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anti-Human IL-10 Antibodies:
anti-Mouse (Murine) IL-10 Antibodies:
anti-Rat (Rattus) IL-10 Antibodies:
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Cow (Bovine) Polyclonal IL-10 Primary Antibody for IHC, ELISA - ABIN1582204
Shi, Diez-Freire, Jun, Qi, Katovich, Li, Sriramula, Francis, Sumners, Raizada: Brain microglial cytokines in neurogenic hypertension. in Hypertension 2010
Show all 16 Pubmed References
Human Monoclonal IL-10 Primary Antibody for - ABIN1383949
Schandené, Alonso-Vega, Willems, Gérard, Delvaux, Velu, Devos, de Boer, Goldman: B7/CD28-dependent IL-5 production by human resting T cells is inhibited by IL-10. in Journal of immunology (Baltimore, Md. : 1950) 1994
Show all 11 Pubmed References
Human Polyclonal IL-10 Primary Antibody for WB - ABIN3042618
Presti, DOrazio, Labra, La Ferla, Mezzasalma, Bizzaro, Giardina, Michelotti, Tursi, Vassallo, Di Gennaro: Evaluation of the probiotic properties of new Lactobacillus and Bifidobacterium strains and their in vitro effect. in Applied microbiology and biotechnology 2015
Show all 10 Pubmed References
Human Monoclonal IL-10 Primary Antibody for ELISA, IHC - ABIN4324436
Sahoo, Dey, Maiti: pSiM24 is a novel versatile gene expression vector for transient assays as well as stable expression of foreign genes in plants. in PLoS ONE 2014
Show all 6 Pubmed References
Human Monoclonal IL-10 Primary Antibody for ELISA (Capture), FACS - ABIN967464
Just, Abrams, Louie, Urbano, Wara, Nicholas, Stein, King: Influence of host genotype on progression to acquired immunodeficiency syndrome among children infected with human immunodeficiency virus type 1. in The Journal of pediatrics 1995
Show all 8 Pubmed References
Rat (Rattus) Polyclonal IL-10 Primary Antibody for IHC (p), ELISA - ABIN3042802
Gao, Zhao, Liu, Yuan, Wu, Xia: Myocardial ischemic post-conditioning protects the lung against myocardial ischemia/reperfusion-induced damage by activating GSK-3β. in Acta cirurgica brasileira 2017
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Mouse (Murine) Polyclonal IL-10 Primary Antibody for ELISA, WB - ABIN3042803
Jin, Yang, Song, Zhang, Li: Protective roles of quercetin in acute myocardial ischemia and reperfusion injury in rats. in Molecular biology reports 2012
Show all 5 Pubmed References
Human Monoclonal IL-10 Primary Antibody for ICC - ABIN2689715
Abrams, Roncarolo, Yssel, Andersson, Gleich, Silver: Strategies of anti-cytokine monoclonal antibody development: immunoassay of IL-10 and IL-5 in clinical samples. in Immunological reviews 1992
Show all 5 Pubmed References
Human Polyclonal IL-10 Primary Antibody for ELISA, IF (p) - ABIN672051
Kou, Hu, Yao, Wang, Shen, Kang, Hong: Transforming growth factor-?1 promotes Treg commitment in nasal polyposis after intranasal steroid treatment. in Inflammation research : official journal of the European Histamine Research Society ... [et al.] 2013
Show all 5 Pubmed References
Human Monoclonal IL-10 Primary Antibody for ICS, Neut - ABIN1177281
Andersson, Christensen, Marker, Thomsen: Changes in cell adhesion molecule expression on T cells associated with systemic virus infection. in Journal of immunology (Baltimore, Md. : 1950) 1994
Show all 4 Pubmed References
this study reveals a key role of IL-10 in controlling cellular metabolism via inhibiting mTORC1, and this metabolic control by IL-10 is critical to control of inflammation.
This review summarizes the paradoxic effects of IL-10 on different types of immune responses, and proposes that different cellular sources of IL-10, in particular IL-10-secreting helper and regulatory T-cells, have different effects on B-cell and CTL responses. Based on this concept we discuss the rationales for targeting the IL-10 pathway in immune-mediated diseases and cancer.
Meta-analysis provides evidence that IL-10 gene rs1800896 polymorphism is associated with the risk of acute pancreatitis.
