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In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. Additionally we are shipping HOXA9 Proteins (10) and and many more products for this protein.
Showing 10 out of 121 products:
Human Polyclonal HOXA9 Primary Antibody for ICC, IF - ABIN4319699
Molino, Jabès, Bonnet, Gaudin, Bernard, Benech, Khrestchatisky: Gene expression comparison reveals distinct basal expression of HOX members and differential TNF-induced response between brain- and spinal cord-derived microvascular endothelial cells. in Journal of neuroinflammation 2016
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Human Monoclonal HOXA9 Primary Antibody for IF, ELISA - ABIN561318
Storlie, Jackson, Hutchinson, Grose: Delayed biosynthesis of varicella-zoster virus glycoprotein C: upregulation by hexamethylene bisacetamide and retinoic acid treatment of infected cells. in Journal of virology 2006
Cow (Bovine) Polyclonal HOXA9 Primary Antibody for WB - ABIN2778526
Whelan, Ludwig, Bertrand: HoxA9 induces insulin-like growth factor-1 receptor expression in B-lineage acute lymphoblastic leukemia. in Leukemia 2008
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Mouse (Murine) Polyclonal HOXA9 Primary Antibody for ELISA, WB - ABIN4221469
Hu, Fong, Ferrell, Largman, Shen: HOXA9 modulates its oncogenic partner Meis1 to influence normal hematopoiesis. in Molecular and cellular biology 2009
MiR (show MLXIP Antibodies)-182/HOXA9 was involved in the process of RUNX3 (show RUNX3 Antibodies)-mediated GC tumor growth.
The present study demonstrated that miR (show MLXIP Antibodies)-139-5p, functioning as a suppressor in the tumorigenesis process, suppressed oral squamous carcinoma cells mobility by targeting HOXA9 directly.
NHA9 (NUP98 (show NUP98 Antibodies)-HOXA9 fusion protein) deregulates the expression of key leukemic genes, including MEIS1 (show MEIS1 Antibodies)-HOXA9-PBX3 complex, through the enhancer binding and the direct interaction of the fusion protein with HDAC (show HDAC3 Antibodies) and p300 (show EP300 Antibodies) transcriptional regulators
Results show that a combination of HOXA9 and HOXA10 (show HOXA10 Antibodies) promoter methylation markers is significantly associated with the prognosis of breast cancer patients a subtype-independent manner.
knockdown of HOXA9 abrogated NAC1induced drug resistance.
HOXA9 expression is upregulated in three-dimensional organotypic culture of claudin-low breast cancer cells.
HOXA9 transcriptionally regulated EPHB4 (show EPHB4 Antibodies) expression to modulate trophoblasts migration and invasion, which may suggest a contribution of HOXA9-EPHB4 (show EPHB4 Antibodies) in the poor placentation in the pathogenesis of preeclampsia.
Overexpression of HOXA4 (show HOXA4 Antibodies) and HOXA9 contributes to self-renewal and overpopulation of stem cells in colorectal cancers.
HOTTIP/WDR5 (show WDR5 Antibodies)/HOXA9/Wnt (show WNT2 Antibodies) axis contributes to pancreatic tumor stem cell stemness.
Beta-catenin (show CTNNB1 Antibodies) regulates expression of downstream targets of a key transcriptional memory gene, Hoxa9 that is highly enriched in myeloid leukemia (show BCL11A Antibodies) cancer stem cells and helps sustain leukemic self-renewal.
The analysis points to a critical role for Hoxa9 and PU.1 in distal regulation of c-myb (show MYB Antibodies) expression in murine myeloid cells during iL-6 (show IL6 Antibodies)-induced cell differentiation.
the Hoxa9- and Meis1 (show MEIS1 Antibodies)-associated upregulation of Flt3 (show FLT3 Antibodies) is a passive event with regard to leukemia development in mice and with limited relevance to the AML (show RUNX1 Antibodies) pathology.
the cohesin complex regulates PRC2 targeting to silence Hoxa7 and Hoxa9 and negatively regulate self-renewal. Our studies identify a novel epigenetic mechanism underlying leukemogenesis in AML patients with cohesin mutations.
data delineate an altered epigenetic stress response in activated satellite cells from aged mice, which limits satellite cell function and muscle regeneration by Hoxa9-dependent activation of developmental pathways
Pbx3 contributes to Hoxa9 leukemogenesis through stabilization of the Meis1 (show MEIS1 Antibodies) protein.
IGF-1 (show IGF1 Antibodies) is a direct HOXA9 target important for hematopoietic transformation.
deficiency of tumor suppressor prep1 accelerates the onset of meis1 (show MEIS1 Antibodies)- hoxa9 leukemogenesis
Hoxa9 collaborates with E2A (show TCF3 Antibodies)-PBX1 (show PBX1 Antibodies) in mouse B cell leukemia in association with Flt3 (show FLT3 Antibodies) activation and decrease of B cell gene expression.
results suggest a previously unidentified role for C/EBPalpha (show CEBPA Antibodies) in maintaining the proliferation required for Hoxa9/Meis1 (show MEIS1 Antibodies)-mediated leukemogenesis
In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. Expression of these proteins is spatially and temporally regulated during embryonic development. This gene is part of the A cluster on chromosome 7 and encodes a DNA-binding transcription factor which may regulate gene expression, morphogenesis, and differentiation. This gene is highly similar to the abdominal-B (Abd-B) gene of Drosophila. A specific translocation event which causes a fusion between this gene and the NUP98 gene has been associated with myeloid leukemogenesis. Read-through transcription exists between this gene and the upstream homeobox A10 (HOXA10) gene.
, homeobox protein Hox-A9
, XIHbox 6
, homeobox protein Hox-1G
, homeodomain protein HOXA9
, homeo box A9
, homeobox protein Hox-1.7
, Homeobox gene A7
, homeobox protein Hox-1.1
, homeobox protein Hox-A7
, homeobox protein R5
, homeobox protein A9