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anti-Human CXCL2 Antibodies:
anti-Mouse (Murine) CXCL2 Antibodies:
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Mouse (Murine) Polyclonal CXCL2 Primary Antibody for Func, IHC (fro) - ABIN2475709
Hollifield, Page, Smith, Michelakis, Staab, Rhamy: Renin-secreting clear cell carcinoma of the kidney. in Archives of internal medicine 1975
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Mouse (Murine) Polyclonal CXCL2 Primary Antibody for IHC (fro), IHC (p) - ABIN2475710
Di Carlo, Comes, Basso, De Ambrosis, Meazza, Musiani, Moelling, Albini, Ferrini: The combined action of IL-15 and IL-12 gene transfer can induce tumor cell rejection without T and NK cell involvement. in Journal of immunology (Baltimore, Md. : 1950) 2000
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Rat (Rattus) Polyclonal CXCL2 Primary Antibody for IP, WB - ABIN110547
Frevert, Farone, Danaee, Paulauskis, Kobzik: Functional characterization of rat chemokine macrophage inflammatory protein-2. in Inflammation 1995
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Mouse (Murine) Polyclonal CXCL2 Primary Antibody for Neut, ELISA - ABIN223564
Asarias, Nguyen, Mings, Gehrich, Pierce: Influence of mesh materials on the expression of mediators involved in wound healing. in Journal of investigative surgery : the official journal of the Academy of Surgical Research 2011
Mouse (Murine) Polyclonal CXCL2 Primary Antibody for EIA, WB - ABIN116265
Sterzenbach, Lee, Brenneke, von Goetz, Schauer, Fox, Suerbaum, Josenhans: Inhibitory effect of enterohepatic Helicobacter hepaticus on innate immune responses of mouse intestinal epithelial cells. in Infection and immunity 2007
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Mouse (Murine) Polyclonal CXCL2 Primary Antibody for EIA, WB - ABIN116266
Gonzalez-Rey, Chorny, Robledo, Delgado: Cortistatin, a new antiinflammatory peptide with therapeutic effect on lethal endotoxemia. in The Journal of experimental medicine 2006
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Human Polyclonal CXCL2 Primary Antibody for ELISA, WB - ABIN4274954
De Filippo, Henderson, Laschinger, Hogg: Neutrophil chemokines KC and macrophage-inflammatory protein-2 are newly synthesized by tissue macrophages using distinct TLR signaling pathways. in Journal of immunology (Baltimore, Md. : 1950) 2008
Rat (Rattus) Polyclonal CXCL2 Primary Antibody for Func, ELISA - ABIN2473897
Vallès, Grijpink-Ongering, de Bree, Tuinstra, Ronken: Differential regulation of the CXCR2 chemokine network in rat brain trauma: implications for neuroimmune interactions and neuronal survival. in Neurobiology of disease 2006
Human Polyclonal CXCL2 Primary Antibody for Func, IHC (p) - ABIN2473896
Haskill, Peace, Morris, Sporn, Anisowicz, Lee, Smith, Martin, Ralph, Sager: Identification of three related human GRO genes encoding cytokine functions. in Proceedings of the National Academy of Sciences of the United States of America 1990
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Overexpressing CXCL2 by lentivirus also apparently inhibited the size and weight of subcutaneous tumours in nude mice and CXCL2 induced HCC cell apoptosis via both nuclear and mitochondrial apoptosis pathways.
We identified CXCL2, an immune-related chemokine, decreased in hepatocellular carcinoma and the regulation mechanism may be controlled by methylation
Results show that the expression of CXCL1 and CXCL2 in tumor cells and tumor-infiltrated CD11b+ myeloid cells is critically involved in the promotion of the generation of monocytic myeloid-derived suppressor cells (mo-MDSC) from bone marrow cells. CXCL1 and CXCL2 were found to specifically promote the expansion of mo-MDSC rather than granulocytic MDSC (G-MDSC).
These results show that AKIP1 is crucial in cervical cancer angiogenesis and growth by elevating the levels of the NF-kappaB-dependent chemokines CXCL1, CXCL2, and CXCL8.
The studies revealed that, although overall structural and oligomerization features of CXCL3 and CXCL2 are similar, prominent differences were observed in their surface characteristics, thus implicating a functional divergence.
Taken together, the data of the present study demonstrated that TcpC can induce MIP2 production, which may contribute to the characteristic histological change associated with pyelonephritis.
Functional effects data suggested that recombinant human CXCL2 significantly enhanced the migration, invasion ability of SMMC7721 hepatocellular carcinoma cells, and weakened adhesion ability.
Reduced rate of sickle-related complications in Brazilian patients carrying HbF-promoting alleles at the BCL11A and HMIP-2 loci
Results identify the CXCL2/MIF-CXCR2 axis as an important mediator in MDSC recruitment and as predictors in bladder cancer.
Chronic inflammation contributes to the change of CXCL12 DNA methylation in buccal cells and that DNA methylation profile of CXCL12 promoter plays important role in development and progression of periodontal disease.
Polymorphisms in the promoter regions of the CXCL1 and CXCL2 genes contribute to increased risk of alopecia areata in the Korean population
high GRO-beta expression correlates with poor prognosis and contributes to ovarian cancer tumorigenesis and metastasis.
In this review, a genetic variant in CXCL12 is described that is associated with type 2 diabetes mellitus and its complications.
