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Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases.
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Oncostatin M (show OSM ELISA Kits) induces RIG-I (show DDX58 ELISA Kits) and MDA5 (show IFIH1 ELISA Kits) expression and enhances the double-stranded RNA response in fibroblasts.
Identified the tumor suppressor RARRES3 as a critical target of G9a (show EHMT2 ELISA Kits). Epigenetic silencing of RARRES3 contributed to the tumor-promoting function of G9a (show EHMT2 ELISA Kits)
Results provide evidence that RIG-I (show DDX58 ELISA Kits) as an essential mediator for influenza A virus-induced COX-2 (show COX2 ELISA Kits) expression.
perifascicular pattern of RIG-I (show DDX58 ELISA Kits) expression supports the diagnosis of dermatomyositis
The current study establishes that hypoxia can profoundly influence the inducible RIG-I (show DDX58 ELISA Kits) protein expression in malignant cells of both human and murine origin, whereas this phenomenon was not identified in nonmalignant cell lines or primary cells.
our study demonstrates that the novel pathway lncRNA Ftx/miR (show MLXIP ELISA Kits)-545/RIG-I (show DDX58 ELISA Kits) promotes hepatocellular carcinoma development
this review describes antiviral activities of RIG-I (show DDX58 ELISA Kits) against influenza viruses (standing on three legs)
Study uncovered a novel aspect of Rig-I (show DDX58 ELISA Kits) in monitoring gut (show GUSB ELISA Kits) microbiota through regulating IgA (show IgA ELISA Kits) and IL6 (show IL6 ELISA Kits)-STAT3 (show STAT3 ELISA Kits)-dependent Reg3gamma pathway. Besides, Rig-I (show DDX58 ELISA Kits) loss could also promote colorectal cancer progression both in the presence and absence of intestinal bacteria.
this study indicated that increased expression of TIG3 in primary glioblastoma is a novel biomarker for predicting poor outcome of patients. We then hypothesize that TIG3 may function in a different pattern in glioblastoma
Foot-and-mouth disease virus Viroporin 2B antagonizes RIG-I (show DDX58 ELISA Kits)-mediated antiviral effects by inhibition of its protein expression.
Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. These effects are mediated by specific nuclear receptor proteins that are members of the steroid and thyroid hormone receptor superfamily of transcriptional regulators. RARRES1, RARRES2, and RARRES3 are genes whose expression is upregulated by the synthetic retinoid tazarotene. RARRES3 is thought act as a tumor suppressor or growth regulator.
HRAS-like suppressor 4
, RAR-responsive protein TIG3
, retinoic acid receptor responder protein 3
, retinoic acid-inducible gene 1
, retinoid-inducible gene 1 protein
, tazarotene-induced gene 3 protein
, retinoic acid receptor responder (tazarotene induced) 3