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Cytochrome b (-245) is composed of cytochrome b alpha (CYBA) and beta (CYBB) chain. Additionally we are shipping CYBB Antibodies (118) and CYBB Proteins (6) and many more products for this protein.
Showing 3 out of 11 products:
Human CYBB ELISA Kit for Sandwich ELISA - ABIN418998
Tassone, Perticone, Sciacqua, Mafrici, Settino, Malara, Mollace, Sesti, Perticone: Low dose of acetylsalicylic acid and oxidative stress-mediated endothelial dysfunction in diabetes: a short-term evaluation. in Acta diabetologica 2015
Rat (Rattus) CYBB ELISA Kit for Sandwich ELISA - ABIN433568
Du, Wang, Wang: Role of RhoA/MERK1/ERK1/2/iNOS signaling in ocular ischemic syndrome. in Graefe's archive for clinical and experimental ophthalmology 2016
The nox1 (show NOX1 ELISA Kits), nox2/cybb expression in zebrafish during early nervous system development from 12 to 48 hours post fertilization
Data indicate that NADPH oxidase (show NOX1 ELISA Kits) NOx2 inhibition attenuates endoplasmic reticulum (ER) stress and apoptosis.
Study demonstrates that there is no increase of NOX2 expression in schizophrenic patients. Unexpectedly, however, the study found that NOX2 expression was decreased in patients with bipolar disorder.
Hyperinsulinemia modulates arteriolar flow-induced dilation via Nox2-mediated superoxide production.
this study shows that aging-associated metabolic disorder induces Nox2 activation and oxidative damage of endothelial function
Results show that Nox2 knockdown attenuated HIV-1 Tat (show TAT ELISA Kits)-induced HDAC6 (show HDAC6 ELISA Kits) expression and subsequent expression of chemokines. Also, the data provide evidence that HDAC6 (show HDAC6 ELISA Kits) mediates HIV-1 Tat (show TAT ELISA Kits)-induced reactive oxygen species generation by regulating the activity and expression of Nox2-based NADPH oxidase (show NOX1 ELISA Kits) in astrocytes, and demonstrate the existence of a crosstalk between HDAC6 (show HDAC6 ELISA Kits) and NADPH oxidase (show NOX1 ELISA Kits) in HIV-1 Tat (show TAT ELISA Kits)-stimulated astrocytes.
Porphyromonas gingivalis modulates the danger signal eATP-induced NOX2 signaling and also induces host glutathione synthesis to likely avoid HOCl mediated clearance.
the increased oxidative stress was mediated by nox2 dependent reactive oxygen species overproduction that could downregulate whole-cell K(+) currents
Nox2 activity efficiently contributes to the mechanism of oxidative stress-induced (show SQSTM1 ELISA Kits) increase in premature aging conferred by doxorubicin. The importance of modulation of Nox2 in human EPCs could reveal a useful tool to restore endothelial progenitor cells physiological function and properties.
We examined the pattern of NOX expression in spinal cords of patients and mouse models of ALS (show IGFALS ELISA Kits) and analyzed the impact of genetic deletion of the NOX1 (show NOX1 ELISA Kits) and 2 isoforms as well as pharmacological NOX inhibition in the SOD1 (show SOD1 ELISA Kits)(G93A) ALS (show IGFALS ELISA Kits) mouse model.In contrast to previous publications, survival of SOD1 (show SOD1 ELISA Kits)(G93A) mice was not modified upon breeding with constitutive NOX1 (show NOX1 ELISA Kits) and NOX2 deficient mice.
Recent studies reveal that AKT2 (show AKT2 ELISA Kits)-NOX2 signaling has critical roles in Ca mobilization, ROS (show ROS1 ELISA Kits) generation, degranulation, and control of the ligand-binding function of cell surface molecules, thereby promoting heterotypic cell-cell interactions in thromboinflammation.
Study demonstrate that Nox2 signaling via inactivation of phosphatase and tensin homolog and activation of the PI3K (show PIK3CA ELISA Kits)/Akt (show AKT1 ELISA Kits) pathway leads to membrane ruffling and macropinocytosis.
Data indicate that the efficiency of NADPH oxidase (show NOX1 ELISA Kits) enzymatic activity is higher at endoplasmic reticulum (ER).
