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anti-Human Vasopressin Antibodies:
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Chicken Polyclonal Vasopressin Primary Antibody for IEM, ICC - ABIN617884
Fleming, Dallemagne, Endre, Yesberg, Cross: Acute lowering of plasma oncotic pressure increases filtration fraction and sodium excretion in conscious sheep. in Renal physiology and biochemistry 1993
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Human Monoclonal Vasopressin Primary Antibody for IF, IHC (p) - ABIN109870
Blume, Torner, Liu, Subburaju, Aguilera, Neumann: Prolactin activates MAP kinase signaling and corticotropin releasing hormone (CRH) transcription in rat hypothalamic neurons. in Endocrinology 2008
Show all 2 Pubmed References
Human Polyclonal Vasopressin Primary Antibody for ICC, IHC - ABIN286067
Aujla, Bora, Monahan, Sweedler, Raetzman: The Notch effector gene Hes1 regulates migration of hypothalamic neurons, neuropeptide content and axon targeting to the pituitary. in Developmental biology 2011
Broad Bean (Vicia faba) Polyclonal Vasopressin Primary Antibody for IHC (fro), IF - ABIN2477492
Radotra, Apostolopoulos, Sandison, Hatfield, Mendoza, Moss, Mehta, Glaser, Meeran, Roncaroli: Primary sellar neuroblastoma presenting with syndrome of inappropriate secretion of anti-diuretic hormone. in Endocrine pathology 2010
Hormone Polyclonal Vasopressin Primary Antibody for IHC (fro) - ABIN2477493
Suzuki, Handa: Estrogen receptor-beta, but not estrogen receptor-alpha, is expressed in prolactin neurons of the female rat paraventricular and supraoptic nuclei: comparison with other neuropeptides. in The Journal of comparative neurology 2005
Cerebrospinal fluid levels of arginine vasopressin significantly differentiates children with autism from controls and is associated with great social symptom severity in children with autism.
The results show that high plasma copeptin levels are associated with the decline of renal function in patients with type 2 diabetes mellitus and, therefore, copeptin may be considered as a biomarker of renal function.
Copeptin levels do not correlate with markers of insulin resistance in type 1 diabetes, but strongly correlates in non-type 1 diabetes.
Plasma VP levels did not differ between schizophrenia patients and healthy controls.
The relationship between copeptin and IGFBP-1 observed in the acute phase of an MI is sustained over time in type 2 diabetics.
Plasma copeptin was positively associated with major cardiovascular events in people with type 2 diabetes. This association cannot be solely accounted for by the association of copeptin with kidney-related traits.
We identified a synonymous variant affecting the second nucleotide of exon 3 in the AVP gene (c.324G>A) in a family in which autosomal dominant familial neurohypophyseal diabetes insipidus
Study found that the novel Ser18del variant in the AVP gene is associated with inherited autosomal dominant familial neurohypophyseal diabetes insipidus that seems to be partial and that mainly females report symptoms thereof.
Serum copeptin concentrations did not vary between the pregnancies complicated by intrahepatic cholestasis of pregnancy and the healthy pregnancy control group.
Copeptin appears to be an independent predictor of long-term mortality in a selected population of patients suspected for an acute coronary syndrome (ACS). In addition, copeptin may be considered as an early marker for the identification of patients at higher risk of the development of heart failure at long term in patients with acute chest pain.
Increased levels of NT-proBNP seems to be a better biomarker of multidrug antihypertensive therapy requirement than CT-proAVP.
High AVP expression is associated with gestational diabetes insipidus.
Plasma concentration of copetin demonstrated associations with stroke occurrence in a West African cohort but was not associated with stroke severity or mortality.
Data suggest that corticotrophin-releasing hormone (CRH) is able to stimulate copeptin release in healthy controls suggesting direct interaction of CRH/CRH-receptor signaling and vasopressin; these interactions appear to be altered in patients with pituitary disease; copeptin may be serum biomarker of altered CRH/CRH-receptor signaling in pituitary diseases.
copeptin might have a potential role in the pathogenesis of restless legs syndrome and be a biomarker for this disease.
Serum copeptin is a strong prognostic marker in both chronic obstructive pulmonary disease and acute heart failure.
Hypothalamic suprachiasmatic nucleus -AVP-ir showed a significant negative correlation with age in the control group and in the male, but not in the female depression group.
elevated plasma copeptin level is associated with an increased risk of heart failure (HF) and all-cause mortality in patients with HF.
Resistin, but not copeptin levels are higher in acute ischemic stroke patients early after the stroke onset, than in age and gender matched stroke-free controls. Moreover, higher copeptin concentrations are predictive of poor short term functional outcome after ischemic stroke
Cord blood copeptin concentration does not seem to be a promising marker of acute kidney injury in asphyxiated neonates.
The abnormal social behaviors of Dio3-/- mice were associated with sexually dimorphic alterations in the physiology of oxytocin (OXT) and arginine vasopressin (AVP), 2 neuropeptides with important roles in determining social interactions.
