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anti-Human TGFB2 Antibodies:
anti-Mouse (Murine) TGFB2 Antibodies:
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Human Polyclonal TGFB2 Primary Antibody for IHC (p), WB - ABIN1882140
Hatsushika, Hirota, Harada, Sakashita, Kanzaki, Takano, Doi, Fujita, Enomoto, Ebisawa, Yoshihara, Sagara, Fukuda, Masuyama, Katoh, Matsumoto, Saito, Ogawa, Tamari, Nakao: Transforming growth factor-beta(2) polymorphisms are associated with childhood atopic asthma. in Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology 2007
Show all 4 Pubmed References
Human Polyclonal TGFB2 Primary Antibody for WB - ABIN4359074
Lin, Teo, Lam, Jeyaseelan, Wang: MicroRNA-10b pleiotropically regulates invasion, angiogenicity and apoptosis of tumor cells resembling mesenchymal subtype of glioblastoma multiforme. in Cell death & disease 2012
Furthermore, upregulation of miR (show MLXIP Antibodies)-328 could further repress the expression of TGF-beta2 and ECM (show MMRN1 Antibodies) proteins. In conclusion, this study demonstrated that miR (show MLXIP Antibodies)-328 could prevent renal fibrogenesis by directly targeting TGF-beta2. Our findings suggested that elevated renal miR (show MLXIP Antibodies)-328 levels might be a novel therapeutic strategy for treating renal fibrosis
Importantly, high expression levels of HIF-1alpha (show HIF1A Antibodies)/TGF-beta2/GLI2 (show GLI2 Antibodies) correlated robustly with the patient relapse following chemotherapy, highlighting a potential biomarker and therapeutic target for chemoresistance in colorectal cancer.
these data suggest that miR (show MLXIP Antibodies)-592 may exert it suppressive role in breast cancer, at least in part, by targeting TGFbeta (show TGFB1 Antibodies)-2, and that miR (show MLXIP Antibodies)-592 may be a novel target for breast cancer treatment
MicroRNA-486-5p suppresses TGFB2-induced proliferation, invasion and epithelial-mesenchymal transition of lens epithelial cells by targeting Smad2 (show SMAD2 Antibodies).
Results show that TGF-beta2 is highly expressed in glioma and correlated with poor prognosis in glioma patients. Further findings elucidate a potential mechanism of autophagy-associated glioma invasion that TGF-beta2 could initiate autophagy via Smad (show SMAD1 Antibodies) and non-Smad (show SMAD1 Antibodies) pathway to promote glioma cells' invasion.
Up-regulation of TGF-beta2 showed a strong association with muscle invasion in bladder cancer.
Breast milk immunomodulators TGFbeta1 (show TGFB1 Antibodies) and TGFbeta2 were significantly associated with neonatal gut (show GUSB Antibodies) microbial composition (R = 0.024, P = 0.041; R = 0.026, P = 0.012, respectively) and increased richness, evenness, and diversity, but IL-10 (show IL10 Antibodies) was not. The effects of TGFbeta1 (show TGFB1 Antibodies) and TGFbeta2, however, were not independent of one another, and the effect of TGFbeta2 was stronger than that of TGFbeta1 (show TGFB1 Antibodies).
Report early adaptive drug-escape in EGFR (show EGFR Antibodies)-mutant lung tumor cells dependent on TGFbeta2-bioenergetics-mitochondrial priming.
The expression of TGFB2 obtained by microarray analysis was consistent with that of RT-PCR. Ion transport could be affected promptly after ANP (show NPPA Antibodies) treatment, and subsequently, the cytolysis of vein endothelial cells may be promoted and endothelial permeability would be enhanced, followed by activated immune responses.
Data indicate that TGFb1 (show TGFB1 Antibodies) and TGFb3 (show TGFB3 Antibodies), but not TGFb2, showed higher expression levels in invasive breast cancer compared to normal tissues.
Suggest that the interplay between TGFbeta (show TGFB1 Antibodies)-2 and LPS (show IRF6 Antibodies) regulates the levels of IL-8 (show IL8 Antibodies) in the immature newborn intestine.
