Mesoderm Specific Transcript (MEST) ELISA Kits

MEST encodes a member of the alpha/beta hydrolase superfamily. Additionally we are shipping MEST Antibodies (45) and MEST Proteins (4) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
MEST 17294 Q07646
MEST 58827 Q6P5P5
MEST 4232 Q5EB52
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Top MEST ELISA Kits at

Showing 4 out of 5 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Delivery Price Details
Mouse 7.81 pg/mL 31.25-2000 pg/mL Typical standard curve 96 Tests 15 to 18 Days
  96 Tests 15 to 18 Days
  96 Tests 15 to 18 Days
  96 Tests 15 to 18 Days

More ELISA Kits for MEST Interaction Partners

Mouse (Murine) Mesoderm Specific Transcript (MEST) interaction partners

  1. Identification of MEST as an endoplasmic reticulum-specific protein that co-localizes with lipid droplets in cells undergoing adipogenic differentiation supports a function for MEST in the facilitation of lipid accumulation and storage in adipocytes.

  2. This study demonstrated that the Mest and its intron product, miR-335, play important roles in neuronal migration with Mest regulating the morphological transition of primary neurons required in the formation of the mammalian neocortex.

  3. diet-induced ATE in MEST-deficient mice diminishes hypoxia and inflammation in WAT leading to improved glucose tolerance and insulin sensitivity

  4. The androgenetic patterns of H19, Snrpn, and Mest were maintained even after differentiation of germline-derived pluripotent stem cells(gPS) into neural stem cells(NSC), whereas the fully unmethylated status of Ndn in SSCs was altered to somatic patterns in gPS cells and gPS-NSCs.

  5. The balance of factors controlling fat deposition can be evaluated in part by the differential expression profiles of Mest and Sfrp5 genes with functions linked to fat deposition as long as there is an active accumulation of fat mass.

  6. the expression of Mest and Sfrp5 were tightly associated across the 5 mouse strains with the highest and lowest expression occurring in DBA/2J and C57BL/6J (B6) respectively suggesting a common mechanism for their regulation.

  7. Although both Mest and miR-335 are highly expressed during muscle development and regeneration, only Mest+/- (maternal/paternal) mice show retardation of body growth.

  8. The paternally expressed imprinted gene Mest was only detected in haploid androgenetic blastocysts and was significantly lower in this group relative to the controls, indicating that the gene maintains expression patterns specific to its parental origin.

  9. analysis of modifications of H3 histone in H19 and MEST imprinted genes in blastocysts produced in vitro from non-vitrified and vitrified two-cell embryos

  10. Results suggest that at least one allele of Mest expression is required in the somatic tissues of adult individuals and that under certain conditions (such as in the presence of a Mest insertional mutation or in an altered genetic background), somatically acquired alterations of allelic expression at the Mest locus may occur.

  11. The data showed that Mest expression was regulated through cAMP-dependent protein kinase A pathways during differentiation of preadipocytes into adipocytes in vitro, supporting the critical role of Mest in proliferation and differentiation of adipocytes.

  12. The identification of new imprinted RNA products at the Mest locus, longer variants of the RNA, called MestXL, transcribed >10 kb into the downstream antisense gene Copg2.

  13. Mest/Peg1 is a novel down-regulator of Wnt/beta-catenin signalling during adipogenic differentiation.

  14. Mest/Peg1 gene is imprinted, with preferential expression from the paternal allele.

  15. These data suggest that mechanisms independent of the CpG methylation status of the Mest promoter must underlie the control of its expression during adipose tissue expansion.

  16. MEST, a gene with a putative mesenchymal cell-derived protein, conceivably plays a role in mammalian metanephric development

  17. Peg1 is expressed in the mouse developing myocardial trabeculae of both the atria and ventricles

  18. decreased expression in high glucose ambience or diabetic state did not follow reciprocal inactivation/activation of imprinted genes, and may be responsible for the perturbed epithelial:mesenchymal interactions and dysmorphogenesis of the metanephros.

