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The protein encoded by KRT16 is a member of the keratin gene family.
Showing 10 out of 161 products:
Human Monoclonal KRT16 Primary Antibody for - ABIN920535
Poschmann, Sitek, Sipos, Ulrich, Wiese, Stephan, Warscheid, Klöppel, Vander Borght, Ramaekers, Meyer, Stühler: Identification of proteomic differences between squamous cell carcinoma of the lung and bronchial epithelium. in Molecular & cellular proteomics : MCP 2009
Show all 5 Pubmed References
Human Polyclonal KRT16 Primary Antibody for ICC, IF - ABIN407713
Sonnylal, Xu, Jones, Tam, Sreeram, Ponticos, Norman, Agrawal, Abraham, de Crombrugghe: Connective tissue growth factor causes EMT-like cell fate changes in vivo and in vitro. in Journal of cell science 2013
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Cow (Bovine) Polyclonal KRT16 Primary Antibody for WB - ABIN2775315
Furukawa, Abe, Wang, Oki, Uda, Tsuneda, Ito: Fluorogenic probe triggered by reduction for nucleic acids sensing. in Nucleic acids symposium series (2004) 2008
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Cow (Bovine) Polyclonal KRT16 Primary Antibody for WB - ABIN2775314
Ito, Kurokawa, Nishimura, Hakamada, Isoda, Yamanaka, Tsubura, Mizutani: Keratin and filaggrin expression in keratoacanthoma. in Journal of the European Academy of Dermatology and Venereology : JEADV 2008
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Human Polyclonal KRT16 Primary Antibody for EIA, IHC (p) - ABIN265495
Zhang, Han, Song, Luo, Jin, Mu, Su, Ji, Ren, Liu: Interferon-? regulates the function of mesenchymal stem cells from oral lichen planus via indoleamine 2,3-dioxygenase activity. in Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology 2015
Human Monoclonal KRT16 Primary Antibody for IHC (fro), IHC (p) - ABIN119040
Hachiya, Sriwiriyanont, Fujimura, Ohuchi, Kitahara, Takema, Kitzmiller, Visscher, Tsuboi, Boissy: Mechanistic effects of long-term ultraviolet B irradiation induce epidermal and dermal changes in human skin xenografts. in The American journal of pathology 2009
Human Monoclonal KRT16 Primary Antibody for IHC (fro), IHC (p) - ABIN119041
Markey, Lane, Macdonald, Leigh: Keratin expression in basal cell carcinomas. in The British journal of dermatology 1992
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results suggest a conditional regulation of KRT16 gene by ATF4 that may be inhibited in normal cells, but engaged during cancer progression. Potential roles of KRT16, FAM129A and HKDC1 genes upregulation in adaptive stress responses and pathologies are discussed
inflammatory cytokines promoted Nrf2 (show GABPA Antibodies) nuclear translocation in psoriatic epidermis, which led to elevated expression of K6, K16, and K17 (show KRT17 Antibodies), thus promoting keratinocyte proliferation and contributing to the pathogenesis of psoriasis.
The wider spectrum of KRT16 mutations suggests that changes in codons 125, 127, and 132 are most commonly responsible for PC-1 (show PCSK1 Antibodies) and that proline substitution mutations at codons 127 or 128 may produce more severe disease
analysis of a new p.Leu421Pro (c.1262T>C) mutation in the highly conserved helix termination motif of K16 in a large Spanish family that may have a role in pachyonychia congenita
Keratin 16 regulates innate immunity in response to epidermal barrier breach.
Data established a seven-gene (AR, ESR2 (show ESR2 Antibodies), GATA3 (show GATA3 Antibodies), GBX2 (show GBX2 Antibodies), KRT16, MMP28 (show MMP28 Antibodies) and WNT11 (show WNT11 Antibodies)) prognostic signature to define a subset of triple-negative breast cancer (TNBC).
Genotype-phenotype correlations among PC patients with codon-125 mutation in KRT16 were established, while the phenotypes caused by the IVS8-2A>C mutation in KRT6A (show KRT6A Antibodies) need further studies to confirm the rare feature of fissured tongue
We found that the KRT16 mutation carriers developed plantar keratoderma at a similar age to KRT6A (show KRT6A Antibodies) carriers wheras KRT6B (show KRT6B Antibodies) and KRT17 (show KRT17 Antibodies) carriers were significantly more likely to report later onset.
Phenotypic differences exist between KRT6A (show KRT6A Antibodies) and KRT16 mutations support adoption of a new classification system.
Patients with p.Asn125Asp and p.Arg127Pro mutations in KRT16 exhibited more severe disease than patients carrying p.Asn125Ser and p.Arg127Cys mutations in terms of age of onset of symptoms, extent of nail (show CD244 Antibodies) involvement, and impact on daily quality of life.
we show that the loss of Krt16 function in mice causes the development of prominent calluses on the plantar side of front and hind paws, which significantly compromise mobility.
Keratin 6 and keratin 16 may play a role in wrinkle formation.
The protein encoded by this gene is a member of the keratin gene family. The keratins are intermediate filament proteins responsible for the structural integrity of epithelial cells and are subdivided into cytokeratins and hair keratins. Most of the type I cytokeratins consist of acidic proteins which are arranged in pairs of heterotypic keratin chains and are clustered in a region of chromosome 17q12-q21. This keratin has been coexpressed with keratin 14 in a number of epithelial tissues, including esophagus, tongue, and hair follicles. Mutations in this gene are associated with type 1 pachyonychia congenita, non-epidermolytic palmoplantar keratoderma and unilateral palmoplantar verrucous nevus.
, keratin 16
, Keratin, type I cytoskeletal 17
, cytokeratin 16
, focal non-epidermolytic palmoplantar keratoderma
, keratin, type I cytoskeletal 16
, keratin complex 1, acidic, gene 16
, keratin intermediate filament 16a
, keratin intermediate filament 16b
, keratin 14
, keratin 16 (focal non-epidermolytic palmoplantar keratoderma)
, type I keratin KA16