TGFbeta (show TGFB1 Antibodies) may play a role in the overall process of luteinization, but it appears not to influence steroidogenesis in luteinizing pig follicles.
data suggest a mechanism whereby a stromal hedgehog (show SHH Antibodies)-TGFbeta2 signaling axis acts to control nephrogenesis.
Data show that just like TGF-beta1 (show TGFB1 Antibodies), TGF-beta2 is expressed in and secreted by both, healthy and diseased hepatocytes and hepatic stellate cell (HSCs).
TGF-beta2 as the crucial mediator of neural precursor cell immunomodulation.
CREBH (show CREB3L3 Antibodies) was identified as a key positive regulator of TGF-beta2 transcription in hepatitis C virus-infected cells.
RUNX1T1 (show RUNX1T1 Antibodies) serves as a common angiogenic driver for vaculogenesis and functionality of endothelial lineage cells
The disruption of decorin (show DCN Antibodies)-restricted TGFbeta (show TGFB1 Antibodies) signalling leads to higher stiffness of articular cartilage matrix, rendering joints more resistant to osteoarthritis.
These data provide new insights in the molecular interaction between Fibulin-4 (show FBLN4 Antibodies) and TGF-beta (show TGFB1 Antibodies) pathway regulation in the pathogenesis of aortic aneurysms.
APC (show APC Antibodies)-derived TSP-1 (show GZMA Antibodies) is essential for the development of an adaptive regulatory immune response induced by TGF-beta2-expressing APCs (show APCS Antibodies) similar to those located at mucosal and ocular sites.
Epidermal Tgfb2 controls proliferation, differentiation and ECM (show MMRN1 Antibodies) production by reticular fibroblasts.
Pathological TGF-beta (show TGFB1 Antibodies) release from osteolytic bone metastases contributes to muscle weakness in cancer by decreasing Ca(2 (show CA2 Antibodies)+)-induced muscle force production.
The results of this study found that Bptf (show BPTF Antibodies) and TGF-beta (show TGFB1 Antibodies)/Smad2 (show SMAD2 Antibodies) mediate nucleosome remodeling to regulate wnt8a (show WNT8A Antibodies) expression and hence neural posteriorization.
Functional investigation of a subset of these genes, fgf10a (show FGF10 Antibodies), tgfb2, pax9 (show PAX9 Antibodies), and smad5 (show SMAD5 Antibodies) revealed their necessity in zebrafish palatogenesis.
These data suggest Pez (show PTPN14 Antibodies) plays a crucial role in organogenesis by inducing TGFbeta (show TGFB1 Antibodies) and epithelial-mesenchymal transition.
Data show that TGF-beta (show TGFB1 Antibodies) pathways operate during ovarian fetal development, and fibrillin 3 (show FBN3 Antibodies) is highly expressed at a critical stage early in developing human and bovine fetal ovaries.
There was no significant change in the expression of TGF-beta(2) and alpha-SMA (show SMN1 Antibodies) after laser-assisted intrastromal scanning.
This gene encodes a member of the transforming growth factor beta (TGFB) family of cytokines, which are multifunctional peptides that regulate proliferation, differentiation, adhesion, migration, and other functions in many cell types by transducing their signal through combinations of transmembrane type I and type II receptors (TGFBR1 and TGFBR2) and their downstream effectors, the SMAD proteins. Disruption of the TGFB/SMAD pathway has been implicated in a variety of human cancers. The encoded protein is secreted and has suppressive effects of interleukin-2 dependent T-cell growth. Translocation t(1\;7)(q41\;p21) between this gene and HDAC9 is associated with Peters' anomaly, a congenital defect of the anterior chamber of the eye. The knockout mice lacking this gene show perinatal mortality and a wide range of developmental, including cardiac, defects. Alternatively spliced transcript variants encoding different isoforms have been identified.
BSC-1 cell growth inhibitor
, glioblastoma-derived T-cell suppressor factor
, transforming growth factor beta-2
, TGF-beta 2
, Transforming growth factor beta-2
, transforming growth factor beta 2
, tgf beta 2
, transforming growth factor, beta 2
, milk growth factor
, transforming growth factor-beta 2
, transforming growth factor beta-2-like
, TGF beta 2 protein
, transforming growth factor-beta2
, TGF beta 2