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may facilitate nerve regeneration. Additionally we are shipping Chromosome 5 Open Reading Frame 13 Proteins (9) and and many more products for this protein.
Showing 10 out of 29 products:
Human Polyclonal C5orf13 Primary Antibody for IF (p), IHC (p) - ABIN668496
Peng, Yuan, Tan, Ma, Bian, Xu, He, Cao, Huang, Cui, Gan, Wang, Zhou, Hu, Yang, Luo, Wu: Identification of ITGB4BP as a new interaction protein of P311. in Life sciences 2012
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Human Polyclonal C5orf13 Primary Antibody for ICC, IF - ABIN4342681
Ek, Andréasson, Hober, Kampf, Pontén, Uhlén, Merz, Borrebaeck: From gene expression analysis to tissue microarrays: a rational approach to identify therapeutic and diagnostic targets in lymphoid malignancies. in Molecular & cellular proteomics : MCP 2006
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P311 is a novel TGFbeta1/Smad signaling-mediated regulator of transdifferentiation in epidermal stem cells during cutaneous wound healing.
P311 could accelerate skin wound reepithelialization by promoting the migration of Epidermal Stem Cell through RhoA and Rac1 activation.
These studies demonstrate that P311 is required for the production of normal cutaneous scars
P311 plays a key role in renal fibrosis via TGFbeta1/Smad signaling, which could be a novel target for the management of renal fibrosis.
Novel RNA-binding protein P311 binds eIF3b to promote translation of TGF-beta1, TGF-beta2, TGF-beta3.
P311 may promote the migration of ESCs both in micewith superficial partial-thickness burns and in an injured cell model in vitro, and it may play an important role in wound healing.
Protein HYI may closely bind with protein P311 by an alpha helix in hypertrophic scar fibroblasts.
P311 and ITGB4BP expression was elevated in non-small cell lung cancer, possibly indicative of a new signaling pathway.
P311 may be involved in the pathogenesis of hypertrophic scar via induction of a myofibrobla
role for P311 in inducing TGF-beta1-independent myofibroblast transformation
These results suggest a role for levels of P311 in regulating glioma motility and invasion through the reorganization of actin cytoskeleton at the cell periphery.
P311 expression is tightly regulated during the critical periods of alveolar formation, and that under pathologic conditions, its relative absence may contribute to failure of alveolar regeneration and lead to the development of human emphysema.
P311 is central to reactive oxygen species-mediated hepatic stellate cell migration induced by different chemokines.
P311 is the first protein identified to modulate TGF-beta translation and the first pan-regulator of TGF-beta expression.
formalin-induced avoidance behavior, which reflects pain-related negative emotion, was significantly attenuated in P311-/- mice
these data establish P311 as playing an important role in learning and memory processes and emotional responses
A novel role for P311 in an alternative pathway of lipid-droplet accumulation that is induced by the regeneration-inducing molecule retinoic acid.
may facilitate nerve regeneration
, neuronal protein 3.1
, neuronal regeneration-related protein
, protein p311
, neuronal regeneration related protein homolog
, high-altitude regulated protein
, Neuronal protein 3.1