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RBMX may control SREBP-1c expression in mouse liver in response to high-fructose diet.
These results indicate a major role for SAFB1 in the activation of Srebp-1c through its interaction with RBMX.
Study demonstrates a novel role for the splice factor hnRNPG in the pregnant myometrium. hnRNPG is required for the exclusion of exon 7 in the derivation of the dominant negative myometrial ERDelta7 isoform and is negatively regulated in an E2 dependent manner.
These results suggest that satellite I RNA plays a role in stabilizing RBMX and Sororin in the ncRNP complex to maintain proper sister chromatid cohesion.
HNRNPG binds m6A-methylated RNAs through its C-terminal low-complexity region, which self-assembles into large particles in vitro. The Arg-Gly-Gly repeats within the low-complexity region are required for binding to the RNA motif exposed by m6A methylation.
A sequence deletion within RBMX is identified as associated with with Shashi X-linked intellectual disability syndrome.
Host RBMX is required for the maintenance of Borna disease virus nuclear viral factories.
Results showed that HNRNPG and HTRA2-BETA1 were specific antagonistic regulators of ERa exon7 splicing and increased HNRNPG levels were associated with improved clinical outcome of endometrial cancer through up-regulation of ERaD7 expression.
RBMX is a cohesion regulator that maintains the proper cohesion of sister chromatids.
Data show that RBMX accumulated at DNA lesions through multiple domains in a poly(ADP-ribose) polymerase 1-dependent manner and promoted HR by facilitating proper BRCA2 expression.
studied the genetic and expression states of hnRNP G in normal, premalignant and malignant human oral tissues to further understand the relationship between the hnRNP G alterations and the development of human oral cancer
evidence provided that deletions in or around RBMX may be involved in non-obstructive azoospermia(NOA)
From these results, it seems that the X-chromosome, through its RBM genes, plays a formerly unknown role in the regulation of programmed cell death (apoptosis) in breast cancer.
p53 modulates DNA DSB repair by, in part, inducing hnRNP G
This identifies RBMX as an ARTS-1-associated protein that regulates both the constitutive release of TNFR1 exosome-like vesicles and the inducible proteolytic cleavage of TNFR1 ectodomains.
These studies indicate that hnRNP G promotes the expression of Txnip and mediates its tumor-suppressive effect.
rbmx is required for normal embryonic development, in particular of the brain, consistent with a role in X-linked mental retardation
This gene belongs to the RBMY gene family which includes candidate Y chromosome spermatogenesis genes. This gene, an active X chromosome homolog of the Y chromosome RBMY gene, is widely expressed whereas the RBMY gene evolved a male-specific function in spermatogenesis. Pseudogenes of this gene, found on chromosomes 1, 4, 9, 11, and 6, were likely derived by retrotransposition from the original gene. Alternatively spliced transcript variants encoding different isoforms have been identified. A snoRNA gene (SNORD61) is found in one of its introns.
RNA-binding motif protein, X chromosome
, heterogeneous nuclear ribonucleoprotein G
, RNA binding motif protein, X chromosome
, glycoprotein p43
, hnRNP G
, RNA-binding motif protein, X chromosome retrogene
, Heterogeneous nuclear ribonucleoprotein G
, RNA binding motif protein, X-linked
, RNA binding motif protein, X-linked-like 1