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UROD encodes an enzyme in the heme biosynthetic pathway. Additionally we are shipping Uroporphyrinogen Decarboxylase Antibodies (76) and Uroporphyrinogen Decarboxylase Kits (8) and many more products for this protein.
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a new mutation in the UROD gene in Egyptian patients with Hepatoerythropoietic porphyria, is reported.
a new insight in the conformational changes occurred in the mutant structures of UROD protein.
Data suggest that the traditional Chinese medicine (TCM) candidate potential three-in-one inhibitors for three drug target proteins epidermal growth factor receptor (EGFR), Her2, and uroporphyrinogen decarboxylase (UROD) against head and neck cancer.
Expression of the D306Y mutation results in an insoluble recombinant protein. G318R and K297N have little effect on the structure or activity of recombinant URO-D, but the proteins display reduced stability in vitro.
Among seventeen F-PCT patients, sixteen UROD mutations were identified.
analysis of uroporphyrinogen decarboxylase as a potential target for specific components of traditional Chinese medicine
Mutations in UROD gene is associated with familial porphyria cutanea tarda.
Hepatoerythropoietic porphyria and familial porphyria cutanea tarda G281E mutation in the uroporphyrinogen decarboxylase gene
function of Arg37 in uroporphyrinogen III decarboxylase
description of 3 siblings with hepatoerythropoietic porphyria; sequencing of the UROD gene revealed compound heterozygosity for a novel missense mutation, V166A, and a complex deletion/insertion, 645del1053ins10
Sequence deletions in uroporphyrinogen decarboxylase is associated with Porphyria Cutanea Tarda.
we identified eight mutations in 18 previously unclassified porphyria cutanea tarda families
Sequencing of the hydroxymethylbilane synthase and uroporphyrinogen decarboxylase genes confirmed the relatively rare diagnosis of dual porphyria, and revealed a novel uroporphyrinogen decarboxylase mutation
There is a high degree of molecular heterogeneity of familial porphyria cutanea tarda in Spain and molecular genetic analysis is useful in distinguish between F-PCT and sporadic PCT.
role of UROD mutations as a strong risk factor for porphyria cutanea tarda even in areas where environmental factors (hepatitis C virus) have been shown to be highly associated with the disease
This gene encodes an enzyme in the heme biosynthetic pathway. This enzyme is responsible for catalyzing the conversion of uroporphyrinogen to coproporphyrinogen through the removal of four carboxymethyl side chains. Mutations and deficiency in this enzyme are known to cause familial porphyria cutanea tarda and hepatoerythropoetic porphyria.
, protein UroD
, Uroporphyrinogen decarboxylase
, uroporphyrinogen III decarboxylase
, porphyrinogen carboxylase