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Sodium-dependent amino acid/proton antiporter. Additionally we are shipping Solute Carrier Family 38 Member 3 Proteins (3) and and many more products for this protein.
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SLC38A3 activated PDK1 (show PDK1 Antibodies)/AKT (show AKT1 Antibodies) signaling and promoted metastasis of non-small cell lung cancer cells through regulating glutamine (show GFPT1 Antibodies) and histidine transport.
The regulation of SNAT3 gene expression by extracellular pH involves post-transcriptional and transcriptional mechanisms, the latter being distinct from the mechanisms that control the tissue-specific expression of the gene.
SNAT3 is expressed in the placenta in the early stage of pregnancy.
SN1 is a target for the ubiquitin ligase Nedd4-2 (show NEDD4L Antibodies), which is inactivated by the serum and glucocorticoid inducible kinase SGK1 (show SGK1 Antibodies), its isoform SGK3 (show SGK3 Antibodies), and protein kinase B (show AKT1 Antibodies).
SNAT3 mRNA showed a 3-5 times stronger expression in gliomas than in metastases or control tissue, and was virtually absent from glioma cultures. Native glioblastoma immunostained positively with anti-SNAT3 antibody.
Transcription of the SLC38A3 gene was impaired in all 5 RCC (show XRCC1 Antibodies) cell lines analyzed. Our data indicate this gene as a putative tumour suppressor gene.
This indicates a role for PKC (show PKC Antibodies) in dynamically controlling the trafficking of SNAT3 transporters in astrocytes in situ.
SNAT3 is required for amino acid homeostasis and determines critical functions in various organs. Loss of Snat3 cannot be compensated.
acid loading increased urinary ammonium excretion in all groups, but to a lesser extent in the O-GI group. SNAT3 mRNA expression was enhanced in both obese groups
Regulation of SNAT3 gene expression by extracellular pH involves post-transcriptional and transcriptional mechanisms, the latter being distinct from the mechanisms that control the tissue-specific expression of the gene
In this study, the in vivo endothelial membrane localization of the sodium-dependent glutamine (show GFPT1 Antibodies) transporters Snat3 (Slc38a3) and Snat1 (Slc38a1 (show SLC38A1 Antibodies)) was investigated in the mouse brain microvasculature.
differential regulation of SNAT3 by insulin (show INS Antibodies) and serum starvation reinforces the functional significance of this transporter in liver physiology
selective regulation of SNAT3 and y(+)LAT1 (show Slc7a7 Antibodies) expression may serve a major role in the renal adaptation to acid secretion and thus for systemic acid-base balance.
ISH (show ANTXR2 Antibodies) and IF revealed SN1 and SN2 (show Slc38a5 Antibodies) expression in the ganglion, inner nuclear, and photoreceptor cell layers.
Potassium restriction, high protein intake, and metabolic acidosis increase expression of the glutamine transporter (show SLC38A1 Antibodies) SNAT3 (Slc38a3) in mouse kidney.
Sodium-dependent amino acid/proton antiporter. Mediates electrogenic cotransport of glutamine and sodium ions in exchange for protons. Also recognizes histidine, asparagine and alanine. May mediate amino acid transport in either direction under physiological conditions. May play a role in nitrogen metabolism and synaptic transmission.
solute carrier family 38, member 3
, sodium-coupled neutral amino acid transporter 3
, sodium-coupled neutral amino acid transporter 3-like
, N-system amino acid transporter 1
, Na(+)-coupled neutral amino acid transporter 3
, system N amino acid transporter 1
, system N1 Na+ and H+-coupled glutamine transporter
, N system amino acids transporter NAT-1
, system N1 amino acid transporter