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Rat (Rattus) RHOA ELISA Kit for Sandwich ELISA - ABIN434358
Du, Wang, Wang: Role of RhoA/MERK1/ERK1/2/iNOS signaling in ocular ischemic syndrome. in Graefe's archive for clinical and experimental ophthalmology 2016
RhoA expression patterns in circulating leucocytes is a biomarker for the breast cancer risk assessment.
Receptor tyrosine kinase (show RET ELISA Kits) activation of RhoA is mediated by AKT (show AKT1 ELISA Kits) phosphorylation of DLC1 (show DYNLL1 ELISA Kits).
DOCK7 (show DOCK7 ELISA Kits) controls neuronal growth via a Rac (show AKT1 ELISA Kits)-dependent pathway, likely by modulating microtubule networks while also regulating F-actin remodeling at the cell rear to promote somal translocation via a previously unrecognized myosin phosphatase-RhoA-interacting protein-dependent pathway.
The ability of enhanced GNA13 signaling to suppress KLK gene expression appears at least in part due to the ability of enhanced GNA13 signaling to negatively impact Rho/ROCK-signaling.
OB-Rb (show LEPR ELISA Kits), RhoA/ROCK, PI3K (show PIK3CA ELISA Kits)/AKT (show AKT1 ELISA Kits), JAK (show JAK3 ELISA Kits)/STAT (show STAT1 ELISA Kits) pathways and NF-kB activation are involved in leptin (show LEP ELISA Kits)-induced upA (show PRAP1 ELISA Kits) expression.
study strongly supported the contribution of the genes ITGA2B, GSN and RHOA and the two pathways "regulation of actin cytoskeleton" and "leukocyte transendothelial migration" to osteoporosis risk.
Suggest RHOA mutations useful for diagnosing cutaneous localizations of angioimmunoblastic T-cell lymphomas.
TET2 (show TET2 ELISA Kits) and RhoA mutations cooperatively disrupt T cell homeostasis
Genetic variant in RhoA gene is associated with progression of prostate cancer.
Downregulation of Cul3 (show CUL3 ELISA Kits) led to a marked increase in RhoA protein expression after 6 days of adipocytes differentiation, suggesting that Cul3 (show CUL3 ELISA Kits) is involved in the regulation of RhoA stability.
Hydrogen peroxide oxidizes RhoA at Cys16 and Cys20, and activates RhoA via Vav2 (show VAV2 ELISA Kits).
These results reveal a novel signaling network, the Sema4D (show SEMA4D ELISA Kits)-RhoA-Akt (show AKT1 ELISA Kits) signal cascade, that coordinates cellular function and morphology and highlights the importance of specific spatiotemporally restricted components of a signaling pathway in the regulation of ameloblast differentiation.
RhoA deficiency could disrupt podocyte cytoskeleton and induce podocyte apoptosis by inhibiting YAP/dendrin signal.
These results suggested that, in addition to inhibiting Noggin (show NOG ELISA Kits) transcription, RhoA activity in wild-type murine embryonic stem cells also prevented neural differentiation by limiting Noggin (show NOG ELISA Kits) secretion.
Rho attenuates the interaction between Amot (show AMOT ELISA Kits) and Nf2 (show NF2 ELISA Kits) by binding to the coiled-coil domain of Amot (show AMOT ELISA Kits).
we uncovered cell state plasticity and adhesion dynamics regulated by Ror2 (show ROR2 ELISA Kits), which influenced Ras Homology Family Member A (show CXCL14 ELISA Kits) (RhoA) and Rho-Associated Coiled-Coil Kinase 1 (ROCK1 (show ROCK1 ELISA Kits)) activity downstream of Dishevelled-2 (Dvl2 (show DVL2 ELISA Kits)).
Active Rho-kinase (show ROCK2 ELISA Kits) diffuses to growing other immature neurites and inhibits their outgrowth to ensure single axon formation.
Data indicate that oxidative stress in diabetes causes a decrease in miR (show MLXIP ELISA Kits)-133a expression leading to an increase in RhoA/Rho kinase (show ROCK2 ELISA Kits) pathway and muscle contraction.
Impaired denitrosylation is associated with detrusor overactivity, which is linked with upregulated RhoA/Rho-kinase (show ROCK2 ELISA Kits) signalling
Mgc's GAP activity down-regulates the active populations of RhoA and Rac1 at localized regions of epithelial cells and is necessary for successful cytokinesis and cell-cell junction structure
Data show that shortly after anaphase onset oocytes and embryonic cells exhibit cortical waves of Rho activity and F-actin polymerization.
CASZ1 (show CASZ1 ELISA Kits)/Egfl7 (show EGFL7 ELISA Kits)/RhoA pathway is necessary for promoting endothelial cell behaviors associated with proper vascular assembly.
RhoA can be considered a component of the intracellular pattern formation system.
Kazrin (show KAZ ELISA Kits) interacts with ARVCF (show ARVCF ELISA Kits)-catenin, spectrin and p190B (show ARHGAP5 ELISA Kits) RhoGAP (show ARHGAP1 ELISA Kits), and modulates RhoA activity.
