anti-Chromosome 5 Open Reading Frame 13 (C5orf13) Antibodies

Human Chromosome 5 Open Reading Frame 13 (C5orf13) interaction partners

1. P311 is a novel TGFbeta1 (show TGFB1 Antibodies)/Smad (show SMAD1 Antibodies) signaling-mediated regulator of transdifferentiation in epidermal stem cells during cutaneous wound healing.

2. P311 could accelerate skin wound reepithelialization by promoting the migration of Epidermal Stem Cell through RhoA and Rac1 activation.

3. These studies demonstrate that P311 is required for the production of normal cutaneous scars

4. P311 plays a key role in renal fibrosis via TGFbeta1 (show TGFB1 Antibodies)/Smad (show SMAD1 Antibodies) signaling, which could be a novel target for the management of renal fibrosis.

5. Novel RNA-binding protein (show PTBP1 Antibodies) P311 binds eIF3b (show EIF3B Antibodies) to promote translation of TGF-beta1 (show TGFB1 Antibodies), TGF-beta2 (show TGFB2 Antibodies), TGF-beta3 (show TGFB3 Antibodies).

6. P311 may promote the migration of ESCs (show NR2E3 Antibodies) both in micewith superficial partial-thickness burns and in an injured cell model in vitro, and it may play an important role in wound healing.

7. Protein HYI (show HYI Antibodies) may closely bind with protein P311 by an alpha helix in hypertrophic scar fibroblasts.

8. P311 and ITGB4BP (show EIF6 Antibodies) expression was elevated in non-small cell lung cancer, possibly indicative of a new signaling pathway.

9. P311 may be involved in the pathogenesis of hypertrophic scar via induction of a myofibrobla

10. role for P311 in inducing TGF-beta1 (show TGFB1 Antibodies)-independent myofibroblast transformation

Mouse (Murine) Chromosome 5 Open Reading Frame 13 (C5orf13) interaction partners

1. P311 is a novel TGFbeta1 (show TGFB1 Antibodies)/Smad (show SMAD1 Antibodies) signaling-mediated regulator of transdifferentiation in epidermal stem cells during cutaneous wound healing.

2. P311 could accelerate skin wound reepithelialization by promoting the migration of Epidermal Stem Cell through RhoA and Rac1 activation.

3. These studies demonstrate that P311 is required for the production of normal cutaneous scars

4. P311 plays a key role in renal fibrosis via TGFbeta1 (show TGFB1 Antibodies)/Smad (show SMAD1 Antibodies) signaling, which could be a novel target for the management of renal fibrosis.

5. P311 is central to reactive oxygen species-mediated hepatic stellate cell migration induced by different chemokines.

6. P311 is the first protein identified to modulate TGF-beta (show TGFB1 Antibodies) translation and the first pan (show SUPT6H Antibodies)-regulator of TGF-beta (show TGFB1 Antibodies) expression.

7. P311 may promote the migration of ESCs (show NR2E3 Antibodies) both in micewith superficial partial-thickness burns and in an injured cell model in vitro, and it may play an important role in wound healing.

8. role for P311 in inducing TGF-beta1 (show TGFB1 Antibodies)-independent myofibroblast transformation

9. P311 expression is tightly regulated during the critical periods of alveolar formation, and that under pathologic conditions, its relative absence may contribute to failure of alveolar regeneration and lead to the development of human emphysema.

10. formalin-induced avoidance behavior, which reflects pain-related negative emotion, was significantly attenuated in P311-/- mice