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anti-Rat (Rattus) GTF2H3 Antibodies:
anti-Mouse (Murine) GTF2H3 Antibodies:
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extensive molecular analyses unveiled that wild-type TFIIH (show GTF2H4 Antibodies) cooperated in an ATP-dependent manner with PGC1-alpha as well as with the deacetylase SIRT1 (show SIRT1 Antibodies), thereby contributing to the PGC1-a deacetylation by SIRT1 (show SIRT1 Antibodies)
Mutations in TFIIH (show GTF2H4 Antibodies) causing trichothiodystrophy are responsible for defects in ribosomal RNA production and processing.
Data show that that B2 RNA, when present with Pol II (show Pol II Antibodies) in promoter-bound complexes, specifically represses CTD phosphorylation by TFIIH (show GTF2H4 Antibodies).
Molecular analyses performed on the mice brain tissue demonstrate that TFIIH (show GTF2H4 Antibodies) is required for the stabilization of thyroid hormone (show PTH Antibodies) receptors (TR) to their DNA-responsive elements.
interface variants between the p34 (show CCNH Antibodies) and p44 (show GTF2H2 Antibodies) subunits only mildly affected the association between the full length proteins and did not impinge on TFIIH (show GTF2H1 Antibodies) activities due to the presence of an additional interface involving the C4 domain of p34 (show CCNH Antibodies).
This gene encodes a member of the TFB4 family. The encoded protein is a subunit of the core-TFIIH basal transcription factor and localizes to the nucleus. The encoded protein is involved in RNA transcription by RNA polymerase II and nucleotide excision repair and associates with the Cdk-activating kinase complex. Alternative splicing results in multiple transcript variants. A related pseudogene has been identified on chromosome 14.
general transcription factor IIH subunit 3
, TFIIH basal transcription factor complex p34 subunit
, basic transcription factor 2 34 kDa subunit
, general transcription factor IIH, polypeptide 3, 34kDa
, general transcription factor IIH polypeptide 3