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Acts as a guanine nucleotide exchange factor (GEF) for Rab5 GTPase. Additionally we are shipping ALS2CL Proteins (4) and many more products for this protein.
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This supports the notion that de novo mutations in ALS2CL are extremely rare in schizophrenia
Data provide further support that ALS2CL, EPHA3, and CMYA1 are bona-fide tumor-suppressor genes and contribute to the tumorigenesis of HNSCC.
These results suggest that amyotrophic lateral sclerosis 2 C-terminal like (ALS2CL), a novel ALS2 homologue, modulates Rab5-mediated endosome dynamics in HeLa cells.
ALS2CL is a novel ALS2-interacting protein implicated in ALS2-mediated endosome dynamics.
Acts as a guanine nucleotide exchange factor (GEF) for Rab5 GTPase. Regulates the ALS2-mediated endosome dynamics (By similarity).
ALS2 C-terminal-like protein