We have demonstrated that GRObeta, as an oncogene product, contributed to tumorigenesis and metastasis of HCC
Our results demonstrated that resistance to anti-proliferative effects of CXCR2 may also arise from feedback increases in MIP-2 secretion.
autophagy is required for Hepatitis B virus-induced NF-kappaB activation and release of IL-6, IL-8, and CXCL2 in Hepatocytes
The results link CXCL1 and CXCL2 chemokines with bone marrow adiposity and implicate CXCR2 signaling in promoting effects of marrow fat on progression of skeletal tumors in bone.
CXCL2 has antimicrobial activity against E. coli and S. aureus.
Simultaneous targeting of hCAP-G2 and MIP-2A is a promising strategy for the development of antitumor drugs as a treatment for intractable tumours.
our results demonstrate the diverse mechanisms by which CXCL2 and CXCL3 mediate normal and asthmatic airway smooth muscle cell migration
Chemokines CXCL1 and CXCL2 and the Atypical Receptor ACKR1 are involved in neutrophil emigration.
CXCL1, CXCL2 and CCL2 binding to the glomerular endothelial glycocalyx appears differentially mediated by specific Heparan sulfate domains.
Tumor cell-expressed transient receptor potential channel, melastatin subfamily type 2 (TRPM2) regulates neutrophil chemoattraction via chemokine CXCL2 (CXCL2).
TIARP independently down-regulated CXCL2 and IL-6 production by fibroblast-like synoviocytes, and the expression of chemokine receptors (CXCR1 and CXCR2) in neutrophils, with resultant reduction of neutrophil migration into arthritic joints.
a paracrine role for Hippo-mediated secretion of CXCL1 and CXCL2 in the production of anti-microbial peptides (beta-defensins), iNOS, NOX2 and pro-inflammatory molecules during mycobacterial infection of the host, is reported.
mip-2 siRNA and the MIP-2 antibody can reduce the inflammatory effects induced by lipopolysaccharide in macrophage cells.
this study demonstrates that in vivo blocking of CXCL1 and CXCL2 can significantly reduce the Mycobacterium tuberculosis-induced bioactive IL-1beta production
These data identify suppression of CXCL2 and CXCL5 chemoattractant expression by 11beta-HSD1 as a novel mechanism with potential for regulation of neutrophil recruitment to the injured myocardium, and cardiac fibroblasts as a key site for intracellular glucocorticoid regeneration during acute inflammation following myocardial injury.
findings show that SRC-3 contributes to host defense against enteric bacteria, at least in part via upregulating CXCL2 expression to recruit neutrophils
p53-mediated induction of PAI-1 expression due to chronic CS exposure exacerbates lung inflammation through elaboration of CXCL1, CXCL2, and CXCR2.
Social defeat induced an exposure-dependent increase in mRNA levels of E-selectin, CXCL1, and CXCL2 that increased with additional days of social defeat.
MIP-2 directly impairs neonatal pulmonary endothelial cell migration in vitro.
These results have suggested that the activation of MyD88 pathway by constitutive low-level IL-1beta expression is essential for pressure force-induced CXCL2 and CCL2 expression in osteoblasts.
SIRT2 regulates LPS induced proximal renal tubules CXCL2 protein expression.
miR26a/-26b-COX-2-MIP-2 loop regulates allergic inflammation and the feedback relationship between allergic inflammation and the enhanced tumorigenic and metastatic potential
huH1N1 virus PA and NA mediated increased MIP-2 expression early postinfection, resulting in substantial pulmonary neutrophilia with enhanced lung pathology and disease
Syndecan-1 is expressed in the parietal peritoneum microvasculature but does not regulate leukocyte recruitment and is not necessary for the presentation of the chemokine MIP-2 in this tissue.
Identified TLR2 and S100A8/S100A9 as key regulators of hepatic CXCL-2 expression and neutrophil recruitment.
reports transmural and perivascular expression patterns of chemokines CCL2 and CXCL2 and of chemokine receptors CCR2, CCR5, and CXCR4 following coronary angioplasty
Results suggested that chemokine expression by cultured equine BECs following exposure to pulmonary hemorrhage conditions may contribute to the development of inflammatory airway disease in horses.
This study examined effects of in vitro exposure to solutions of hay dust, lipopolysaccharides, or beta-glucan on cytokine expression in pulmonary mononuclear cells isolated from healthy horses and horses with recurrent airway obstruction.
The acute pulmonary neutrophilia characteristic of recurrent airway obstruction was not associated with an increase in expression of chemokines in pulmonary mononuclear cells from disease-susceptible horses.
Produced by activated monocytes and neutrophils and expressed at sites of inflammation. Hematoregulatory chemokine, which, in vitro, suppresses hematopoietic progenitor cell proliferation. GRO-beta(5-73) shows a highly enhanced hematopoietic activity.
C-X-C motif chemokine 2
, GRO2 oncogene
, MGSA beta
, growth-regulated protein beta
, macrophage inflammatory protein 2-alpha
, melanoma growth stimulatory activity beta
, GRO3 oncogene
, GRO3, growth related gene 3
, growth-regulated protein homolog gamma
, macrophage inflammatory protein 2
, small inducible cytokine subfamily, member 2
, cytokine-induced neutrophil chemoattractant 3
, chemokine ligand 2