A p47(phox) and Src kinase (show CSK ELISA Kits) activation of peroxide production by Nox2 appears to be an important contributor to vascular contractile mechanisms mediated through activation of protein kinase C (show PKC ELISA Kits)
We propose that NOX2-derived ROS (show ROS1 ELISA Kits) facilitate metastasis of melanoma cells by downmodulating NK-cell function and that inhibition of NOX2 may restore IFNgamma-dependent, NK cell-mediated clearance of melanoma cells
NOX2 knockout mice was used to study the role of NOX2 in mediating NLRP3 (show NLRP3 ELISA Kits) inflammasome expression and activation following a controlled cortical impact.
Myricitrin attenuated the generation of intracellular ROS by inhibiting the assembly of components of the gp91(phox) and p47(phox). Suppression of ROS generation using NAC or apocynin or by silencing gp91(phox) and p47(phox) all demonstrated that decreasing the level of ROS inhibited the LPS-induced inflammatory response.
Ablation of NOX2 prevented inflammation-induced axon initial segment plasticity.
the present study suggests that acute lung injury-induced upregulation of neutrophilic and neuronal NOX-2 may contribute to depression-like symptoms in mice
Findings indicate that microglial NOX2-generated H2O2 is able to regulate the immunological functions of astroglia via a STAT1 (show STAT1 ELISA Kits)/3-dependent mechanism.
Role of Protein Kinase C (show PKC ELISA Kits) and Nox2-Derived Reactive Oxygen Species Formation in the Activation and Maturation of Dendritic Cells by Phorbol Ester and Lipopolysaccharide.
this study shows that neutrophil pyroptosis mediates pathology of Pseudomonas aeruginosa lung infection in the absence of the NADPH oxidase (show NOX1 ELISA Kits) NOX2
Data indicate that increased NADPH oxidase (show NOX1 ELISA Kits) activity after traumatic brain injury drives M1-like activation in microglia/macrophages that contributes to inflammatory-mediated neurodegeneration.
Neurofibromin (show NF1 ELISA Kits) regulates macrophage superoxide production via p21Ras.Neurofibromin-deficient smooth muscle cells are sensitive to oxidative stress.NADPH oxidase 2 is required for NF1 (show NF1 ELISA Kits) arterial stenosis.Neurofibromatosis type 1 patients have evidence of chronic oxidative stress.
These results suggested that resveratrol strongly enhances the RA-induced O2(-)-generating activity via up-regulation of gp91-phox gene expression in U937 cells.
sub-vasomotor concentration of ET-1 (show EDN1 ELISA Kits) leads to vascular dysfunction by impairing endothelium-dependent NO-mediated dilation via p38 (show MAPK14 ELISA Kits) kinase-mediated production of superoxide from NADPH oxidase (show NOX1 ELISA Kits) following ETA receptor activation
Reactive oxygen species generated by NADPH oxidase (show NOX1 ELISA Kits) contribute to the aberrant pulmonary arterial responses in piglets exposed to 3 days of hypoxia.
Upregulation of PPAR-gamma and NADPH oxidases are involved in restenosis.
Cytochrome b (-245) is composed of cytochrome b alpha (CYBA) and beta (CYBB) chain. It has been proposed as a primary component of the microbicidal oxidase system of phagocytes. CYBB deficiency is one of five described biochemical defects associated with chronic granulomatous disease (CGD). In this disorder, there is decreased activity of phagocyte NADPH oxidase\; neutrophils are able to phagocytize bacteria but cannot kill them in the phagocytic vacuoles. The cause of the killing defect is an inability to increase the cell's respiration and consequent failure to deliver activated oxygen into the phagocytic vacuole.
cytochrome b-245, beta polypeptide
, NADPH oxidase 2
, cytochrome b-245, beta polypeptide (chronic granulomatous disease)
, Cytochrome b-245 heavy chain
, NADPH oxidase heavy chain subunit
, NADPH oxidase 1
, NADPH oxidase flavocytochrome b subunit
, cytochrome b-245 heavy chain
, cytochrome b(558) subunit beta
, cytochrome b558 subunit beta
, heme-binding membrane glycoprotein gp91phox
, neutrophil cytochrome b 91 kDa polypeptide
, p22 phagocyte B-cytochrome
, superoxide-generating NADPH oxidase heavy chain subunit
, endothelial type gp91-phox
, predicted NADPH oxidase-2