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ART4 encodes a protein that contains a mono-ADP-ribosylation (ART) motif. Additionally we are shipping ADP-Ribosyltransferase 4 (Dombrock Blood Group) Proteins (14) and many more products for this protein.
Showing 10 out of 37 products:
our results indicate that miR (show MLXIP Antibodies)-330-5p inhibits non-small-cell lung cancer (NSCLC) cell growth through downregulation of NOB1 (show NOB1 Antibodies) expression. Our study suggests that miR (show MLXIP Antibodies)-330-5p may serve as a potential therapeutic target for the treatment of NSCLC
Authors identified that SNHG1 increased human nin one binding protein (NOB1 (show NOB1 Antibodies)), an oncogene (show RAB1A Antibodies), through sponging miR (show MLXIP Antibodies)-326 as competing endogenous RNA (ceRNA), finally prompting cell growth, migration and invasion in OS.
RIOK2 and NOB1 (show NOB1 Antibodies) were highly expressed in NSCLC cells and tissues, and their expression profiles were significantly associated with the Tumour Node Metastasis (TNM (show ODZ1 Antibodies)) clinical stage, lymph node metastasis, and differentiation. RIOK2 expression was correlated with NOB1 (show NOB1 Antibodies).
The proto-oncogene (show RAB1A Antibodies) NOB1 (show NOB1 Antibodies) as a direct target of miR (show MLXIP Antibodies)-326 in gastric cancer.
The expression of NOB1 (show NOB1 Antibodies) was also found to be higher in multidrug-resistant gastric cancer cells than that of sensitive cells. This novel MAb will be valuable for investigating the role of NOB1 (show NOB1 Antibodies) in carcinogenesis and multidrug resistance of gastric cancer.
CONCLUSION: Our results suggest that enhanced expression of NOB1 (show NOB1 Antibodies) related with poor early response to cisplatin-based chemotherapy in patients with advanced non-small cell lung cancer
NOB1 (show NOB1 Antibodies) plays an oncogenic role in laryngeal cancer cells through the regulation of JNK (show MAPK8 Antibodies) signaling pathway.
miR (show MLXIP Antibodies)-139-3p may act as a tumor suppressor that can inhibitcervical cancer cell proliferation, migration and invasion and induce cell apoptosis through down-regulation of NOB1 (show NOB1 Antibodies) expression.
we suggest that targeting miR (show MLXIP Antibodies)-192 and NOB1 (show NOB1 Antibodies) is a novel strategy which will assist in the development of new therapeutics that will be used in the future to prevent and treat prostate cancer.
The results suggested that NOB1 (show NOB1 Antibodies) is important in OSCC development and serves as a candidate indicator of aggressiveness and a therapeutic target of oral squamous cell carcinoma.
findings demonstrate that Dok-3 (show DOK3 Antibodies) and Dok-1 (show DOK1 Antibodies)/-2 play distinctive but cooperative roles in osteoclastogenesis and protect mice from osteopenia, providing physiological and pathophysiological insight into bone homeostasis.
Platelets from Dok-1 (show DOK1 Antibodies)-/- mice displayed normal aggregation, activation of integrin alphaIIbbeta3, P-selectin (show SELP Antibodies) surface expression, and soluble fibrinogen binding. These findings indicate that Dok-1 (show DOK1 Antibodies) does not affect "inside-out" platelet signalling.
in the absence of PAG, Csk becomes more associated with alternative partners; i.e., phosphatase PTPN22 and Dok adaptors. Combining PAG deficiency with PTPN22 or Dok adaptor deficiency further enhances effector T cell responses. Unlike PAG, Cbl ubiquitin ligases inhibit the activation of naive, but not of effector, T cells.
These results reveal the critical involvement of Dok-1 (show DOK1 Antibodies) and Dok-2 in a negative-feedback loop that prevents overactivation of CD8 (show CD8A Antibodies)(+) T cells and promotes memory formation.
Thus, Dok-1 (show DOK1 Antibodies) and Dok-2 promote survival of glycoprotein B-specific CD8 (show CD8A Antibodies)(+) T cells in trigeminal ganglia latently infected with herpes simplex virus 1.
this study shows that Dok1 and Dok2 proteins are involved in the control of hematopoietic stem cell cycle regulation
Taken together, our results demonstrate that Dok-1 (show DOK1 Antibodies) and Dok-2 negatively regulate intestinal inflammation, apparently through the induction of IL-17A (show IL17A Antibodies) and IL-22 (show IL22 Antibodies) expression.
Dok1 (show DOK1 Antibodies) knockdown attenuated TLR2-induced NF-kappaB (show NFKB1 Antibodies) activation and IL-6 (show IL6 Antibodies) production in microglia.
Triple Dok1 (show DOK1 Antibodies) Doc2 (show DOC2A Antibodies) Doc3 knockout leads to spontaneous pulmonary inflammation with hallmarks of asthma.
Dok-1 (show DOK1 Antibodies) overexpression promotes the generation of an innate-like CD8 (show CD8A Antibodies)(+) T-cell population that expresses Eomesodermin (show EOMES Antibodies).
In yeast, over 200 protein and RNA cofactors are required for ribosome assembly, and these are generally conserved in eukaryotes. These factors orchestrate modification and cleavage of the initial 35S precursor rRNA transcript into the mature 18S, 5.8S, and 25S rRNAs, folding of the rRNA, and binding of ribosomal proteins and 5S RNA. Nob1 is involved in pre-rRNA processing. In a late cytoplasmic processing step, Nob1 cleaves a 20S rRNA intermediate at cleavage site D to produce the mature 18S rRNA (Lamanna and Karbstein, 2009
ADP-ribosyltransferase 4 (DO blood group)
, ADP-ribosyltransferase C2 and C3 toxin-like 4
, Dombrock blood group carrier molecule
, NAD(P)(+)--arginine ADP-ribosyltransferase 4
, ecto-ADP-ribosyltransferase 4
, mono(ADP-ribosyl)transferase 4
, mono-ADP-ribosyltransferase 4
, PSMD8 binding protein 1
, RNA-binding protein NOB1
, adenocarcinoma antigen recognized by T lymphocytes 4
, nin one binding protein
, phosphorylation regulatory protein HP-10
, protein ART-4
, NAD(P)(+)--arginine ADP-ribosyltransferase
, mono (ADP-ribosyl)transferase
, downstream of tyrosine kinase 1
, Dombrock blood group
, Dombrock glycoprotein
, dombrock molecule 1