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Core subunit of the mitochondrial membrane respiratory chain NADH dehydrogenase (Complex I) that is believed to belong to the minimal assembly required for catalysis. Additionally we are shipping Mitochondrially Encoded NADH Dehydrogenase 5 Antibodies (59) and Mitochondrially Encoded NADH Dehydrogenase 5 Proteins (4) and many more products for this protein.
Showing 7 out of 12 products:
Human MT-ND5 ELISA Kit for Sandwich ELISA - ABIN1874498
Zhang, Diao, Duan, Yi, Cai: In vitro antioxidant effect of curcumin on human sperm quality in leucocytospermia. in Andrologia 2017
Data suggest that mutations in MT-CO2 (show COX2 ELISA Kits) and MT-ND5 can be involved in MIDD (maternally inherited diabetes and deafness); a Tunisian family (mother, daughter, son) with clinical features of MIDD associated with retinopathy exhibit mutations in MT-CO2 (show COX2 ELISA Kits) (m.8241T>G - p. F219C) and MT-ND5 (m.13276G>A - p. M314V); these two mutations could explain retinopathy in some family members. (MT-CO2 (show COX2 ELISA Kits) = cytochrome c oxidase subunit II (show COX2 ELISA Kits))
A MELAS/Leigh syndrome phenotype caused by a mtDNA mutation [m.13513G>A; p.Asp393Asn] in the ND5 gene encoding the ND5 subunit of respiratory chain complex I was identified in a patient in a second family
FASTKD4 is required to promote expression of ND5. FASTKD4 may promote processing of ND5-CYB (show CSTB ELISA Kits) precursor or it may stabilize multiple mature RNAs including ND5.
The m.13565C>T mutation in MTND5 causes defects in both mitochondrial oxidative metabolism and mitochondrial calcium sequestration in a MELAS syndrome fibroblasts.
Mitochondrially encoded NADH dehydrogenase subunit, complex I extracts energy from NADH, produced by the oxidation of sugars and fats, and traps the energy in a potential difference or voltage across the mitochondrial inner membrane.
Study demonstrates a link between p53 (show TP53 ELISA Kits) and Bcl-2 (show BCL2 ELISA Kits) proteins as regulators of ROS (show ROS1 ELISA Kits) production and cellular invasiveness, and reveals complex-I, especially ND5, as their functional target in lung tumor cells.
Three mutations were significantly related to the presence of epilepsy. These mutations were found at the 8502, 11994, and 13,231 bp of mtDNA, which resulted in amino acid changes at the MT-ATP-8, MT-ND4 and MT-ND5 genes.
Point mutations m.10191T>C in mitochondrial ND3 gene, m.13513G>A in ND5 gene and m.14,453G>A in ND6 (show MT-ND6 ELISA Kits) gene were detected in three Chinese children with Leigh synrome dur to complex I deficiency.
Mitochondrial ND5 12338T>C variant is associated with maternally inherited hypertrophic cardiomyopathy in a Chinese pedigree
Mitochondrial DNA mutations in respiratory complex-I in never-smoker lung cancer patients contribute to lung cancer progression in association with EGFR (show EGFR ELISA Kits) gene mutation.
Core subunit of the mitochondrial membrane respiratory chain NADH dehydrogenase (Complex I) that is believed to belong to the minimal assembly required for catalysis. Complex I functions in the transfer of electrons from NADH to the respiratory chain. The immediate electron acceptor for the enzyme is believed to be ubiquinone (By similarity).
NADH dehydrogenase subunit 5