Mannose Receptor, C Type 1 (MRC1) ELISA Kits

Human Mannose Receptor, C Type 1 (MRC1) interaction partners

1. Loss of antral CD206-positive anti-inflammatory macrophages is a key feature in human gastroparesis and it is associates with interstitial cells of Cajal loss.

2. The effect of LPS in the expression of CD163 and CD206 on monocytes is not reverted in LPS tolerant cells, and the inhibition of inflammatory cytokines in tolerant cells is not related with modulation of these receptors

3. regulatory effect of the mannose receptor (MR) was mediated by a direct interaction with CD45 (show PTPRC ELISA Kits) on the T cell, inhibiting its phosphatase activity, which resulted in up-regulation of CTLA-4 (show CTLA4 ELISA Kits) and the induction of T-cell tolerance. Inhibition of CD45 (show PTPRC ELISA Kits) prevented expression of B-cell lymphoma 6 (Bcl-6 (show BCL6 ELISA Kits)), a transcriptional inhibitor that directly bound the CTLA-4 (show CTLA4 ELISA Kits) promoter and regulated its activity

4. Mannose Receptor and CLEC5A (show CLEC5A ELISA Kits) on Macrophage first attracts the Dengue Virus with high avidity, and the virus concurrently interacts with CLEC5A (show CLEC5A ELISA Kits) in close proximity to form a multivalent hetero-complex and facilitate CLEC5A (show CLEC5A ELISA Kits)-mediated signal transduction. Dengue Virus Infection Is through a Cooperative Interaction between a Mannose Receptor and CLEC5A (show CLEC5A ELISA Kits) on Macrophage as a Multivalent Hetero-Complex.

5. Our work demonstrates a key role for MR in the modulation of the TLR4 (show TLR4 ELISA Kits)-AhR (show AHR ELISA Kits)-IDO (show IDO1 ELISA Kits) axis, which has a significant effect on DC behavior and the development of immune responses against allergens.

6. MRC1 and S100A9 (show S100A9 ELISA Kits) may contribute to the determination of the mechanisms and screening involved in CRC (show CALR ELISA Kits).

7. Findings show that CD206 was highly expressed in the hepatocellular carcinoma (HCC) tissues and indicate that CD206 is a potential biomarker for predicting HCC prognosis.

8. Data show that umbilical cord-derived mesenchymal stem cells (UCMSCs) transplantation promoted the expression of CD206 antigen and increased phagocytic activity of macrophages through interleukin-6 (IL-6 (show IL6 ELISA Kits)) in systemic lupus erythematosus (SLE) patients.

9. Data indicate that mannose receptor (CD206) is a major binding receptor for gamma-tilmanocept.

10. CD206 is a novel biomarker of alternative macrophage activation and treatment outcome in multiple myeloma.

Mouse (Murine) Mannose Receptor, C Type 1 (MRC1) interaction partners

1. current study shows that Mrc1-/- mice develop less severe dextran sulfate sodium-induced colitis

2. the key roles of the ManR and ASGR (show ASGR1 ELISA Kits) in controlling circulating levels of numerous glycoproteins critical for regulating reproductive hormones and blood coagulation.

3. PLA2 (show PLA2G2A ELISA Kits) may prevent inflammatory responses in cisplatin-induced acute kidney injury by modulating Treg cells and IL-10 (show IL10 ELISA Kits) through the CD206 mannose receptor.

4. Hepatic CD206-positive macrophages express amphiregulin (show AREG ELISA Kits) to promote the immunosuppressive activity of regulatory T cells in HBV infection.

5. CD206-positive pulmonary macrophages have a critical role in triggering inflammatory cascade during endotoxemic lung inflammation

6. Syk was involved in the CLR-mediated activation of mast cells by BP

7. Genetic ablation of the Mrc1 impaired the intracellular collagen degradation pathway.

8. Report CD44 (show CD44 ELISA Kits)/macrophage mannose receptor as a ligand/receptor pair involved in the process of lymphocyte transit via the lymphatic vasculature.

9. study reports the macrophage response against Leishmania infantum in vivo is characterized by an M2b-like phenotype and C-type lectin receptors signature composed of Dectin-1, mannose receptor (MR)and the DC-SIGN homolog SIGNR3 expression

10. These findings suggest that MRC1 plays an important role in the uptake of various bacteria by trophoblast giant cells and may provide an opportunity for those bacteria to escape from phagosomes.