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Human Polyclonal CRP Primary Antibody for ICC, IF - ABIN258345
Tabuchi, Inoue, Usui-Kataoka, Kobayashi, Teramoto, Takasugi, Shikata, Yamamura, Ando, Nishida, Kasahara, Kume, Lopez, Mitsudo, Nobuyoshi, Yasuda, Kita, Makino, Matsuura: The association of C-reactive protein with an oxidative metabolite of LDL and its implication in atherosclerosis. in Journal of lipid research 2007
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Human Monoclonal CRP Primary Antibody for ELISA (Capture), IF - ABIN191157
Cook, Boy, Flowers, Daroca: The influence of high-altitude living on body iron. in Blood 2005
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Human Monoclonal CRP Primary Antibody for IF, ELISA - ABIN191156
Silaste, Rantala, Alfthan, Aro, Witztum, Kesäniemi, Hörkkö: Changes in dietary fat intake alter plasma levels of oxidized low-density lipoprotein and lipoprotein(a). in Arteriosclerosis, thrombosis, and vascular biology 2004
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Human Monoclonal CRP Primary Antibody for IF, ELISA - ABIN191159
Tarkkinen, Palenius, Lövgren: Ultrarapid, ultrasensitive one-step kinetic immunoassay for C-reactive protein (CRP) in whole blood samples: measurement of the entire CRP concentration range with a single sample dilution. in Clinical chemistry 2002
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Elevated CRP level is correlated with poor prognosis in patients with metastatic renal cell carcinoma (show MOK Antibodies) receiving tyrosine kinase (show TXK Antibodies) inhibitors treatment. (Meta-analysis)
The study reveals that CRP might be a clear cell renal cell carcinoma (show MOK Antibodies) development promoter.
CRP distinguished sepsis and non-septic ICU patients.
Suggest that hs-CRP may be a marker of comorbidities associated with worse health status in myocardial infarction.
CRP, an upstream inflammatory marker, was independently associated with the risk of major adverse cardiovascular events, cardiovascular and all-cause mortality, myocardial infarction, heart failure, and cancer mortality in patients with stable coronary heart diseas
Cumulative exposure to hs-CRP was dose dependently associated with a subsequent increased risk of cardiovascular disease and myocardial infarction.
Preoperative CRP level is an independent predicting factor of over survival and cancer-specific survival in patients with metastatic renal cell carcinoma (show MOK Antibodies) who receive cytoreductive nephrectomy.
Interval change in CRP is a comparable measure to absolute CRP in the prognostication of acute pancreatitis severity.
Serum CRP levels were correlated with abdominal aorta diameter and rs1205 was related to serum CRP concentrations in a Chinese population.
Higher serum CRP levels were associated with higher baseline disability, suggesting systemic inflammation may contribute to disability independently of clinical vascular events.
Enhanced external counterpulsation decreased C-reactive protein and increased flow-mediated dilation in hypercholesterolemia.
serum C-reactive protein correlates with macrophage accumulation and coronary artery disease in hypercholesterolemic pigs
CRP inhibits endothelium-dependent NO-mediated dilation in coronary arterioles by producing superoxide from NAD(P)H (show NQO1 Antibodies) oxidase via p38 (show MAPK14 Antibodies) kinase activation.
The present study shows that elevated levels of serum CRP and IL-10 (show IL10 Antibodies) were associated with porcine circovirus type 2-infected piglets that subsequently developed severe PMWS. (postweaning multisystemic wasting syndrome)
These data suggest that CRP might play a pathogenic role in the development of myointimal hyperplasia in polytetrafluoroethylene grafts.
myocardial infarction induced proinflammatory gene and protein expression in peripheral blood mononuclear cells of tissue factor (show F3 Antibodies) cyclo-oxygenase-2, monocyte chemoattractant protein-1 (show CCL2 Antibodies) and CRP
CRP stimulates superoxide production and the subsequent formation of peroxynitrite from basal released nitric oxide compromises PGI(2 (show PTGIR Antibodies)) synthesis.
Data show that during posttranplant obliterative bronchiolitis development, CRP is localized in inflammatory cells, myofibroblasts and endothelial cells probably as a part of the local inflammatory response.
