Macrophage Stimulating 1 (Hepatocyte Growth Factor-Like) (MST1) ELISA Kits

The protein encoded by MST1 contains four kringle domains and a serine protease domain, similar to that found in hepatic growth factor. Additionally we are shipping MST1 Antibodies (104) and MST1 Proteins (17) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
MST1 4485 P26927
MST1 24566  
MST1 15235 P26928
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Top MST1 ELISA Kits at antibodies-online.com

Showing 10 out of 49 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Supplier Delivery Price Details
Human 10pg/mL 156-10000 pg/mL Human MSP/MST1 PicoKine ELISA Kit standard curve 96 Tests Log in to see 4 to 6 Days
$369.00
Details
Mouse 1.47 pg/mL 3.125 pg/mL - 200 pg/mL 96 Tests Log in to see 15 to 17 Days
$891.06
Details
Rat 0.109 ng/mL 0.312 ng/mL - 20 ng/mL 96 Tests Log in to see 15 to 17 Days
$932.09
Details
Rabbit 37.5 pg/mL 62.5-4000 pg/mL   96 Tests Log in to see 12 to 14 Days
$715.00
Details
Pig 0.094 ng/mL 0.156-10 ng/mL   96 Tests Log in to see 12 to 14 Days
$715.00
Details
Chicken 0.094 ng/mL 0.156-10 ng/mL   96 Tests Log in to see 12 to 14 Days
$715.00
Details
Monkey 0.094 ng/mL 0.156-10 ng/mL   96 Tests Log in to see 12 to 14 Days
$715.00
Details
Guinea Pig
  96 Tests Log in to see 15 to 18 Days
$707.14
Details
Sheep
  96 Tests Log in to see 15 to 18 Days
$707.14
Details
Cow
  96 Tests Log in to see 15 to 18 Days
$707.14
Details

More ELISA Kits for MST1 Interaction Partners

Human Macrophage Stimulating 1 (Hepatocyte Growth Factor-Like) (MST1) interaction partners

  1. Legionella pneumophila effector protein LegK7(lpg1924) hijacks the conserved Hippo signaling pathway by molecularly mimicking host Hippo kinase (MST1 in mammals), which is the key regulator of pathway activation.

  2. Mst1 has an important role in inhibiting the growth of NSCLC in vitro and in vivo; its antiproliferative effect is associated with induction of apoptosis.

  3. These data suggest that MSP is an effective inhibitor of inflammation and lipid accumulation in the stressed liver, thereby indicating that MSP has a key regulatory role in non-alcoholic steatohepatitis.

  4. Identify MST1/MSP as a mitogen for tracheal basal cells.

  5. MSP appears to promote the migration of fibroblasts, enhances collagen synthesis and remodeling, and effectively improves wound healing.

  6. Elevated serum levels of MST1 were found in subjects with excessive alcohol use.

  7. These results support the hypothesis that the alpha-chain of MSPalphabeta mediates RON dimerization.

  8. functional consequences of the macrophage stimulating protein 689C inflammatory bowel disease risk allele

  9. Results suggest that the [AA] genotype of the common MST1 variant rs3197999 enhances genetic risk of sporadic extrahepatic cholangiocarcinoma irrespective of primary sclerosing cholangitis status.

  10. MSP may be one of the major determinants that affects the properties of tumor stroma and that produces a permissive microenvironment to promote cancer metastasis

  11. The present results refine the known genetic architecture in primary sclerosing cholangitis by confirming MST1 locus association.

  12. the missense SNP impairs MSP function by reducing its affinity to RON and perhaps through a secondary effect on in vivo concentration arising from reduced thermodynamic stability, resulting in down-regulation of the MSP/RON signaling pathway.

  13. HAT cleaves proMSP at the physiological activation site

  14. Studies indicate that the cylindromatosis/turban tumor syndrome gene (CYLD) ranked highest, followed by acylaminoacyl-peptidase (APEH), dystroglycan (DAG1), macrophage-stimulating protein (MST1) and ubiquitin-specific peptidase 4 (USP4).

  15. These findings suggest that the MSP/RON signaling pathway may be regulated by hepsin in tissue homeostasis and in disease pathologies, such as in cancer and immune disorders.

  16. considerable number of Merkel cell carcinoma cases expressed both RON and MSP, while Merkel cells do not express these molecules

  17. MSP-induced RSK2 activation is a critical determinant linking RON signaling to cellular EMT program.

  18. the oncogenic effect of BRAF(V600E) is associated with the inhibition of MST1 tumor suppressor pathways, and that the activity of RASSF1A-MST1-FoxO3 pathways determines the phenotypes of BRAF(V600E) tumors.

  19. results suggest that MSP induces uPAR expression via MAPK, AP-1 and NF-kappaB signaling pathways

  20. These results suggest that activation of RON by macrophage-stimulating protein inhibits LPS-induced macrophage Cox-2 expression.

Mouse (Murine) Macrophage Stimulating 1 (Hepatocyte Growth Factor-Like) (MST1) interaction partners

  1. our results implicate Mst1 upregulation as a critical step in exacerbating HFD-mediated hepatic injury. Increased Mst1 blocks the AMPK pathway and thus diminishes Parkin expression, repressing mitophagy and consequently activating mitochondrial apoptosis in HFD-treated livers.

