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Human MCM5 Protein expressed in Wheat germ - ABIN1310557
Henderson, Hall, Prpic, Hessling, Parker, Sampson, Simkins, Brough, Dixon, Lenz, Knapp, Murphy, Taylor, Fischer, Malinowski: The selection and characterization of antibodies to minichromosome maintenance proteins that highlight cervical dysplasia. in Journal of immunological methods 2011
the decrease in recombination observed in females homozygous for mcm5(A7) is not due to a failure to create or repair meiotically induced double strand breaks (DSBs), but rather to a failure to resolve those DSBs into meiotic crossovers
different tissues may employ distinct cellular programs in responding to the depletion of MCM5
MCM5 expression is associated with the malignant status and poor prognosis in cervical adenocarcinoma patients.
The RAS-related nuclear protein (show RAN Proteins) ((P) ran), breast cancer metastasis suppressor 1 (show BRMS1 Proteins) ((P) brms1 (show BRMS1 Proteins)) and minichromosome maintenance complex component 5 ((P) mcm5) promoters have the specificity and strength needed for cancer-specific expression-targeted gene therapy.
reveal the Minichromosome Maintenance Complex to be a critical and directly regulated node within the miR (show MLXIP Proteins)-183 signaling network in MYCN (show MYCN Proteins)-amplified neuroblastoma (show ARHGEF16 Proteins) cells. Randomly selected MCMs 3 and 5 were experimentally confirmed as direct targets of miR (show MLXIP Proteins)-183.
the incorporation of physiological levels of MCM5 into HIV-1 virions facilitates viral cDNA accumulation in newly infected cells, probably by modifying nucleic acid configuration during reverse transcription.
MCM5 is a novel gene involved in Meier-Gorlin syndrome.
Our findings support the hypothesis that MCM5 targeting may be regarded as an effective molecular therapy against anaplastic thyroid carcinoma
we could consider miRNA-10b and MCM5 mRNA as prognostic markers and potential therapeutic targets in breast cancer to be applied to other patient data sets.
The increased expression of MCM5 protein begins at the oral pre-cancerous stage and is significantly associated with the aggressive progression and poor prognosis of OSCC.
Mcm2-7 (show MCM2 Proteins) loads onto origins during initiation as a double hexamer, yet does not act as a double-stranded DNA pump during elongation.
Data indicate that MCM-BP (show MCMBP Proteins) binds USP7 (show USP7 Proteins) on chromatin and can mediate an interaction between the USP7 (show USP7 Proteins) and MCM proteins.
SOX10 (show SOX10 Proteins) directly activates MCM5 transcription by binding to conserved SOX10 (show SOX10 Proteins) consensus DNA sequences in the MCM5 promoter.
The protein encoded by this gene is structurally very similar to the CDC46 protein from S. cerevisiae, a protein involved in the initiation of DNA replication. The encoded protein is a member of the MCM family of chromatin-binding proteins and can interact with at least two other members of this family. The encoded protein is upregulated in the transition from the G0 to G1/S phase of the cell cycle and may actively participate in cell cycle regulation.
, mini-chromosome maintenance 5
, minichromosome maintenance 5
, MCM5 minichromosome maintenance deficient 5, cell division cycle 46
, DNA replication licensing factor mcm5
, DNA replication licensing factor MCM5
, CDC46 homolog
, minichromosome maintenance deficient 5 (cell division cycle 46)
, minichromosome maintenance deficient protein 5
, CDC46 homolog A
, DNA replication licensing factor mcm5-A
, minichromosome maintenance deficient 5, cell division cycle 46
, mini chromosome maintenance deficient 5