  19. loss of imprinting is correlated with increased body weight and increased weight of two of the organs tested, kidney and spleen

  20. Mest appears to enlarge adipocytes

Human Mesoderm Specific Transcript (MEST) interaction partners

  1. Imprinting methylation in SNRPN and MEST1 in adult blood predicts cognitive ability.

  2. Study provides evidence that MEST mediates the impact of prenatal bisphenol A exposure on long-term body weight development in offspring by triggering adipocyte differentiation.

  3. We conclude methylation changes at some CpG sites of MEST and DLK differentially methylated regions in preeclamptic group

  4. Some growth-regulating imprinted genes such as MEST and MEG3, are susceptible to non-imprinted allele during development and differentiation, whereas the intergenic differentially methylated region of others (i.e. PEG3) are strictly maintained.

  5. strongly expressed in invasive extravillous trophoblasts during the first trimester

  6. G4 formation at motifs not previously identified through bioinformatic analysis of the MEST promoter, is reported.

  7. altered DNA methylation at imprinted domains including IGF2/H19 and PEG1/MEST may mediate the association between human papillomavirus infection and invasive cervical cancer

  8. The expression levels of miR-335 significantly correlated with those of MEST, supporting the notion that the intronic miR-335 is co-expressed with its host gene

  9. DNA methylation level at the H19 and MEST differentially methylated regions (DMRs)is reduced in placentas from pregnancies conceived by IVF/ICSI when compared with placentas from spontaneous conception.

  10. Paternal methylation aberrations at imprinting control regions of DLK1-GTL2, MEST (PEG1), and ZAC (PLAGL1) and global methylation levels are not associated with idiopathic recurrent spontaneous miscarriages.

  11. These results support the idea that intrauterine exposure to gestational diabetes mellitus has long-lasting effects on the epigenome of the offspring.

  12. Data indicate that ARMCX2, COL1A1, MDK, MEST and MLH1 genes acquired methylation in drug-resistant ovarian cancer-sustaining (side population) cells.

  13. MEST showed tissue-specific imprinting, being paternally expressed in skeletal muscle, fat, pituitary gland, heart, kidney, lung, stomach and uterus, and maternally expressed in spleen and liver.

  14. In cortices, the MEST promoter was hemimethylated, as expected for a differentially methylated imprinting control region, whereas the COPG2 and TSGA14 promoters were completely demethylated, typical for transcriptionally active non-imprinted genes.

  15. Regardless of conception method, the PEG1 methylation percentage in chorionic villus from spontaneous abortions is significantly higher than in villus from induced abortions and multifetal reduction.

  16. data demonstrated that tumorigenesis of leiomyoma is associated with overexpression of isoform 1 of PEG1/MEST gene, but not with loss of imprinting of the gene

  17. MEST gene is imprinted in an isoform-specific manner in adult lymphocytes.

  18. MEST gene is imprinted, with preferential expression from the paternal allele in fetal tissues.

  19. Findings suggest that PEG1/MEST can be excluded as a major determinant of Silver-Russell syndrome.

  20. An imprinted PEG1/MEST antisense expressed predominantly in human testis and in mature spermatozoa.

MEST Antigen Profile

Antigen Summary

This gene encodes a member of the alpha/beta hydrolase superfamily. It is imprinted, exhibiting preferential expression from the paternal allele in fetal tissues, and isoform-specific imprinting in lymphocytes. The loss of imprinting of this gene has been linked to certain types of cancer and may be due to promotor switching. The encoded protein may play a role in development. Alternatively spliced transcript variants encoding multiple isoforms have been identified for this gene. Pseudogenes of this gene are located on the short arm of chromosomes 3 and 4, and the long arm of chromosomes 6 and 15.

Gene names and symbols associated with MEST

  • mesoderm specific transcript (Mest) antibody
  • mesoderm specific transcript (MEST) antibody
  • AA408879 antibody
  • AI256745 antibody
  • PEG1 antibody

Protein level used designations for MEST

mesoderm-specific transcript protein , paternally-expressed gene 1 protein , mesoderm specific transcript homolog , mesoderm-specific transcript homolog protein

17294 Mus musculus
58827 Rattus norvegicus
4232 Homo sapiens
607717 Canis lupus familiaris
404180 Bos taurus
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