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The protein encoded by Prok1 induces proliferation, migration, and fenestration (the formation of membrane discontinuities) in capillary endothelial cells derived from endocrine glands.
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Mouse (Murine) Prokineticin 1 ELISA Kit for Sandwich ELISA - ABIN415589
Xu, Wang, Wang, Liu, Wang, Huang, Tan: Hypoxia-induced secretion of IL-10 from adipose-derived mesenchymal stem cell promotes growth and cancer stem cell properties of Burkitt lymphoma. in Tumour biology 2016
important role for PK-1 in luteal function by acting as a mitogen and survival factor in corpus luteum derived endothelial cells
PROK1, acting via PROKR1 (show PROKR1 ELISA Kits), may be involved in the recruitment of monocytes to regressing CL
EG-VEGF/PK1 and Bv8/PK2 (show PROK2 ELISA Kits) thus represent new regulatory peptides acting as autocrine mitogens for endocrine cells
The VEGF (show VEGFA ELISA Kits)/sVEGF-R1 ratio in follicular fluid on the day of oocyte retrieval in women undergoing IVF procedure, regardless of the type of stimulation protocol, might predict the risk of developing ovarian hyperstimulation syndrome (OHSS). To the best of our knowledge this is the first paper in the literature to show interplay among VEGF (show VEGFA ELISA Kits), EG-VEGF and sVEGF-R1 and the correlation between their concentration and OHSS risk.
These findings demonstrate a novel function of primary cilia in controlling EG-VEGF-regulated trophoblast invasion and reveal the underlying molecular mechanism.
miR (show MLXIP ELISA Kits)-346 and miR (show MLXIP ELISA Kits)-582-3p regulate EG-VEGF-induced trophoblast invasion through repressing MMP 2 (show MMP2 ELISA Kits) and MMP 9 (show MMP9 ELISA Kits), and may become novel diagnostic biomarkers or therapeutic targets for EG-VEGF-related obstetric disorders. (c) 2016 BioFactors, 43(2):210-219, 2017.
Each follicular fluid (FF) was individually aspirated and FF/serum EG-VEGF, inhibin-a (show INHA ELISA Kits), and FF IGF-1 (show IGF1 ELISA Kits) levels were evaluated. The pregnant group was characterized by increased numbers of WVFs (p = 0.044), a WVE (p = 0.022), and increased levels of FF IGF-1 (show IGF1 ELISA Kits) (p = 0.001) and serum EG-VEGF (p = 0.03).
Results show the biological effects of PROK1-V67I on cell functions are similar to those of wild type, and the common variant of V67I may act as a modifier in the PROK1-PROKR system through down-regulation of PROK1 expression.
Data suggest that prokineticins (PROK1 and PROK2 (show PROK2 ELISA Kits)) and prokineticin receptors (PROKR1 (show PROKR1 ELISA Kits) and PROKR2 (show PROKR2 ELISA Kits)) act as main regulators of physiological functions of ovary, uterus, placenta, and testis. [REVIEW]
EG-VEGF and its receptor PKR1 (show PROKR1 ELISA Kits) might play a role in the pathogenesis of adrenocortical tumors and could serve as prognostic markers for this rare malignant disease.
The prognosis was poorer in colorectal cancers that expressed both PROK1 and VEGF (show VEGFA ELISA Kits) relative to the cases that expressed only 1 protein, and the expression of both proteins was found to be an independent prognostic factor.
PROK1 levels in follicular fluid and fertilization culture media could constitute new predictive noninvasive markers of successful embryo implantation in conventional in vitro fertilization-embryo transfer
Simultaneous targeting of both angiogenic growth factors (VEGF (show VEGFA ELISA Kits)/PROK1) may prove more useful in colorectal cancer
Clinical studies strongly suggested the potential involvement of the new angiogenic factor (show VEGFA ELISA Kits) EG-VEGF in pregnancy-induced hypertension. Here is described the protocol that served to demonstrate that maintenance of EG-VEGF production over 11.5 days post coitus (dpc) in the gravid mice caused the development of pregnancy-induced hypertension.
EG-VEGF is a new mediator of PPARgamma (show PPARG ELISA Kits) effects during pregnancy and bring new insights into the fine mechanism of trophoblast invasion.
Bv8 (show PROK2 ELISA Kits) and EG-VEGF, along with other factors such as VEGF-A (show VEGFA ELISA Kits), may maintain the integrity and also regulate proliferation of the blood vessels in the testis
This is the first report demonstrating that Prok-1 acts as a gut (show GUSB ELISA Kits) mucosa/mesenchyme-derived factor and maintains proliferation and differentiation of enteric neural crest cells.
EG-VEGF was mainly localised in the labyrinth in mouse placenta. It may have direct effect on both endothelial & trophoblastic cells & is likely to play an important role in mouse placentation.
Prokineticin-1 (Prok-1) works coordinately with glial cell line-derived neurotrophic factor (GDNF) to mediate proliferation and differentiation of enteric neural crest cells.
The protein encoded by this gene induces proliferation, migration, and fenestration (the formation of membrane discontinuities) in capillary endothelial cells derived from endocrine glands. It has little or no effect on a variety of other endothelial and non-endothelial cell types. Its expression is restricted to the steroidogenic glands (ovary, testis, adrenal, and placenta), is induced by hypoxia, and often complementary to the expression of vascular endothelial growth factor (VEGF), suggesting that these molecules function in a coordinated manner.
, black mamba toxin-related protein
, endocrine-gland-derived vascular endothelial growth factor