We believe that current findings derived from human and mouse experiments will promote the development of new drugs and therapies based on IL-10 modulation, which may enhance host immunity and bacterial clearance during infection. However, because IL-10 can play both favorable and unfavorable effects over the host depending on the bacterial infection, it may act as a double edge sword.
IL-10 -592 A/C polymorphism is associated with cognitive dysfunction in first-episode drug-naive schizophrenia.
the production of IL-10 in Th2 cells was higher in the patients with sepsis
Our observations suggested that IL-10 -592 A/C, -1082 G/A, and IFN-gamma (show IFNG Antibodies) +874 T/A polymorphisms had a strong association with susceptibility to HCV infection. However, no significant association was observed between the cytokines (IL-10 and IFN-gamma (show IFNG Antibodies)) genotypes profile and HCV-liver cirrhosis risk in the studied population, except for the high frequency of IFN-gamma (show IFNG Antibodies) +874 T allele in cirrhotic patients.
searched for the relationship between single nucleotide polymorphism in the promoter region of the CD209, IL-10, IL-28 and 32 base pair deletion in CCR5 coding region (Delta 32) with the human predisposition to development of various clinical presentations of tick-borne encephalitis
studies demonstrated that ovarian cancer cells isolated from patients with type II tumors released high levels of immunosuppressive cytokines (i.e., interleukin 10 and transforming growth factor beta) and HspA1A (show HSPA1A Antibodies) in vitro.
The present study reveals that TLR4 (show TLR4 Antibodies) drives the production of TNFalpha (show TNF Antibodies) and IL-10 induced by L. braziliensis amastigotes and that the parasites decrease TLR4 (show TLR4 Antibodies) expression on monocyte surface.
The activation of STAT3 (show STAT3 Antibodies) was much higher in Gal12 (show LGALS12 Antibodies)(-/-) macrophages activated by lipopolysaccharide, which was correlated with higher levels of IL-10. Adipocytes showed higher insulin (show INS Antibodies) sensitivity when treated with Gal12 (show LGALS12 Antibodies)(-/-) macrophage-conditioned media than those treated with Gal12 (show LGALS12 Antibodies)(+/+) macrophages.
IL-10-MSCs offered superior protection against LPS (show TLR4 Antibodies)-induced ALI.
methane-rich saline may activate the PI3K-AKT (show AKT1 Antibodies)-GSK-3beta (show GSK3b Antibodies) pathway to induce IL-10 expression and produce anti-inflammatory effects via the NF-kappaB (show NFKB1 Antibodies) and MAPK (show MAPK1 Antibodies) pathways. The findings provide a new pharmacological strategy for management of inflammatory response after acute liver injury.
plasma adiponectin and leptin (show LEP Antibodies) were also decreased in IL 10tm.These findings suggest that frailty observed in this mouse model of chronic inflammation may in part be driven by alterations in fat mass, hormone secretion and energy metabolism
IL-12p35 suppresses lymphocyte proliferation, induces expansion of IL-10-expressing and IL-35-expressing B cells and ameliorates autoimmune uveitis.
aerobic interval training enhanced the anti-inflammatory indices IL-10/TNF-alpha (show TNF Antibodies) ratio and IL-15 (show IL15 Antibodies) expression in skeletal muscle in tumor-bearing mice.
Both IL-6 (show IL6 Antibodies) protein production and transcript levels were downregulated by RA in respiratory tract epithelial cells (LETs) , but upregulated in macrophages (MACs). RA also increased transcript levels of MCP-1 (show CPT1B Antibodies), GMCSF (show CSF2 Antibodies), and IL-10 in MACs, but not in LETs. Conversely, when LETs, but not MACs, were exposed to RA
DnaK was able to induce TGF-beta (show TGFB1 Antibodies) mRNA in treated macrophages in an IL-10 dependent manner.
This study seems to indicate that PGE2 in cattle does not produce an anti-inflammatory effect by increasing the synthesis of IL-10.
IL-10 mRNA expression increased on day 8 in the mononuclear leukocytes of pregnant cows. In a cell culture experiment, interferon-tau stimulated stimulated IL-10 mRNA expression in mononuclear leukocytes.
Bovine IL-10 potently inhibits the activation of human myeloid cells in response to toll (show TLR4 Antibodies) like receptor activation.
Monocytes obtained from cows with subclinical infection with MAP had upregulated expression of IL-10 and SOCS-3 (show SOCS3 Antibodies), which may have attenuated the capacity of mononuclear phagocytes to initiate inflammatory and adaptive immune responses.
