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HIVEP2 encodes a member of a family of closely related, large, zinc finger-containing transcription factors. Additionally we are shipping Human Immunodeficiency Virus Type I Enhancer Binding Protein 2 Kits (4) and many more products for this protein.
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Cow (Bovine) Polyclonal HIVEP2 Primary Antibody for ELISA - ABIN4220935
Mungall, Palmer, Sims, Edwards, Ashurst, Wilming, Jones, Horton, Hunt, Scott, Gilbert, Clamp, Bethel, Milne, Ainscough, Almeida, Ambrose, Andrews, Ashwell, Babbage, Bagguley, Bailey, Banerjee, Barker et al.: The DNA sequence and analysis of human chromosome 6. ... in Nature 2003
Schnurri-2 knockout mice have behavioral abnormalities that resemble those of schizophrenics.
Shn-2 plays an important role in the activation and function of NK cells and the development of T cell lymphoma in vivo.
repression of T cell receptor-induced death pathways is critical for proper interpretation of positive selecting signals in vivo; identification of schnurri-2 (Shn2; or Hivep2) as a crucial death dampener
Shn2-deficient mice maintain bone mass at the levels comparable to wild-type sham mice even after ovariectomy-induced bone loss.
Results indicate that Schnurri-2-mediated repression of NF-kappaB (show NFKB1 Antibodies) is required for cell survival and the successful generation of memory Th1 (show HAND1 Antibodies)/Th2 cells.
report that mice bearing parallel null mutations in the adapter proteins Schnurri2 (Shn2) and Schnurri3 (Shn3) exhibit defects in patterning of the axial skeleton during embryogenesis.
Data suggest that Schnurri-2 plays a crucial role in the control of T helper type 2 cell differentiation by regulating NF-kappaB (show NFKB1 Antibodies) function.
Shn-2 plays a critical role in locomotion and anxiety-like behavior. Stress-induced corticosterone levels were significantly higher in Shn-2(-/-) mice compared to wild-type controls.
Shn-2 appears to control the generation of memory Th1 (show HAND1 Antibodies)/Th2 cells through a change in their susceptibility to cell death.
Shn-2 in Th2 cells plays an important role as a negative regulator in allergic airway inflammation.
HIVEP2 loss-of-function mutations were identified in intellectual disability patients.
a 154-amino acid region of MIBP1 was necessary for its O-GlcNAc transferase binding and O-GlcNAcylation.
This gene encodes a member of a family of closely related, large, zinc finger-containing transcription factors. The encoded protein regulates transcription by binding to regulatory regions of various cellular and viral genes that maybe involved in growth, development and metastasis. The protein contains the ZAS domain comprised of two widely separated regions of zinc finger motifs, a stretch of highly acidic amino acids and a serine/threonine-rich sequence.
human immunodeficiency virus type I enhancer binding protein 2
, schnurri 2
, immunodeficiency virus type I enhancer-binding protein 2 homolog
, transcription factor HIVEP2-like
, human immunodeficiency virus type I enhancer-binding protein 2 homolog
, myc intron-binding protein 1
, transcription factor HIVEP2
, DNA-binding protein AGIE-BP1
, angiotensinogen gene-inducible enhancer-binding protein 1
, c-myc intron binding protein 1
, human immunodeficiency virus type 1 enhancer-binding protein 2
, MHC binding protein-2
, MHC-binding protein 2
, human immunodeficiency virus type I enhancer-binding protein 2