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D-dopachrome tautomerase converts D-dopachrome into 5,6-dihydroxyindole. Additionally we are shipping D-Dopachrome Tautomerase Antibodies (76) and D-Dopachrome Tautomerase Proteins (16) and many more products for this protein.
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D-dopachrome tautomerase transcription may be regulated in a cell-dependent manner, and was enhanced by AMPK (show PRKAA1 ELISA Kits) activation in SGBS (show GPC3 ELISA Kits) adipocytes through inhibiting the mTOR (show FRAP1 ELISA Kits) signaling
The knockdown of D-DT and MIF (show AMH ELISA Kits), individually and additively, inhibited the proliferation, migration, and invasion in HeLa and SiHa cells and restrained the growth of xenograft tumor.
These results have implications for the manner in which D-DT and MIF (show AMH ELISA Kits) compete with each other for binding to the CD74 (show CD74 ELISA Kits) receptor and for the relative potency of DRa1-MOG-35-55 and RTL1000 for competitive inhibition of D-DT and MIF (show AMH ELISA Kits) binding and activation through CD74 (show CD74 ELISA Kits).
Study demonstrated that DDT was over- expressed in pancreatic ductal adenocarcinoma (PDAC) tissues and cell lines in a pattern correlated with MIF (show AMH ELISA Kits), and knockdown of DDT and MIF (show AMH ELISA Kits) in PANC- 1 cells cooperatively inhibited cell proliferation, invasion and tumor formation. The tautomerase activities of both MIF (show AMH ELISA Kits) and DDT are required for their negative regulatory role in p53 (show TP53 ELISA Kits) and their tumor-promoting functions.
DDT was increased in burn patients.
Gene expression level of DDT is significantly higher in AD patients when compared to normal controls.
High MIF-2 (show CENPC1 ELISA Kits) levels are predictive of the development of organ dysfunction in myocardial ischemia reperfusion injury.
Both p53 (show TP53 ELISA Kits) wildtype and mutant human lung adenocarcinoma tumors rely on MIF (show AMH ELISA Kits) family members for maximal cell growth and survival.
findings identify DDT as a functionally redundant but more potent cytokine to MIF (show AMH ELISA Kits) in cancer and suggest that current attempts to inhibit MIF (show AMH ELISA Kits) signaling may fail because of DDT compensation.
D-dopachrome tautomerase secreted from adipocytes acts on preadipocytes to promote IL-6 (show IL6 ELISA Kits) expression and to inhibit adipogenesis by suppressing the induction of genes encoding adipogenic regulators
cardiomyocyte secretion of DDT has important autocrine/paracrine effects during ischemia-reperfusion that protect the heart against injury
These data point to a potential involvement of D-dopachrome tautomerase activity in the mature mouse brain, and suggest some functional and evolutionary relationship between innate immunity and tautomerization of D-dopachrome in mammalian species
These data indicate that D-DT is a MIF (show MIF ELISA Kits)-like cytokine.
D-dopachrome tautomerase converts D-dopachrome into 5,6-dihydroxyindole. The DDT gene is related to the migration inhibitory factor (MIF) in terms of sequence, enzyme activity, and gene structure. DDT and MIF are closely linked on chromosome 22.
, D-dopachrome decarboxylase-A
, D-dopachrome tautomerase-A
, D-dopachrome decarboxylase-B
, D-dopachrome tautomerase-B
, D-dopachrome tautomerase
, dopachrome isomerase
, phenylpyruvate tautomerase II