  2. data revealed that OSM alleviated postinfarction cardiac remodeling and dysfunction by enhancing cardiomyocyte autophagy. OSM holds promise as a therapeutic target in treating HF after MI through Obeta receptor by inhibiting Mst1 phosphorylation.

  3. The results identify Mst1 as a malefactor in the development of post-infarction cardiac injury and that it acts through the JNK-Drp1-mitochondrial fission pathway.

  4. The results suggest that melatonin alleviates cardiac remodeling and dysfunction in diabetic cardiomyopathy by upregulating autophagy, limiting apoptosis, and modulating mitochondrial integrity and biogenesis. The mechanisms are associated with Mst1/Sirt3 signaling.

  5. These data suggest that MSP is an effective inhibitor of inflammation and lipid accumulation in the stressed liver, thereby indicating that MSP has a key regulatory role in non-alcoholic steatohepatitis.

  6. kinases, including Slk, Lok and Mst1, are inhibited by BI-D1870 but to a much lesser extent by BIX 02565 and that phosphorylation of some of their substrates is blocked by BI-D1870 in living cells.

  7. Nicorandil alleviates post-MI cardiac dysfunction and remodeling. The mechanisms were associated with enhancing autophagy and inhibiting apoptosis through Mst1 inhibition.

  8. Using a standard two-thirds partial hepatectomy (PH) model in young and aged mice, the activity of the core kinases MST1 and LATS1 increased during the early hypertrophic phase and returned to steady state levels in the proliferative phase, coinciding with activation of YAP1 target genes and hepatocyte proliferation.

  9. The MST1 acts as a molecular brake to maintain immune tolerance by regulating T cell-mediated B cell activation.

  10. Mst1 knockout alleviated while Mst1 overexpression aggravated cardiac dysfunction in diabetes.

  11. these findings highlight a role for MST1 in vesicle trafficking and extravasation in neutrophils, providing an additional mechanistic explanation for the severe immune defect

  12. Results identify L-plastin as a key effector of Mst1 and establish a novel mechanism linking a signaling intermediate to an actin-binding protein critical to T cell migration.

  13. MIST1 is a scaling factor necessary and sufficient by itself to induce and maintain secretory cell architecture

  14. Mst1 deficiency diminishes atherosclerosis and stabilizes atherosclerotic plaques in ApoE(-/-) mice. Mst1 may participate in atherosclerosis progression through inhibition of macrophage autophagy and promotion of macrophage apoptosis.

  15. These results suggest that melatonin alleviates postinfarction cardiac remodeling and dysfunction by upregulating autophagy, decreasing apoptosis, and modulating mitochondrial integrity and biogenesis. The attributed mechanism involved, at least in part, Mst1/Sirt1 signaling

  16. TRAF2 functions as a key activator of MST1 in oxidative stress-induced intracellular signaling processes.

  17. Mst1 regulates hepatic lipid metabolism by inhibiting Sirt1 ubiquitination in mice.

  18. Mst1 enhances Foxp3 expression and Treg function at the post-translational level.

  19. our study demonstrates the existence of a direct crosstalk between mTORC2 and MST1 that is critical for cardiac cell survival and growth.

  20. Mst1 regulates Myosin IIa dynamics to organize high and low affinity LFA-1 to the anterior and posterior membrane during T cell migration.

MST1 Antigen Profile

Antigen Summary

The protein encoded by this gene contains four kringle domains and a serine protease domain, similar to that found in hepatic growth factor. Despite the presence of the serine protease domain, the encoded protein may not have any proteolytic activity. The receptor for this protein is RON tyrosine kinase, which upon activation stimulates ciliary motility of ciliated epithelial lung cells. This protein is secreted and cleaved to form an alpha chain and a beta chain bridged by disulfide bonds.

Gene names and symbols associated with MST1

  • macrophage stimulating 1 (MST1) antibody
  • macrophage stimulating 1 (Mst1) antibody
  • macrophage stimulating 1 (hepatocyte growth factor-like) (Mst1) antibody
  • macrophage stimulating 1 (hepatocyte growth factor-like) (MST1) antibody
  • macrophage stimulating 1 L homeolog (mst1.L) antibody
  • d3f15s2 antibody
  • D3F15S2h antibody
  • D9H3F15S2 antibody
  • dnf15s2 antibody
  • DNF15S2h antibody
  • E1A antibody
  • E2F2 antibody
  • HGF1/MSP antibody
  • hgfl antibody
  • msp antibody
  • mst1 antibody
  • nf15s2 antibody
  • XHL antibody

Protein level used designations for MST1

hepatocyte growth factor-like protein , hepatocyte growth factor-like protein homolog , macrophage-stimulating protein , D8h3f15s2 , E2F transcription factor 2 , MSP , macrophage stimulatory protein , hepatocyte growth factor-like/macrophage stimulating protein , macrophage stimulating 1 (hepatocyte growth factor-like) , 12S E1A , macrophage stimulating 1 L homeolog

GENE ID SPECIES
4485 Homo sapiens
24566 Rattus norvegicus
15235 Mus musculus
484768 Canis lupus familiaris
514667 Bos taurus
396135 Gallus gallus
100734937 Cavia porcellus
100512987 Sus scrofa
379214 Xenopus laevis
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