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The Golgi complex plays a key role in the sorting and modification of proteins exported from the endoplasmic reticulum. Additionally we are shipping GOLPH3 Kits (7) and GOLPH3 Proteins (5) and many more products for this protein.
Showing 10 out of 88 products:
Human Monoclonal GOLPH3 Primary Antibody for WB - ABIN1882249
Bechtel, Rosenfelder, Duda, Schmidt, Ernst, Wellenreuther, Mehrle, Schuster, Bahr, Blöcker, Heubner, Hoerlein, Michel, Wedler, Köhrer, Ottenwälder, Poustka, Wiemann, Schupp: The full-ORF clone resource of the German cDNA Consortium. in BMC genomics 2008
Show all 6 Pubmed References
Human Monoclonal GOLPH3 Primary Antibody for WB - ABIN1882248
Bell, Ward, Blackstock, Freeman, Choudhary, Lewis, Chotai, Fazel, Gushue, Paiement, Palcy, Chevet, Lafrenière-Roula, Solari, Thomas, Rowley, Bergeron: Proteomics characterization of abundant Golgi membrane proteins. in The Journal of biological chemistry 2001
Show all 6 Pubmed References
Human Polyclonal GOLPH3 Primary Antibody for FACS, IHC (p) - ABIN391649
Li, You, Mo, He, Tang, Jiang, Chen, Chen, Zhang, Hu: GOLPH3 Mediated Golgi Stress Response in Modulating N2A Cell Death upon Oxygen-Glucose Deprivation and Reoxygenation Injury. in Molecular neurobiology 2015
Human Polyclonal GOLPH3 Primary Antibody for ICC, IF - ABIN4315389
Sotgia, Whitaker-Menezes, Martinez-Outschoorn, Salem, Tsirigos, Lamb, Sneddon, Hulit, Howell, Lisanti: Mitochondria "fuel" breast cancer metabolism: fifteen markers of mitochondrial biogenesis label epithelial cancer cells, but are excluded from adjacent stromal cells. in Cell cycle (Georgetown, Tex.) 2012
Golph3 is a target gene of miR34a. miR34a/GOLPH3 axis abrogates cancer cell stemness involved in urothelial bladder cancer chemoresistance.
The current study found that GOLPH3, an outer membrane protein (show SYNJ2BP Antibodies) of the Golgi complex, was significantly upregulated in N2A cells upon oxygen-glucose deprivation and reoxygenation.
High GOLPH3 expression is associated with breast cancer.
This study provides a brand new evidence that GOLPH3 promotes glioma cell proliferation by facilitating Wls (show WLS Antibodies) recycling and Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) signaling.
High GOLPH3 expression is associated with glioma progression.
Proteomic-based screens identified GOLPH3 as a resident protein of the Golgi apparatus, and second as an oncogene (show RAB1A Antibodies) product in a screen for genes amplified in cancer. A third screen uncovered the association of GOLPH3 with the Golgi resident phosphatidyl inositol 4 phosphate to maintain the characteristic ribbon structure of the Golgi apparatus favoring vesicular transport of secretory proteins. [review]
Overexpression of GOLPH3 resulted in the downregulation of p21 protein (show NRAS Antibodies), upregulation of cyclin D1 (show CCND1 Antibodies) and increased retinoblastomaassociated protein phosphorylation, consequently leading to accelerated cell cycle progression.
Results indicate that GOLPH3 is an independent predictive factor for neoadjuvant chemoradiotherapy (nCRT) sensitivity and prognosis in locally advanced rectal cancer (LARC (show CCL20 Antibodies)).
Results provide evidences that overexpression of GOLPH3 stimulates EMT (show ITK Antibodies) via the Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) signaling pathway, which further promotes metastasis of epithelial ovarian cancer.
GOLPH3 levels may be a novel prognostic biomarker of tobacco related lung disease.
Authors confirm that the decrease of GOLPH3 that promotes the apoptosis of glioma cells may be regulated by the activation of NDRG1 and cleaved capcase 3. There was a inverse association between GOLPH3 and NDRG1 in glioma samples.
Data found that miR (show MLXIP Antibodies)-134 was highly downregulated in gastric cancer tissues and cell lines and inversely correlated with GOLPH3. These results suggest that miR (show MLXIP Antibodies)-134 regulates gastric cancer cell proliferation, at least potentially, through downregulation of the GOLPH3 gene, implicating a candidate tumor-suppressor miRNA in the pathogenesis of gastric cancer.
The selective S-nitrosylation of EMMPRIN and GOLPH3 at the Golgi apparatus in endothelial cells results from the physical proximity to eNOS (show NOS3 Antibodies)-derived nitric oxide.
The Golgi complex plays a key role in the sorting and modification of proteins exported from the endoplasmic reticulum. The protein encoded by this gene is a peripheral membrane protein of the Golgi stack and may have a regulatory role in Golgi trafficking. Several alternatively spliced transcript variants of this gene have been described, but the full-length nature of these variants has not been determined.
golgi phosphoprotein 3
, Golgi phosphoprotein 3
, coat protein GPP34
, golgi peripheral membrane protein 1, 34 kDa
, golgi protein
, golgi-associated protein
, mitochondrial DNA absence factor
, trans-Golgi protein GMx33