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Protein kinase A-alpha directly phosphorylates FoxO1 (show FOXO1 Proteins) in vascular endothelial cells to regulate expression of vascular cellular adhesion molecule (show NCAM1 Proteins)-1 mRNA.
the p38-MAPK (show MAPK14 Proteins)/CREB (show CREB1 Proteins) pathway plays a critical role in TNFalpha (show TNF Proteins)-induced VCAM-1 expression
Association with the efficiency of hematopoietic stem cells (HSC (show FUT1 Proteins)) mobilization to PB were found in the SNPs of the DGKB (show DGKB Proteins) gene involved in cell transport and SDF-1 (show CXCL12 Proteins)-induced migration ability and of the VCAM1 gene which is essential for HSC (show FUT1 Proteins) homing, suggesting that SNPs involved in cell migration ability might be partly involved in HSC (show FUT1 Proteins)
Augmented expression of endothelial adhesion molecules ICAM1 (show ICAM1 Proteins)/VCAM1 is involved in the pathophysiology of patients with antiphospholipid syndrome.
VCAM-1 knockdown reduced TNFalpha (show TNF Proteins)-induced HUVEC migration and tube formation.
High levels of serum ICAM-1 (show ICAM1 Proteins) was associated with the development of multiple organ failure . High levels of VCAM-1 was associated with both multiple organ failure and in-hospital mortality.
VCAM1 protein expression increased in cartilage tissue from spinal tuberculosis patients.
This study provided direct evidence that increasing extracellular Ca(2 (show CA2 Proteins)+) enhanced TNF-alpha (show TNF Proteins)-induced VCAM-1 activation and monocytes adhesion. Moreover, we identified a novel TRPC1 (show TRPC1 Proteins)/ERK1/2/NFkappaB (show NFKB1 Proteins) signaling pathway mediating VCAM-1 activation and monocyte adhesion in this pathological process.
patients with newly-diagnosed MM have increased serum levels of VCAM-1 and ICAM-1 (show ICAM1 Proteins) that correlate with advanced disease features. VCAM-1 is also independently correlated with survival, suggesting an important role in the biology of the disease and supporting its targeting with novel anti-myeloma drugs.
VCAM-1 was statistically elevated in major depressive disorder.
Data show that podoplanin (PDPN (show PDPN Proteins)), CD106 (VCAM-1) and CD248 (show CD248 Proteins) protein were increased in diseased compared to healthy tendon cells.
Peptide nanoparticles targeting VCAM-1 are suggested as a theranostic carrier for drug delivery to inflamed endothelium.
VCAM-1 promotes CD34 (show CD34 Proteins)(+)VLA-4(+) cell mobilization into the damaged myocardial tissues.
These studies define differential roles for VCAM-1 in SC depending on the state of their niche.
VCAM1 affects the neural stem cells fate by signaling through its intracellular domain to regulate beta-catenin (show CTNNB1 Proteins) signaling in a context-dependent manner
Reducing Cadm1 (show CADM1 Proteins) expression in the hypothalamus and hippocampus promoted a negative energy balance and weight loss
Here we delineated an inflammation-driven inhibitory mechanism of beige adipogenesis in obesity that required direct adhesive interactions between macrophages and adipocytes mediated by the integrin alpha4 and its counter-receptor VCAM-1, respectively; expression of the latter was upregulated in obesity.
High VCAM1 expression is associated with Experimental Autoimmune Myocarditis
Aortic VCAM-1 was expressed early and indicated vascular inflammation in collagen-induced arthritis.
this manuscript shows that endothelial cell FAK (show PTK2 Proteins) activity induces VCAM-1 expression and contributes to atherosclerosis
Gas6 (show GAS6 Proteins)-FoxO-1 (show FOXO1 Proteins) signaling axis plays an important role in VCAM-1 expression in the context of venous thromboembolism by promoting bone marrow mononuclear cell-endothelial cell adhesion.
These results suggest that Tie2 (show TEK Proteins) signaling induces alpha4beta1 integrin activation on bone marrow-mast cell progenitor for adhesion to VCAM-1.
Data show that all five molecules, BNP, ICAM-1 (show ICAM1 Proteins), TNF-alpha (show TNF Proteins), VCAM-1 and IL-6 (show IL6 Proteins), quickly and reliably signaled adverse interactions.
Altered shear stress stimulates upregulation of endothelial VCAM-1 and ICAM-1 (show ICAM1 Proteins) in a BMP-4 (show BMP4 Proteins)- and TGF-beta1 (show TGFB1 Proteins)-dependent pathway.
This gene is a member of the Ig superfamily and encodes a cell surface sialoglycoprotein expressed by cytokine-activated endothelium. This type I membrane protein mediates leukocyte-endothelial cell adhesion and signal transduction, and may play a role in the development of artherosclerosis and rheumatoid arthritis. Three alternatively spliced transcripts encoding different isoforms have been described for this gene.
vascular cell adhesion protein 1
, vascular cell adhesion molecule 1
, vascular cell adhesion protein 1-like
, CD106 antigen
, V-CAM 1
, vascular cell adhesion molecule-1