Morphogenesis of the primitive gut tube is generated by Rho/ROCK/myosin II-mediated endoderm rearrangements.
RhoA and membrane fluidity mediates the spatially polarized Src (show SRC ELISA Kits)/FAK (show PTK2 ELISA Kits) activation in response to shear stress.
the Lbc (show AKAP13 ELISA Kits)/alpha-catulin (show CTNNAL1 ELISA Kits) axis participates in 5-HT (show DDC ELISA Kits)-induced PASMC mitogenesis and RhoA/ROCK signaling, and may be an interventional target in diseases involving vascular smooth muscle remodeling.
The RhoA/ROCK signaling pathway is an important negative regulator of vascular calcification.
Vascular endothelial-cadherin signals through RhoA and actin cytoskeletal and affects cell-matrix adhesion
Thrombospondin has a role in inducing RhoA inactivation through FAK (show PTK2 ELISA Kits)-dependent signaling to stimulate focal adhesion disassembly
KCl directly increased Rho and ROCK activities in a concentration-dependent fashion that paralleled closely the effect of KCl on lung smooth muscle tone and [Ca(2 (show CA2 ELISA Kits)+)](i), as well as the voltage-dependent Ca(2 (show CA2 ELISA Kits)+) currents
the Rho-ROCK signal pathway contributes to VEGF-induced hyperpermeability. Myosin light-chain phosphorylation and actin stress fiber formation occur concomitantly with the increase in permeability upon VEGF stimulation.
Formation of polygonal actin network in endothelial cells is a novel rhoA associated response to hypertonic stress.
Cadherins, RhoA and Rac1, have important roles in mechanotransduction and that endothelial and smooth muscle cells use different mechanisms to respond to stretch.
Results indicate that hypergravity induces ATP release and actin reorganization via RhoA activation and FAK (show PTK2 ELISA Kits) phosphorylation, thereby activating cell proliferation and migration in bovine aortic endothelial cells.
Pseudorabies virus US3 expression led to RhoA phosphorylation at serine 188 to induce actin rearrangements.
Data indicate that TNF-alpha (show TNF ELISA Kits) stimulates Rac (show AKT1 ELISA Kits), ADAM17/TACE (show ADAM17 ELISA Kits), and RhoA through the guanine nucleotide exchange factor (show ARHGEF12 ELISA Kits) (GEF)-H1 (show ARHGEF2 ELISA Kits).
Contractile pulmonary arterial myocytes exhibit marked Rho-dependent actin polymerization in hypoxia, with increased active RhoA and LIMK (show LIMK1 ELISA Kits) phosphorylation.
Results suggest that Rac1 and RhoA are regulated by TGFbeta1 (show TGFB1 ELISA Kits) in the process of endothelial tube formation in collagen I gels.
The concentration of RhoA mRNA and activated RhoA enzyme were greater in urothelium than in detrusor. Rho kinase (show ROCK1 ELISA Kits) inhibitor Y-27632 showed a stronger inhibitory effect in detrusor with intact urothelium.
Thrombin (show F2 ELISA Kits) stimulates swine smooth muscle cell differentiation from peripheral blood mononuclear cells via protease-activated receptor-1 (show F2R ELISA Kits), RhoA, and myocardin (show MYOCD ELISA Kits).
Activating Rho could be beneficial to suppress Kras mutant-induced liver malignancies.
Regulates a signal transduction pathway linking plasma membrane receptors to the assembly of focal adhesions and actin stress fibers. Involved in a microtubule-dependent signal that is required for the myosin contractile ring formation during cell cycle cytokinesis. Plays an essential role in cleavage furrow formation. Required for the apical junction formation of keratinocyte cell-cell adhesion. Serves as a target for the yopT cysteine peptidase from Yersinia pestis, vector of the plague, and Yersinia pseudotuberculosis, which causes gastrointestinal disorders. Stimulates PKN2 kinase activity. May be an activator of PLCE1. Activated by ARHGEF2, which promotes the exchange of GDP for GTP. Essential for the SPATA13-mediated regulation of cell migration and adhesion assembly and disassembly. The MEMO1-RHOA- DIAPH1 signaling pathway plays an important role in ERBB2- dependent stabilization of microtubules at the cell cortex. It controls the localization of APC and CLASP2 to the cell membrane, via the regulation of GSK3B activity. In turn, membrane-bound APC allows the localization of the MACF1 to the cell membrane, which is required for microtubule capture and stabilization.
Aplysia ras-related homolog 12
, oncogene RHO H12
, ras homolog gene family, member A
, rho cDNA clone 12
, small GTP binding protein RhoA
, transforming protein RhoA
, Ras family member A
, Rho family GTPase
, aplysia ras-related homolog A
, aplysia ras-related homolog A1
, aplysia ras-related homolog A2
, ras homolog A1
, ras homolog A2
, ras homolog gene family, member A1
, ras homolog gene family, member A2
, plysia ras-related homolog A2
, rho1 GTP-binding protein
, RhoA GTPase
, Rho A