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hindbrain-repressive Wnt3a/Meis3/Tsh1 circuit promotes neuronal differentiation and coordinates tissue maturation
Synergy between XMeis3 and Hoxd1 in Hoxd1 autoregulation in mesoderm during gastrulation, is shown.
XMeis3 protein knock down also causes a loss of primary neuron and neural crest cell lineages, without altering expression of Zic, Sox or Pax3 genes.
XMeis3 activates FGF/MAP-kinase signaling, which then modulates the PCP pathway.
XMeis3 protein appears crucial for initial hindbrain induction, whereas Retinoic Acid signaling defines the spatial limits of hindbrain gene expression by modifying XMeis3 protein activity
Findings support a model in which Meis3 is required for neural crest proliferation, migration into, and colonization of the gut such that its loss leads to severe defects in enteric nervous system development.
This work suggests that the transcriptional activity of all members of the Meis/Prep Hth protein family is subject to autoinhibition by their Hth domains, and that the Meis3.2 splice variant encodes a protein that bypasses this autoinhibitory effect.
Data have demonstrated the presence of Meis, Pbx, and Prep proteins during early lineage segregation in mammals: Meis1, Meis3, Pbx1, Pbx2, Pbx3, and Prep1 in the mouse.
Meis3 is abundantly expressed in pancreatic islets and beta-cells and regulates beta-cell survival through 3-phosphoinositide-dependent protein kinase 1 (PDK1).
The mRNA level of this protein was elevated in limb buds after treatment with ectopic acid.
Novel collaboration between Icsbp deficiency and Meis1/Meis3 in the acceleration of chronic myeloid leukemia-like disease.
Meis3 and pbx4 regulate shh expression in anterior endoderm, thereby influencing patterning and growth of the foregut.
Caudalizing protein which is required to pattern the anterior/posterior (A/P) axis during central nervous system (CNS) formation. Inhibits anterior neural expression and acts as a transcriptional activator to induce posterior neural gene expression. Maintains a proper A/P balance required for hindbrain formation by activating the FGF/MAPK pathway, which modulates the planar cell polarity (PCP) pathway. Interacts with retinoid signaling during hindbrain patterning.
homeobox protein meis3-B
, Meis1, myeloid ecotropic viral integration site 1 homolog
, homeobox protein meis3
, Meis homeobox 3
, Meis homeobox 3 pseudogene 1
, Meis1, myeloid ecotropic viral integration site 1 homolog 3
, homeobox protein Meis3
, meis1-related protein 2
, Meis3 homeoprotein
, homeobox protein meis3-A
, myeloid ecotropic viral integration site-related gene 2