Browse our anti-BNP (BNP) Antibodies

Pig (Porcine) Brain Natriuretic Peptide (BNP) interaction partners

1. CNP and BNP (show BNC2 Antibodies) serve as oocyte maturation inhibitor to inhibit oocyte maturation in mammals

2. ANP (show NPPA Antibodies) and BNP (show BNC2 Antibodies) gene expression differs considerably between cardiac chambers in the first 72 hours of life in healthy piglets, resembling the transition from fetal to neonate circulation. The cardiac gene expression does not explain plasma concentrations.

3. Up-regulation of BNP (show BNC2 Antibodies) and a reduction in a ratio of sarcoplasmic reticulum calcium ATPase (show CA-P60A Antibodies) 2a in failing myocardium are observed in a porcine model of tachycardia.

4. Data show that all five molecules, BNP (show BNC2 Antibodies), ICAM-1 (show ICAM1 Antibodies), TNF-alpha (show TNF Antibodies), VCAM-1 (show VCAM1 Antibodies) and IL-6 (show IL6 Antibodies), quickly and reliably signaled adverse interactions.

5. identification and characterization of a novel alternatively spliced variant of porcine and human NPPB resulting from exon 2 skipping

6. Arial natriuretic factor (ANF (show HESX1 Antibodies)) and N-terminal proANF may be better suited than brain natriuretic peptide as markers of cardiac preload during the development and treatment of acute heart failure.

7. Regional differences in BNP (show BNC2 Antibodies) and CNP expression were observed in myocardium with sustained left ventricular dyssynchronous contraction.

Mouse (Murine) Brain Natriuretic Peptide (BNP) interaction partners

1. These results suggested that the papillary tip express a substance that can stimulate BNP (show BNC2 Antibodies) production and secretion from cardiomyocytes.

2. This study provides the first evidence of the ability of IL-18 (show IL18 Antibodies) to induce B-type natriuretic peptide synthesis in vitro and outlines the relationship between the two molecules in acute HF patients with an ongoing inflammatory status.

3. The present study provides a novel insight into the potential underlying mechanisms of telmisartan-induced inhibition of cardiomyocyte hypertrophy, which involves inhibition of NFAT (show NFATC1 Antibodies) activation, nuclear translocation and the ANP (show NPPA Antibodies)/BNP (show BNC2 Antibodies) cascade.

4. Data suggest that the negative inhibition of P2X3R activity by the BNP (show BNC2 Antibodies)/NPR-A (show NPR1 Antibodies) pathway results in a decreased P2X3R-mediated excitability of trigeminal neurons in wildetype cultures. In familial hemiplegic migraine type-1 model cultures, however, lack of efficient P2X3Rs downregulation contributes to the neuronal hyperexcitability phenotype.

5. we have validated a novel gene, Myh14, as a negative regulator of ISO-induced left ventricular mass hypertrophy in an in vivo mouse model and demonstrated the up-regulation of immediate early (show JUN Antibodies) gene Myc (show MYC Antibodies), fetal gene Nppb, and fibrosis gene Lgals3 (show LGALS3 Antibodies) in ISO-treated Myh14 deficient hearts compared to controls.

6. P2X3 (show P2RX3 Antibodies) receptors on mouse trigeminal ganglion neurons are subjected to contrasting modulation by inhibitory brain natriuretic peptide and facilitatory calcitonin gene-related peptide (show CALCA Antibodies) that both operate via complex intracellular signaling.

7. mouse trigeminal neurons endogenous BNP (show BNC2 Antibodies) acts on NPR-A (show NPR1 Antibodies) receptors to determine constitutive depression of P2X3 (show P2RX3 Antibodies) receptor function. Tonic inhibition of P2X3 (show P2RX3 Antibodies) receptor activity by BNP (show BNC2 Antibodies)/NPR-A (show NPR1 Antibodies)/PKG (show PRKG1 Antibodies) pathways occurs via two distinct mechanisms: P2X3 (show P2RX3 Antibodies) serine phosphorylation and receptor redistribution to non-raft membrane compartments

8. The findings demonstrated a substantial role of mitochondrial dysfunction in mediating the downregulation of NKCC2 (show SLC12A1 Antibodies) and ENaCalpha (show SCNN1A Antibodies) in obstructive kidney disease, possibly via iNOS (show NOS2 Antibodies)-derived nitric oxide and BNP (show BNC2 Antibodies).

9. Data (including data from studies in knockout mice) suggest expression of ANP (show NPPA Antibodies)/BNP (show BNC2 Antibodies)/GC-A (show GUCA1A Antibodies) (atrial natriuretic peptide (show NPPA Antibodies), brain natriuretic peptide, and guanylyl cyclase-A (show NPR1 Antibodies)) is important in embryonic neovascularization and organogenesis.

10. Goalpha plays a role in ANF (show HESX1 Antibodies) sorting during intracellular vectorial transport and with the presence of a mechanism that preserves the molar relationship between cellular ANF (show HESX1 Antibodies) and BNP (show BNC2 Antibodies).