Browse our anti-C-Reactive Protein (CRP) Antibodies

Human C-Reactive Protein (CRP) interaction partners

1. A significant positive association of Fibromyalgia to serum C-reactive protein was linked in part to body mass index.

2. Normal C-reactive protein (CRP) values were observed in up to 28% of children with Crohn disease and 42% of children with ulcerative colitis at diagnosis. Correlation of CRP with Crohn disease activity during follow-up depended on their value at diagnosis and mode of analysis. CRP was not useful in predicting ulcerative colitis disease activity.

3. In patients with food protein-induced enterocolitis syndrome (FPIES) caused by cow's milk, serum CRP increased in 50% of subjects at onset. Age at tolerance acquisition was significantly positively correlated with serum CRP at onset. Serum CRP is a useful parameter of poor prognosis in FPIES.

4. Higher longitudinal serum high-sensitivity C-reactive protein levels elevated over time, were predictive of worse prognosis among continuous ambulatory peritoneal dialysis patients.

5. The association between CRP level, symptoms, and ADL (show SGCA Antibodies) observed in advanced cancer patients receiving palliative care indicates that CRP level might be a good biomarker.

6. these results suggest that that CRP may be a potent inducer of the anti-inflammatory cytokine IL-10 (show IL10 Antibodies)

7. C-reactive protein (CRP) and N/L ratio did not predict poor clinical outcome in STS (show STS Antibodies) patients receiving neoadjuvant radiotherapy (RT).

8. The research showed that the enhancement degree on contrast-enhanced ultrasound was closely correlated to C-reactive protein levels, the neovessel density and the macrophage number.

9. preoperative serum C-reactive protein level is an independent and significant indicator for elevated morbidity and mortality after pulmonary resection

10. Multivariate analysis identified thrombocytosis and serum C-reactive protein (CRP) level as independent prognostic factors in pancreatic cancer.

Mouse (Murine) C-Reactive Protein (CRP) interaction partners

1. CRP is pathogenic in type-2 diabetes (T2DN). CRP may promote CD32b- NF-kappaB (show NFKB1 Antibodies) signaling to mediate renal inflammation; whereas, CRP may enhance renal fibrosis in T2DN via CD32b-Smad3 (show SMAD3 Antibodies)-mTOR (show FRAP1 Antibodies) signaling.

2. FcgammaRIIb and FcgammaRIII are involved in the CRP-induced expression of MMPs and TIMP-1 and the CRP-induced phosphorylation of p38, whereas the FcgammaR-independent pathway may regulate the CRP-induced MMP-11 expression and the CRP-induced ERK1/2 phosphorylation.

3. C-reactive protein may promote acute kidney injury by impairing tubular epithelial cell regeneration via the CD32-Smad3 (show SMAD3 Antibodies)-p27 (show CDKN1B Antibodies)-driven inhibition of the CDK2 (show CDK2 Antibodies)/cyclin E (show CCNE1 Antibodies) complex.

4. These results suggested that CRP/oxidized low density lipoprotein/beta 2-Glycoprotein I (show APOH Antibodies) complexes may induce lipid accumulation and inflammation in macrophages via the p38/MAPK (show MAPK14 Antibodies) and NFkappaB signaling pathways.

5. these data unexpectedly identify CBS (show CBS Antibodies) as the major recognition site of mCRP and suggest that this motif may be exploited to tune the proinflammatory actions of mCRP.

6. CRP decreased adiponectin expression and multimerization, while CRP-induced decline in adiponectin might be mediated through the PI3K/Akt (show AKT1 Antibodies) pathway.

7. Effects of C-Reactive Protein on the Cardiac Differentiation of Mouse Embryonic Stem Cells

8. These data indicated that CRP may have a direct role on osteoclast and osteoblast differentiation via toll (show TLR4 Antibodies)-like receptor signaling pathways.

9. Data suggest that serum amyloid P (SAP (show APCS Antibodies)) activates CD209 DC-SIGN (show CD209 Antibodies) to regulate the innate immune system differently from C-reactive protein (CRP), and that DC-SIGN (show CD209 Antibodies) is a target for antifibrotics.

10. the acute phase protein (show ORM1 Antibodies) C (show PROC Antibodies)-reactive protein (CRP), a ligand for Fc receptors on phagocytes, enhances antibody-mediated platelet destruction by phagocytes in vivo

Cow (Bovine) C-Reactive Protein (CRP) interaction partners

1. data show, for the first time, a significant increase of c-reactive protein (CRP) in the blood of calves 1 d after colostrum intake.

Pig (Porcine) C-Reactive Protein (CRP) interaction partners

1. Enhanced external counterpulsation decreased C-reactive protein and increased flow-mediated dilation in hypercholesterolemia.

2. serum C-reactive protein correlates with macrophage accumulation and coronary artery disease in hypercholesterolemic pigs

3. CRP inhibits endothelium-dependent NO-mediated dilation in coronary arterioles by producing superoxide from NAD(P)H (show NQO1 Antibodies) oxidase via p38 (show MAPK14 Antibodies) kinase activation.

4. The present study shows that elevated levels of serum CRP and IL-10 (show IL10 Antibodies) were associated with porcine circovirus type 2-infected piglets that subsequently developed severe PMWS. (postweaning multisystemic wasting syndrome)

5. These data suggest that CRP might play a pathogenic role in the development of myointimal hyperplasia in polytetrafluoroethylene grafts.

6. myocardial infarction induced proinflammatory gene and protein expression in peripheral blood mononuclear cells of tissue factor (show F3 Antibodies) cyclo-oxygenase-2, monocyte chemoattractant protein-1 (show CCL2 Antibodies) and CRP

7. CRP stimulates superoxide production and the subsequent formation of peroxynitrite from basal released nitric oxide compromises PGI(2 (show PTGIR Antibodies)) synthesis.

8. Data show that during posttranplant obliterative bronchiolitis development, CRP is localized in inflammatory cells, myofibroblasts and endothelial cells probably as a part of the local inflammatory response.

Rabbit C-Reactive Protein (CRP) interaction partners

1. Rabbit CRP acute-phase system binds phosphocholine and phosphoethanolamine.

2. IL-6 (show IL6 Antibodies) augments mechanical strain-induced CRP synthesis in vessels via the stretch-activated channel-NF-kappaB (show NFKB1 Antibodies) pathway.

3. CRP antagonism of eNOS (show NOS3 Antibodies) is mediated by coupling of FcgammaRI (show FCGR1A Antibodies) to FcgammaRIIB by Src kinase (show CSK Antibodies) and activation of inositol 5'-phosphatase 1, and consistent with this mechanism, both FcgammaRI (show FCGR1A Antibodies) and FcgammaRIIB are required for CRP to blunt endothelial repair in vivo.

4. Metformin attenuated the progression of aortic atherosclerosis via down-regulation of serum C-reactive protein.