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anti-Human TWIST1 Antibodies:
anti-Mouse (Murine) TWIST1 Antibodies:
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Human Monoclonal TWIST1 Primary Antibody for ICC, IF - ABIN2668516
Ezponda, Popovic, Shah, Martinez-Garcia, Zheng, Min, Will, Neri, Kelleher, Yu, Licht: The histone methyltransferase MMSET/WHSC1 activates TWIST1 to promote an epithelial-mesenchymal transition and invasive properties of prostate cancer. in Oncogene 2013
Show all 5 Pubmed References
Human Polyclonal TWIST1 Primary Antibody for ELISA, ICC - ABIN6267722
Yang, Lian, Sun, Qi, Ding, Zhang: High nuclear expression of Twist1 in the skeletal extramedullary disease of myeloma patients predicts inferior survival. in Pathology, research and practice 2016
Show all 4 Pubmed References
Human Monoclonal TWIST1 Primary Antibody for IP, RNAi - ABIN563296
Saito, Murata-Kamiya, Hirayama, Ohba, Hatakeyama: Conversion of Helicobacter pylori CagA from senescence inducer to oncogenic driver through polarity-dependent regulation of p21. in The Journal of experimental medicine 2010
Show all 3 Pubmed References
Human Monoclonal TWIST1 Primary Antibody for ELISA, WB - ABIN1098133
Yu, Li, Lu, Tian, Ma, Wang, Xu: Down-regulation of TWIST decreases migration and invasion of laryngeal carcinoma Hep-2 cells by regulating the E-cadherin, N-cadherin expression. in Journal of cancer research and clinical oncology 2011
Show all 2 Pubmed References
Human Monoclonal TWIST1 Primary Antibody for IP, ELISA - ABIN521172
Cosset, Hamdan, Jeanpierre, Voeltzel, Sagorny, Hayette, Mahon, Dumontet, Puisieux, Nicolini, Maguer-Satta: Deregulation of TWIST-1 in the CD34+ compartment represents a novel prognostic factor in chronic myeloid leukemia. in Blood 2011
Human Monoclonal TWIST1 Primary Antibody for ICC, FACS - ABIN1724845
Weiss, Abel, Mayberry, Basile, Berger, Aplin: TWIST1 is an ERK1/2 effector that promotes invasion and regulates MMP-1 expression in human melanoma cells. in Cancer research 2012
Cow (Bovine) Polyclonal TWIST1 Primary Antibody for IHC, WB - ABIN2780347
Reinhold, Kapadia, Liao, Naski: The Wnt-inducible transcription factor Twist1 inhibits chondrogenesis. in The Journal of biological chemistry 2006
Show all 2 Pubmed References
Human Polyclonal TWIST1 Primary Antibody for CyTOF, FACS - ABIN4898987
Satelli, Mitra, Brownlee, Xia, Bellister, Overman, Kopetz, Ellis, Meng, Li: Epithelial-mesenchymal transitioned circulating tumor cells capture for detecting tumor progression. in Clinical cancer research : an official journal of the American Association for Cancer Research 2015
Human Polyclonal TWIST1 Primary Antibody for IHC (p), IHC - ABIN189738
Jin, Ren, Macarak, Rosenbloom et al.: Pathobiological mechanisms of peritoneal adhesions: The mesenchymal transition of rat peritoneal mesothelial cells induced by TGF-β1 and IL-6 requires activation of Erk1/2 and Smad2 linker region ... in Matrix biology : journal of the International Society for Matrix Biology 2016
Frame-shift mutation in TWIST1 is associated with type 2 scurs syndrome.
Interaction with Snail1/2, and Twist function more generally, is regulated by GSK-3-beta-mediated phosphorylation of conserved sites in the WR domain.
TWIST1-miR (show MLXIP Antibodies)-214 pathway in the control of migration and invasion of lung adenocarcinoma.
the SDF1 (show CXCL12 Antibodies)/CXCR4 (show CXCR4 Antibodies) signaling pathway is involved in Lowintensity pulsed ultrasoundpromoted periodontal ligament stem cell migration.
Findings suggest that cytoplasmic, rather than nuclear expression of Twist1 can be considered as a prognostic marker especially for patients with clear cell renal cell carcinoma (show MOK Antibodies).
Chromatin immunoprecipitation (ChIP), quantitative ChIP and dual luciferase activity assays were used to confirm the binding of SOX6 to the promoter region of TWIST1.
we demonstrated a mechanistic cascade of TMPRSS4 (show TMPRSS4 Antibodies) up-regulating STAT3 (show STAT3 Antibodies) activation and subsequent TWIST1 expression, leading to prostate cancer migration.
In cancer patients, elevated levels of Twist1 are associated with greater degrees of muscle wasting.
