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BEX2 belongs to the brain expressed X-linked gene family. Additionally we are shipping Brain Expressed X-Linked 2 Proteins (5) and many more products for this protein.
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The study reveals that BEX2 promotes colorectal cancer cell proliferation via the JNK/c-Jun pathway, suggesting BEX2 as a potential candidate target for the treatment of CRC.
Bex2 affects the invasion and migration ability of glioma cells by regulating beta-catenin
Bex2 overexpression promoted cell migration and invasion in glioma cell lines.
Bex2 may be an important player during the development of glioma
Both BEX2 and INI1/hSNF5 mainly localized in cell nucleus.
BEX2 overexpression was associated with breast cancer.
BEX2 has a functional interplay with c-Jun and p65/RelA in breast cancer.
These results suggest that the conspicuous expression of the tumor suppressor genes BEX2, IGSF4 and TIMP3 in MLLmu acute myeloid leukemias cell lines is the consequence of altered epigenetic properties of MLL fusion proteins.
hBex2 may act as a specific regulator during embryonic development by modulating the transcriptional activity of a novel E-box sequence-binding complex that contains hBex2, LMO2, NSCL2 and LDB1.
Two novel brain expressed genes, BEX1 and BEX2 are identified; they are silenced in all glioma specimens and exhibit extensive promoter hypermethylation.
Correlation between BEX2 expression and mixed lineage leukemia chromosomal aberrations are shown in the cell lines.
a NGF/BEX2/NF-kappaB pathway is involved in regulating apoptosis in breast cancer cells and in modulating response to tamoxifen in primary tumors
BEX2 is implicated in oligodendroglioma biology.
enhanced STAT3/NF-kappaB-BEX2-VEGF signaling pathway contributes to hyperactive mTOR-induced tumorigenesis
These results suggest the significance and potential applications of Bex2 in studies of endocrine and stem/progenitor cells.
Expression of ALDH1A2, BEX2, EGR2, CCL3 and PLAU are upregulated in Toxoplasma gondiisusceptible C57BL/6 mice.
We report the identification of an olfactory marker protein binding partner, Bex2, by phage-display screening of an olfactory mucosal cDNA-library
Bex1 and 2 have been characterized as interacting partners of the olfactory marker protein (OMP). Bex immunoreactivity (ir) was primarily localized to neuronal cells within several regions of the brain.
This gene belongs to the brain expressed X-linked gene family. The encoded protein interacts with the transcription factor LIM domain only 2 in a DNA-binding complex that recognizes the E-box element and promotes transcription. This gene has been found to be a tumor suppressor that is silenced in human glioma. In breast cancer cells, this gene product modulates apoptosis in response to estrogen and tamoxifen, and enhances the anti-proliferative effect of tamoxifen. Multiple transcript variants encoding different isoforms have been found for this gene.
brain-expressed X-linked protein 2
, protein BEX2
, brain-expressed X-linked protein 2 homolog
, expression gene 2 in rat visual cortex