thrombospondin type 1 (TS) motif. Individual members of this family differ in the number of C-terminal TS motifs, and some have unique C-terminal domains. Members of the ADAMTS family have been implicated in the cleavage of proteoglycans, the control of organ shape during development, and the inhibition of angiogenesis. This gene is localized to chromosome 3p14.3-p14.2, an area known to be lost in hereditary renal tumors.

Gene names and symbols associated with ADAMTS9

  • ADAM metallopeptidase with thrombospondin type 1 motif 9 (ADAMTS9) antibody
  • a disintegrin-like and metallopeptidase (reprolysin type) with thrombospondin type 1 motif, 9 (Adamts9) antibody
  • ADAM metallopeptidase with thrombospondin type 1 motif, 9 (Adamts9) antibody
  • 1810011L16Rik antibody
  • 8430403M15Rik antibody
  • AW743315 antibody
  • E030027K14Rik antibody

Protein level used designations for ADAMTS9

a disintegrin-like and metalloprotease (reprolysin type) with thrombospondin type 1 motif, 9 , A disintegrin and metalloproteinase with thrombospondin motifs 9 , ADAM-TS 9 , ADAM-TS9 , ADAMTS-9

GENE ID SPECIES
100049887 Equus caballus
101401 Mus musculus
56999 Homo sapiens
606847 Canis lupus familiaris
100520218 Sus scrofa
537051 Bos taurus
312566 Rattus norvegicus
101119080 Ovis aries
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