Twist, E-cadherin (show CDH1 Antibodies), and N-cadherin (show CDH2 Antibodies) protein were differently expressed in endometrioid adenocarcinoma tissues and in normal endometrium which indicates their potential function for endometrioid adenocarcinoma development.
Three SNPs were associated with survival: rs2526614 (TWIST1) (genotype CA + AA, adjusted HR=0.58, 95%CI=0.37-0.93), rs6953766 (TWIST1) (genotype GG, crude HR=2.02, 95%CI=1.06-3.82, adjusted HR=2.14, 95%CI=1.07-4.25), and rs431073 (ZEB1 (show ZEB1 Antibodies)) (genotype AC + CC, crude HR=1.62, 95%CI=1.01-2.59, adjusted HR=1.96, 95%CI=1.18-3.25).
Study shows that molecular dynamic simulations provide a structural explanation for the loss-of-function associated with the Saethre-Chotzen syndrome TWIST1 mutation and provides a proof of concept of the predictive value of these MD simulations; MD simulations highlighted a clear decrease in the stability of the alpha-helix during the dimerization of the mutated R154P TWIST1/E12 (show ELSPBP1 Antibodies) dimer compared to the wild-type TE comp (show COMP Antibodies)...
Basic performance testing showed that the combined restriction digital PCR assay enabled detection of 0.14% of the TWIST1 methylation level for the lymphocyte DNA
Overexpression of Twist1 in mouse muscle progenitor cells, either constitutively during development or inducibly in adult animals, caused severe muscle atrophy with features reminiscent of cachexia.
This study evaluated the role of Twist1 in the expression of other epithelial-mesenchymal transition transcription factors in tumor cells, including tumor progression, intravasation, and metastasis.
The authors demonstrate that Twist1 serine (Ser (show SIGLEC1 Antibodies)) 42 phosphorylation is required for endothelial-to-mesenchymal transition through TGF-beta (show TGFB1 Antibodies)-Smad (show SMAD1 Antibodies) signaling in vitro and in the mouse lung gel implantation system.
Overall, hypoxia-induced activation of Twist/miR (show MLXIP Antibodies)-214/E-cadherin (show CDH1 Antibodies) axis is involved in the EMT (show ITK Antibodies) of TECs, and anti-miR (show MLXIP Antibodies)-214 may be an attractive strategy to ameliorate the progression of renal fibrosis.
molecular and cellular processes that regulate dural Cerebral vein development in mammals and describe venous malformations in humans with craniosynostosis and TWIST1 mutations that are recapitulated in mouse models, are reported.
Methyltransferase G9A (show EHMT2 Antibodies) Regulates Osteogenesis via Twist Gene Repression in mice.
this study shows that loss of Twist1 in collagen-producing cells leads to increased bleomycin-induced pulmonary fibrosis, which is mediated by increased expression of CXCL12 (show CXCL12 Antibodies)
RNF8 (show RNF8 Antibodies)-promoted Twist ubiquitination is required for Twist localization to the nucleus for subsequent epithelial-mesenchymal transition and cancer stem cells functions, thereby conferring chemoresistance.
These results indicate that Twist1 Ser42 phosphorylation contributes to the pathogenesis of bleomycin-induced pulmonary fibrosis through angiopoietin-Tie2 (show TEK Antibodies) signaling.
the mesenchymal properties of the cranial mesoderm are likely to be regulated by a network of TWIST1 targets that influences the extracellular matrix and cell-matrix interactions, and collectively they are required for the morphogenesis of the craniofacial structures.
the ventral migration of Cranial neural crest cells (CNCCs) away from a source of Bmps in the dorsal ectoderm promotes ectomesenchyme development by relieving Id2a-dependent repression of Twist1 function.
twist1a and twist1b control skeletal development and dorsoventral patterning by regulating runx2b in zebrafish
These observations are consistent with a role for twist1 in craniofacial, vertebral, and early renal development.
Basic helix-loop-helix (bHLH) transcription factors have been implicated in cell lineage determination and differentiation. The protein encoded by this gene is a bHLH transcription factor and shares similarity with another bHLH transcription factor, Dermo1. The strongest expression of this mRNA is in placental tissue\; in adults, mesodermally derived tissues express this mRNA preferentially. Mutations in this gene have been found in patients with Saethre-Chotzen syndrome.
twist homolog 1
, twist transcription factor
, twist homolog 1 (acrocephalosyndactyly 3; Saethre-Chotzen syndrome)
, hypothetical protein
, twist-like protein
, twist-related protein 1
, twist-related protein
, B-HLH DNA binding protein
, TWIST homolog of drosophila
, class A basic helix-loop-helix protein 38
, charlie chaplin
, polydactyly EMS
, twist gene homolog 1
, twist 1
, twist homolog 1 (Drosophila)