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SCGB1A1 encodes a member of the secretoglobin family of small secreted proteins.
Showing 10 out of 126 products:
Human Monoclonal SCGB1A1 Primary Antibody for FACS, ELISA - ABIN1098131
Paantjens, Otten, van Ginkel, van Kessel, van den Bosch, Kwakkel-van Erp, van de Graaf: Clara cell secretory protein and surfactant protein-D do not predict bronchiolitis obliterans syndrome after lung transplantation. in Transplantation 2010
Show all 2 Pubmed References
Human Monoclonal SCGB1A1 Primary Antibody for ELISA - ABIN563338
Archer, Abi-Rizk, Desloire, Dolmazon, Gineys, Guiguen, Cottin, Mornex, Leroux: Lung progenitors from lambs can differentiate into specialized alveolar or bronchiolar epithelial cells. in BMC veterinary research 2014
Mouse (Murine) Polyclonal SCGB1A1 Primary Antibody for IF (p), IHC (p) - ABIN687262
Mori, Sasaki, Makabe, Yoshinari, Yajima: The genes Scgb1a1, Lpo and Gbp2 characteristically expressed in peri-implant epithelium of rats. in Clinical oral implants research 2015
Our data suggest that rCC16 suppresses LPS (show IRF6 Antibodies)-mediated inflammatory mediator TNF-alpha (show TNF Antibodies), IL-6 (show IL6 Antibodies), and IL-8 (show IL8 Antibodies) production by inactivating NF-kappaB (show NFKB1 Antibodies) and p38 MAPK (show MAPK14 Antibodies) but not AP-1 (show FOSB Antibodies) in RAW264.7 cells.
Lung-specific (CC-16) and novel (RelB (show RELB Antibodies)) biomarkers are associated with systemic cardiovascular changes over time. CC-16 can predict subsequent exacerbations in subjects with severe COPD (show ARCN1 Antibodies) and may be an important biomarker of pulmonary and systemic stress in COPD (show ARCN1 Antibodies).
Smokers and COPD patients had reduced airway CC16 immunostaining that decreased with in (show NFKB1 Antibodies)creasing COPD severity.
the uteroglobin G38A gene polymorphism was not associated with IgAN risk
In chromium-exposed workers, blood levels of CC16, and CC16/SP-D (show SFTPD Antibodies) were lower than in controls. Positive relationships were shown between CC16 or CC16/SP-D (show SFTPD Antibodies) and indicators of lung function.
Increased plasma clara cell secretory protein levels are associated with primary graft dysfunction.
Uteroglobin is a possible ligand of the lipoxin receptor and it may have a role in inhibiting serum amyloid A-driven inflammation
role of Secretoglobin (show SCGB3A1 Antibodies) 1A1 (show SLC45A2 Antibodies) and 1A1A in neutrophil reactive oxygen species production, phagocytosis and extracellular trap formation [secretoglobin (show SCGB3A1 Antibodies) 1A1 (show SLC45A2 Antibodies)][secretoglobi 1A1A]
In the oviduct tissue, a decrease in the level of SCGB1A1 mRNA expression was observed from 144 h post-ovulation.In the endometrium a significant decline in the level of SCGB1A1 mRNA expression from 16 to 144 h post-ovulation induction was observed.
This study extends the function of the Scgb1a1-expressing epithelial cell population as a major mediator of the antiviral response. It
CC16 protects lungs from cigarette smoke (CS)-induced injury by reducing lung NF-kappaB (show NFKB1 Antibodies) activation. CS-induced airway CC16 deficiency increases CS-induced pulmonary inflammation and injury and likely contributes to the pathogenesis of COPD (show ARCN1 Antibodies).
Suggest role for Scgb1a1(+) cells in the generation of p63 (show CKAP4 Antibodies)(+) cells in the damaged lung parenchyma.
Serum CC-16 is associated with disease progression in chronic obstructive pulmonary disease (COPD (show ARCN1 Antibodies)). However, the absence of CC-16 does not appear to modify the risk of cigarette-related COPD (show ARCN1 Antibodies) in mice.
the role of clara cells and clara cell secretory protein in lung radiation injury exacerbated by viral infection
AAV2/9-CC10 vector virus guaranteed sufficient CC10 expression and had an anti-inflammatory effect in asthmatic mice.
Claudin-3 (show CLDN3 Antibodies) and Clara cell 10 kDa protein were identified as possible markers of early tobacco smoke-induced epithelial injury along alveolar ducts.
Scgb1a1-expressing cells, most likely Clara cells, are a major cell type that gives rise to alveolar type I and II cells during the regeneration of alveolar epithelia in response to severe pulmonary damage in mice.
These results indicate that CC10 gene transfer may inhibit airway inflammation through suppressing the activation of NF-kappaB (show NFKB1 Antibodies).
Clara cell secretory protein expression is elevtaed in lungs after long-term n-hexane inhalation exposure.
This gene encodes a member of the secretoglobin family of small secreted proteins. The encoded protein has been implicated in numerous functions including anti-inflammation, inhibition of phospholipase A2 and the sequestering of hydrophobic ligands. Defects in this gene are associated with a susceptibility to asthma.
, clara cell phospholipid-binding protein
, clara cells 10 kDa secretory protein
, urinary protein 1
, urine protein 1
, secretoglobin family 1A member 1
, PCB-binding protein
, clara cell 17 kDa protein
, clara cell secretory protein
, Uteroglobin (Clara cell secretory protein)
, secretoglobin, family 1A, member 1 (uteroglobin)