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a Golgi resident sialoglycoprotein that binds basic fibroblast growth factor [RGD, Feb 2006].. Additionally we are shipping GLG1 Proteins (6) and GLG1 Kits (1) and many more products for this protein.
Showing 10 out of 72 products:
Human Polyclonal GLG1 Primary Antibody for ICC, IF - ABIN4315350
Morisaki, Yashiro, Kakehashi, Inagaki, Kinoshita, Fukuoka, Kasashima, Masuda, Sakurai, Kubo, Muguruma, Ohira, Wanibuchi, Hirakawa: Comparative proteomics analysis of gastric cancer stem cells. in PLoS ONE 2014
Exogenous acetate augments Acss2 (show ACSS2 Antibodies)/HIF-2 dependent cancer growth and metastasis in cell culture and mouse models
the structural model of the HIF2a (show EPAS1 Antibodies)-pVHL (show VHL Antibodies) complex presented in this study enhances understanding of how HIF2a (show EPAS1 Antibodies) is captured by pVHL (show VHL Antibodies). Moreover, the important contact amino acids that we identified may be useful in the development of drugs to treat HIF2a (show EPAS1 Antibodies)-related diseases.
Thus, we provide evidence here that HIF-2a is a critical regulator of PD-L1 (show CD274 Antibodies) at both mRNA and protein levels and that HIF-2a regulates the expression of PD-L1 (show CD274 Antibodies) by binding directly to the HRE-4 in the PD-L1 (show CD274 Antibodies) proximal promoter.
HIF2alpha (show EPAS1 Antibodies) has a role and is an independent marker of the metastatic potential of bone metastatic clear cell renal cell cancer; however, unlike HIF1alpha (show HIF1A Antibodies), increased HIF2alpha (show EPAS1 Antibodies) expression is a favorable prognostic factor
Knockdown of either HIF-1 (show HIF1A Antibodies) or CREB (show CREB1 Antibodies) or both in hypoxia reduced the expression of hypoxia-response elements- and CRE-mediated gene expression, diminished cell proliferation and increased caspase-3 (show CASP3 Antibodies) activity. We did not detect any significant effect of the efficiently knocked down HIF-2 on any of the functions tested in vitro.
miR (show MLXIP Antibodies)-558 facilitates the expression of HIF-2alpha (show EPAS1 Antibodies) through binding to its 5'-UTR (show UTS2R Antibodies), thus promoting the tumorigenesis and aggressiveness of neuroblastoma (show ARHGEF16 Antibodies)
Study identifies NRP2 (show NELL2 Antibodies) and ESL-1 as targets for polysialylation in murine microglia and human THP-1 (show GLI2 Antibodies) macrophages and reveals a striking convergence in the regulation of the two polysialylated acceptors in the course of inflammatory activation. A pool of polysialylated NRP2 (show NELL2 Antibodies) and ESL-1 is assembled after injury or in culture and its shedding is an early response to lipopolysaccharide-induced activation of microglia.
Over-expression of HIF-2alpha (show EPAS1 Antibodies) induced apoptosis in HCC (show FAM126A Antibodies) cells and increased the levels of pro-apoptotic proteins, Bak (show BAK1 Antibodies), ZBP-89 (show ZNF148 Antibodies) and PDCD4 (show PDCD4 Antibodies), whereas the inhibition of HIF-2alpha (show EPAS1 Antibodies) expression achieved opposite results. HIF-2alpha (show EPAS1 Antibodies) was decreased and played an anti-tumorigenic role in hepatocellular carcinoma.
Probiotic Bifidobacterium bifidum MIMBb75 may help attenuating EPAS1 (show EPAS1 Antibodies) overexpression associated with intestinal inflammation.
Data suggest that HIF2alpha (show EPAS1 Antibodies) mediates hypoxia-induced cancer growth/metastasis and that EFEMP1 (show FBLN3 Antibodies) is a downstream effector of hypoxia-induced HIF2alpha (show EPAS1 Antibodies) during breast tumorigenesis.
Study identifies NRP2 (show NRP2 Antibodies) and ESL-1 as targets for polysialylation in murine microglia and human THP-1 (show GLI2 Antibodies) macrophages and reveals a striking convergence in the regulation of the two polysialylated acceptors in the course of inflammatory activation. A pool of polysialylated NRP2 (show NRP2 Antibodies) and ESL-1 is assembled after injury or in culture and its shedding is an early response to lipopolysaccharide-induced activation of microglia.
A cell-intrinsic mechanism whereby haematopoietic cells limit proliferation within the bone marrow is repressed by E-selectin ligand 1 (ESL-1).
Data indicate that only the combined deficiency in PSGL-1 (show SELPLG Antibodies) and ESL-1 completely abrogated leukocyte recruitment.
Heterozygous deficiency of ESL-1 is associated with features of increased atherosclerotic plaque stability while complete deficiency of ESL-1 leads to embryonic lethality.
striking differences between the requirement of FucT-VII (show FUT7 Antibodies) and C2GlcNAcT-I for Ligands for E-selectin (show SELE Antibodies) and P-selectin (show SELP Antibodies) expression in CD4 (show CD4 Antibodies)+ T cells.
Data revealed interaction between Glg1 and Fgf18 (show FGF18 Antibodies) both genetically and physically and reveals a novel regulatory mechanism for Fgf18 (show FGF18 Antibodies) signaling involving Glg1 and Dlk (show DAPK3 Antibodies).
E-selectin ligand-1 (ESL-1), P-selectin glycoprotein ligand-1 (PSGL-1 (show SELPLG Antibodies)), and CD44 (show CD44 Antibodies) encompassed all endothelial-selectin ligand activity on neutrophils by using gene- and RNA-targeted loss of function.
This gene encodes a transcription factor involved in the induction of genes regulated by oxygen, which is induced as oxygen levels fall. The encoded protein contains a basic-helix-loop-helix domain protein dimerization domain as well as a domain found in proteins in signal transduction pathways which respond to oxygen levels. Mutations in this gene are associated with erythrocytosis familial type 4.
, HIF-1-alpha-like factor
, HIF-1alpha-like factor
, PAS domain-containing protein 2
, basic-helix-loop-helix-PAS protein MOP2
, class E basic helix-loop-helix protein 73
, endothelial PAS domain-containing protein 1
, hypoxia-inducible factor 2 alpha
, hypoxia-inducible factor 2-alpha
, member of PAS protein 2
, Golgi apparatus protein 1
, Golgi membrane sialoglycoprotein MG160
, golgi apparatus protein 1
, golgi glycoprotein 1
, golgi apparatus protein 1-like
, E-selectin ligand 1
, golgi sialoglycoprotein MG-160
, selectin, endothelial cell, ligand
, cysteine-rich fibroblast growth factor receptor
, Golgi sialoglycoprotein MG-160
, Latent TGF-beta complexed protein 1
, latent TGF-beta complexed protein (LTCP)