anti-Cholecystokinin 8, Octapeptide (CCK8) Antibodies

Human Cholecystokinin 8, Octapeptide (CCK8) interaction partners

1. We found the suicide-associated gene coexpression network. The reconstructed network consisted of 104 genes. Topological analysis showed that in total, CCK (show CCK Antibodies), INPP1 (show INPP1 Antibodies), DDC (show DDC Antibodies), and NPY (show NPY Antibodies) genes are the most fundamental hubs in the network

2. L-trp (show TBPL1 Antibodies) is a luminal regulator of CCK (show CCK Antibodies) release with effects on gastric emptying, an effect that could be mediated by CCK (show CCK Antibodies). L-trp's effect on GLP-1 (show GCG Antibodies) secretion is only minor. At the doses given, the two amino acids did not affect subjective appetite feelings.

3. The CCK (show CCK Antibodies) polymorphism have reported significant association of -45C>T polymorphism with the presence of hallucinations.

4. CCK (show CCK Antibodies) does not appear to play a unique independent role in satiety/satiation.

5. CCK (show CCK Antibodies) release has been found to be halved in pregnant women with hyperemesis gravidarium, which supports the hypothesis that gastrointestinal motility is increased in pregnant women with hyperemesis gravidarium.

6. CCK (show CCK Antibodies) in plasma is an independent marker of cardiovascular mortality in elderly female patients.

7. These data offer preliminary evidence supporting an association between the rs1799923 polymorphism in the CCK (show CCK Antibodies) gene and PTSD

8. Data suggest that endocrine responses differ between jejunal and gastric enteral feeding, with higher peak plasma CCK (cholecystokinin), PYY (peptide YY), and GLP-1/2 (glucagon-like peptides 1/2) concentrations being attained after jejunal feeding.

9. active GLP-1 (show GCG Antibodies) produced in the islet stimulates cholecystokinin (show CCK Antibodies) production and secretion in a paracrine manner via cyclic AMP (show APRT Antibodies) and CREB (show CREB1 Antibodies).

10. Cardiac expression of pro-cholecystokinin (show CCK Antibodies) is cell-specific, which differentiates the expression from that of intestinal endocrine cells and cerebral neurons. Plasma Pro-CCK (show CCK Antibodies) is a prognostic marker in patients with stable heart failure.

Mouse (Murine) Cholecystokinin 8, Octapeptide (CCK8) interaction partners

1. Study shows that independent of the neurochemical content, cholecystokinin (show CCK Antibodies)/type 1 cannabinoid receptor-expressing interneurons have similar physiological and morphological properties, providing an endocannabinoid-sensitive synaptic inhibition onto the amygdalar principal neurons.

2. In summary, our study is the first to indicate that CCK/CCK (show CCK Antibodies)-AR (show CCKAR Antibodies) pathway is critical and protective against liver I/R injury. The activation of this pathway not only prevents hepatocellular apoptosis, but also reduces inflammatory response by suppressing NF-kappaB (show NFKB1 Antibodies).

3. CCK (show CCK Antibodies)/GLP-1 (show GCG Antibodies) play contributory roles in anorexia induction by trichothecenes T-2 toxin, HT-2 toxin, diacetoxyscirpenol and neosolaniol.

4. GPR120 (show O3FAR1 Antibodies)-induced incretin glucse-dependent insulinotropic polypeptide secretion is indirectly mediated by cholecystokinin (show CCK Antibodies).

5. Medullary interstitial cells respond to body fluid expansion by CCK (show CCK Antibodies) release for feedback regulation of the late proximal tubular reabsorption.

6. results suggest that normal integration of CCK (show CCK Antibodies)+ basket cells in cortical networks is key to support spatial coding in the hippocampus.

7. PC7 (show PCSK7 Antibodies) has a critical role in normal processing of cholecystokinin (show CCK Antibodies) in mouse brain

8. These studies reemphasize the beneficial effects imparted by co-administration of obestatin and CCK8 (show CCK Antibodies) and their potential use towards countering obesity.

9. new information on the cell specific localization of NUCB2/nesfatin-1 (show NUCB2 Antibodies) in the intestinal mucosa, and a novel function for nesfatin-1 (show NUCB2 Antibodies) in modulating intestinal CCK (show CCK Antibodies) and PYY expression and secretion

10. Results showed that CCK (show CCK Antibodies) is important for lipid transport and energy expenditure to control body weight in response to dietary lipid feeding