CRP is pathogenic in type-2 diabetes (T2DN). CRP may promote CD32b- NF-kappaB (show NFKB1 Antibodies) signaling to mediate renal inflammation; whereas, CRP may enhance renal fibrosis in T2DN via CD32b-Smad3 (show SMAD3 Antibodies)-mTOR (show FRAP1 Antibodies) signaling.
FcgammaRIIb and FcgammaRIII are involved in the CRP-induced expression of MMPs and TIMP-1 and the CRP-induced phosphorylation of p38, whereas the FcgammaR-independent pathway may regulate the CRP-induced MMP-11 expression and the CRP-induced ERK1/2 phosphorylation.
C-reactive protein may promote acute kidney injury by impairing tubular epithelial cell regeneration via the CD32-Smad3 (show SMAD3 Antibodies)-p27 (show CDKN1B Antibodies)-driven inhibition of the CDK2 (show CDK2 Antibodies)/cyclin E (show CCNE1 Antibodies) complex.
These results suggested that CRP/oxidized low density lipoprotein/beta 2-Glycoprotein I (show APOH Antibodies) complexes may induce lipid accumulation and inflammation in macrophages via the p38/MAPK (show MAPK14 Antibodies) and NFkappaB signaling pathways.
these data unexpectedly identify CBS (show CBS Antibodies) as the major recognition site of mCRP and suggest that this motif may be exploited to tune the proinflammatory actions of mCRP.
CRP decreased adiponectin expression and multimerization, while CRP-induced decline in adiponectin might be mediated through the PI3K/Akt (show AKT1 Antibodies) pathway.
Effects of C-Reactive Protein on the Cardiac Differentiation of Mouse Embryonic Stem Cells
These data indicated that CRP may have a direct role on osteoclast and osteoblast differentiation via toll (show TLR4 Antibodies)-like receptor signaling pathways.
Data suggest that serum amyloid P (SAP (show APCS Antibodies)) activates CD209 DC-SIGN (show CD209 Antibodies) to regulate the innate immune system differently from C-reactive protein (CRP), and that DC-SIGN (show CD209 Antibodies) is a target for antifibrotics.
the acute phase protein (show ORM1 Antibodies) C (show PROC Antibodies)-reactive protein (CRP), a ligand for Fc receptors on phagocytes, enhances antibody-mediated platelet destruction by phagocytes in vivo
data show, for the first time, a significant increase of c-reactive protein (CRP) in the blood of calves 1 d after colostrum intake.
Rabbit CRP acute-phase system binds phosphocholine and phosphoethanolamine.
IL-6 (show IL6 Antibodies) augments mechanical strain-induced CRP synthesis in vessels via the stretch-activated channel-NF-kappaB (show NFKB1 Antibodies) pathway.
CRP antagonism of eNOS (show NOS3 Antibodies) is mediated by coupling of FcgammaRI (show FCGR1A Antibodies) to FcgammaRIIB by Src kinase (show CSK Antibodies) and activation of inositol 5'-phosphatase 1, and consistent with this mechanism, both FcgammaRI (show FCGR1A Antibodies) and FcgammaRIIB are required for CRP to blunt endothelial repair in vivo.
Metformin attenuated the progression of aortic atherosclerosis via down-regulation of serum C-reactive protein.
crystal of C-reactive protein diffracted to 2.3 A resolution and belonged to space group R3, with unit-cell parameters a = b = 114.7, c = 61.0 A
The protein encoded by this gene belongs to the pentaxin family. It is involved in several host defense related functions based on its ability to recognize foreign pathogens and damaged cells of the host and to initiate their elimination by interacting with humoral and cellular effector systems in the blood. Consequently, the level of this protein in plasma increases greatly during acute phase response to tissue injury, infection, or other inflammatory stimuli.
, pentraxin 1
, C-reactive protein, petaxin related
, C-reactive protein member of the pentraxin family
, C-reactive protein, member of the pentraxin family
, amyloid P component, serum
, serum amyloid P component
, pentraxin-related C-reactive protein
, uncharacterized protein LOC751795