Data suggested that arginine vasopressin in the brain, not spinal cord and peripheral system of spinocerebellar ataxia type 3 mice related with the change of the spatial learning and memory of Morris water maze test.
The suprachiasmatic nucleus (SCN)network consists of multiple clusters of cellular circadian rhythms that are differentially integrated by AVP and VIP signaling, depending on the postnatal period
These results identify G9a-induced histone methylation at the OXT and AVP promoters in the Basolateral Amygdala as a mechanism for mediating stress-induced lasting behavioral depression and its reversal by exercise.
PDZ domain-containing protein Sipa1l1 (signal-induced proliferation-associated 1 like 1) binds to the cytoplasmic PDZ-ligand motif of aquaporin-2 and accelerates its endocytosis in the absence of vasopressin
Spatiotemporal profiles of arginine vasopressin (AVP) expression at the pixel level revealed circadian rhythms in the entire area of AVP-positive cells in the suprachiasmatic nucleus, and at least two clusters that showed different circadian oscillations.
Results suggest morphological and functional differences among the paraventricular nucleus' subnuclei and complex interactions among Chronic Unpredictable Stress, gonadal hormones and AVP immunoreactivity
Autophagy was induced in the AVP neurons subjected to endoplasmic reticulum stress in familial neurohypophysial diabetes insipidus mice.
A new role of PcG proteins in priming early-life stress-responsive DNA methylation at the Avp enhancer prior to epigenetic programming.
The noradrenaline-nitric oxide pathway, which stimulates the expression of vasopressin, involves free radicals.
AVP, via activation of V1 receptors, is able to modulate positively contractile activity of longitudinal muscle of mouse distal colon.
Dehydration increased plasma osmolality and vasopressin levels, and suppressed the increase in blood flow induced by neural activity, by the endothelium-dependent vasodilator acetylcholine and the smooth muscle relaxant adenosine.
Vasopressin expression in the suprachiasmatic nucleus is independent of Crx (cone-rod homeobox)-regulated photoreceptor systems.
This suggests that increased IL-1beta and NO levels may cause significant bioenergetics changes in magnocellular neurons, affecting copeptin expression and compromising AVP synthesis and secretion in the late phase of sepsis.
Folate deficiency decreases the circadian amplitude of vasopressin and the clock protein PER2.
medial amygdala likely modulates hostile aggressive behavior associated with immediate early gene expression in oxytocin and vasopressin neurons
results support the idea that variation in ovarian hormones could be related to individual differences in social recognition, at least partly through modulation of the OT and/or AVP systems, particularly in the DS, LA and MPOA.
The data suggested that vasopressin or oxytocin exert a minimal effect on most GABA neurons in the lateral hypothalamus but exert a robust excitatory effect on presumptive GABA cells that contain melanin-concentrating hormone.
Results indicate that Inpp5k 5-phosphatase is important for the control of the arginine vasopressin/aquaporin-2 signalling pathway and water transport in kidney collecting ducts.
Hypothalamic COX-2 activity was not necessary for enhanced arginine vasopressin expression and secretion in response to water deprivation.
Atrial natriuretic peptide and vasopressin-presence in the ciliary body of eye in the pig (sus domesticus).
This gene encodes a precursor protein consisting of arginine vasopressin and two associated proteins, neurophysin 2 and a glycopeptide, copeptin. Arginine vasopressin is a posterior pituitary hormone which is synthesized in the supraoptic nucleus and paraventricular nucleus of the hypothalamus. Along with its carrier protein, neurophysin 2, it is packaged into neurosecretory vesicles and transported axonally to the nerve endings in the neurohypophysis where it is either stored or secreted into the bloodstream. The precursor is thought to be activated while it is being transported along the axon to the posterior pituitary. Arginine vasopressin acts as a growth factor by enhancing pH regulation through acid-base transport systems. It has a direct antidiuretic action on the kidney, and also causes vasoconstriction of the peripheral vessels. This hormone can contract smooth muscle during parturition and lactation. It is also involved in cognition, tolerance, adaptation and complex sexual and maternal behaviour, as well as in the regulation of water excretion and cardiovascular functions. Mutations in this gene cause autosomal dominant neurohypophyseal diabetes insipidus (ADNDI).
, prepro-AVP-NP II
, prepro-arginine-vasopressin-neurophysin II
, vasopressin-neurophysin 2-copeptin
, vasopressin-neurophysin II-copeptin
, arginine vasopressin (Diabetes insipidus), same as Di (conflicting physical mapping)
, vasopressin-neurophysin prepropeptide
, arginine vasopressin-neurophysin II
, arginine vasopressin (neurophysin II, antidiuretic hormone, diabetes insipidus, neurohypophyseal)
, arginine vasopressin
, neurophysin 2
, arginine vasopressin (neurophysin 2, antidiuretic hormone, diabetes insipidus, neurohypophyseal)
, neurophysin II