The ability of an EHV-1 isolate to down-regulate IL-10 production might contribute to increased local inflammation and a higher risk for neurological manifestation of the disease after infection with Ab4.
The data suggested in foals there is an impaired Th2 response, the immune response is Th1 (show TH1L Antibodies) biased, interferon-gamma (show IFNG Antibodies) production is qualitatively similar to adult horses, and regulatory IL-10 production by T cells is mature.
serum IL-6 (show IL6 Antibodies):IL-10 ratio is likely to provide a valuable prognosticator for neonatal septicemia
The contribution of bronchial epithelium to airway inflammation, with focus on mRNA and protein expression of IL-6 (show IL6 Antibodies), IL-10 and TNF-alpha (show TNF Antibodies), in horses with recurrent airway obstruction during exacerbation and in remission is reported.
The complete open reading frame consists of 537 base pairs which encodes a protein of 179 amino acids. This cDNA sequence exhibited 87% homology with human IL-10.
ICAM1 (show ICAM1 Antibodies) and IL10 were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 (show JUN Antibodies) may be responsible for this upregulation.
The PRRSV vaccination induced higher levels of IL-10 expression in the first week and this may be why the CSFV vaccination is immunosuppressed.
These data indicate that the type 2 porcine reproductive and respiratory syndrome virus N protein plays an important role in IL-10 induction and the N-N non-covalent domain is associated with this activity.
The effects of semen, spermatozoa in extender, or extender alone on the expression of TGF-beta1 (show TGFB1 Antibodies), IL-10, and IL-6 (show IL6 Antibodies) in ovarian follicles are reported.
Prostaglandin E2 potentiates mesenchymal stem cell-induced IL-10+IFN-gamma (show IFNG Antibodies)+CD4 (show CD4 Antibodies)+ regulatory T cells to control transplant arteriosclerosis.
Porcine reproductive and respiratory syndrome virus strain CH-1a could significantly up-regulate IL-10 production through p38 MAPK (show MAPK14 Antibodies) activation.
Boar seminal plasma contained TGF- beta1 (show TGFB1 Antibodies) and IL-10 but with high individual variation.
Changes in interleukin-10 mRNA expression are predictive for 9-day survival of pigs in an emergency preservation and resuscitation model.
These results suggest that TNF-alpha (show TNF Antibodies) and IL-10, but not IL-6 (show IL6 Antibodies), are involved in the late reparatory phases of the experimental disk lesion.
The present study shows that elevated levels of serum CRP (show CRP Antibodies) and IL-10 were associated with porcine circovirus type 2-infected piglets that subsequently developed severe PMWS.(postweaning multisystemic wasting syndrome)
Subclinically porcine circovirus type 2 (PCV2)-infected pigs developed a transient PCV2-specific IL-10 response during the viremic phase of infection which coincided with the inversion of the IgM/IgG ratio.
IL-10 produced during the immune response to malaria in this model contributes to suppression of mucosal inflammatory responses to invasive NTS (show NTS Antibodies), which may contribute to differences in the clinical presentation of NTS (show NTS Antibodies) infection in the setting of malaria.
Findings demonstrate that internalization of IL-10R with the resultant impact on IL-10 signaling and dysregulation of the IL-10-mediated anti-inflammatory responses might play a crucial role in epithelial cell damage and subsequent simian immunodeficiency virus pathogenesis.
IL-10 plays a crucial role in maintaining mucosal homeostasis by regulating mucosal IFNgamma and TNFalpha (show TNF Antibodies) cytokine production.
The protein encoded by this gene is a cytokine produced primarily by monocytes and to a lesser extent by lymphocytes. This cytokine has pleiotropic effects in immunoregulation and inflammation. It down-regulates the expression of Th1 cytokines, MHC class II Ags, and costimulatory molecules on macrophages. It also enhances B cell survival, proliferation, and antibody production. This cytokine can block NF-kappa B activity, and is involved in the regulation of the JAK-STAT signaling pathway. Knockout studies in mice suggested the function of this cytokine as an essential immunoregulator in the intestinal tract. Mutations in this gene are associated with an increased susceptibility to HIV-1 infection and rheumatoid arthritis.
T-cell growth inhibitory factor
, cytokine synthesis inhibitory factor
, uncharacterized protein LOC100715618
, Cytokine synthesis inhibitory factor