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anti-Human TWIST1 Antibodies:
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Human Monoclonal TWIST1 Primary Antibody for ICC, IF - ABIN2668516
Ezponda, Popovic, Shah, Martinez-Garcia, Zheng, Min, Will, Neri, Kelleher, Yu, Licht: The histone methyltransferase MMSET/WHSC1 activates TWIST1 to promote an epithelial-mesenchymal transition and invasive properties of prostate cancer. in Oncogene 2013
Show all 5 Pubmed References
Human Monoclonal TWIST1 Primary Antibody for IP, RNAi - ABIN563296
Saito, Murata-Kamiya, Hirayama, Ohba, Hatakeyama: Conversion of Helicobacter pylori CagA from senescence inducer to oncogenic driver through polarity-dependent regulation of p21. in The Journal of experimental medicine 2010
Show all 3 Pubmed References
Human Monoclonal TWIST1 Primary Antibody for ELISA, WB - ABIN1098133
Yu, Li, Lu, Tian, Ma, Wang, Xu: Down-regulation of TWIST decreases migration and invasion of laryngeal carcinoma Hep-2 cells by regulating the E-cadherin, N-cadherin expression. in Journal of cancer research and clinical oncology 2011
Show all 2 Pubmed References
Human Polyclonal TWIST1 Primary Antibody for IHC (p), IHC - ABIN189738
Jin, Ren, Macarak, Rosenbloom et al.: Pathobiological mechanisms of peritoneal adhesions: The mesenchymal transition of rat peritoneal mesothelial cells induced by TGF-β1 and IL-6 requires activation of Erk1/2 and Smad2 linker region ... in Matrix biology : journal of the International Society for Matrix Biology 2016
Human Monoclonal TWIST1 Primary Antibody for IP, ELISA - ABIN521172
Cosset, Hamdan, Jeanpierre, Voeltzel, Sagorny, Hayette, Mahon, Dumontet, Puisieux, Nicolini, Maguer-Satta: Deregulation of TWIST-1 in the CD34+ compartment represents a novel prognostic factor in chronic myeloid leukemia. in Blood 2011
Human Monoclonal TWIST1 Primary Antibody for ICC, FACS - ABIN1724845
Weiss, Abel, Mayberry, Basile, Berger, Aplin: TWIST1 is an ERK1/2 effector that promotes invasion and regulates MMP-1 expression in human melanoma cells. in Cancer research 2012
Human Polyclonal TWIST1 Primary Antibody for CyTOF, FACS - ABIN4898987
Satelli, Mitra, Brownlee, Xia, Bellister, Overman, Kopetz, Ellis, Meng, Li: Epithelial-mesenchymal transitioned circulating tumor cells capture for detecting tumor progression. in Clinical cancer research : an official journal of the American Association for Cancer Research 2015
Cow (Bovine) Polyclonal TWIST1 Primary Antibody for IHC, WB - ABIN2780347
Reinhold, Kapadia, Liao, Naski: The Wnt-inducible transcription factor Twist1 inhibits chondrogenesis. in The Journal of biological chemistry 2006
Show all 2 Pubmed References
Frame-shift mutation in TWIST1 is associated with type 2 scurs syndrome.
Interaction with Snail1/2, and Twist function more generally, is regulated by GSK-3-beta-mediated phosphorylation of conserved sites in the WR domain.
Tumor cell hypoxia induced Bcl-2 (show BCL2 Antibodies)/Twist1 complex formation, promoting tumor cell invasion in oral squamous cell carcinoma.
Addition of a TWIST1/NID2 (show NID2 Antibodies)-based DNA methylation (show HELLS Antibodies) assay adds diagnostic value to urine cytology and the model is sensitive to the classification of equivocal cytology.
Data show that TUSC2 (show TUSC2 Antibodies) is a direct target of miR (show MLXIP Antibodies)-584, which is transcriptionally regulated by TWIST1.
These results demonstrated that BrMC inhibits the stemness of LCSLCs originated from SMMC-7721 cell line by inhibiting STAT3 (show STAT3 Antibodies)/Twist signal axis.
Twist1 could promote the accumulation of DNA damage repair and inhibit the apoptosis of nasopharyngeal carcinoma cells, promoting radioresistance.
The mutual correlation between the expression level of TWIST1 and ID1 (show ID1 Antibodies) might be a critical factor driving the process of the human odontoblasts' differentiation.
microRNA miR (show MLXIP Antibodies)-33a suppresses IL-6 (show IL6 Antibodies)-induced tumor progression by binding Twist 3'UTR (show UTS2R Antibodies) in gallbladder cancer, preventing proliferation and migration.
TWIST1 expression is dynamically regulated during in vitro chondrogenic differentiation of human BMSCs.
Twist1 induces stem cell-like characteristics in colon cancer cell lines related to epithelial-mesenchymal transition via AKT (show AKT1 Antibodies) signaling pathways, and those pathways depend on microsatellite instability status
PDGF-D (show PDGFD Antibodies) promotes tumor growth and aggressiveness of colorectal carcinoma. Down-regulation of PDGF-D (show PDGFD Antibodies) inactivates Notch1 (show NOTCH1 Antibodies)/Twist1 axis, which could reverse epithelial mesenchymal transformation and prevent CRC (show CALR Antibodies) progression.
The authors demonstrate that Twist1 serine (Ser (show SIGLEC1 Antibodies)) 42 phosphorylation is required for endothelial-to-mesenchymal transition through TGF-beta (show TGFB1 Antibodies)-Smad (show SMAD1 Antibodies) signaling in vitro and in the mouse lung gel implantation system.
Overall, hypoxia-induced activation of Twist/miR (show MLXIP Antibodies)-214/E-cadherin (show CDH1 Antibodies) axis is involved in the EMT (show ITK Antibodies) of TECs, and anti-miR (show MLXIP Antibodies)-214 may be an attractive strategy to ameliorate the progression of renal fibrosis.
molecular and cellular processes that regulate dural Cerebral vein development in mammals and describe venous malformations in humans with craniosynostosis and TWIST1 mutations that are recapitulated in mouse models, are reported.
Methyltransferase G9A (show EHMT2 Antibodies) Regulates Osteogenesis via Twist Gene Repression in mice.
this study shows that loss of Twist1 in collagen-producing cells leads to increased bleomycin-induced pulmonary fibrosis, which is mediated by increased expression of CXCL12 (show CXCL12 Antibodies)
RNF8 (show RNF8 Antibodies)-promoted Twist ubiquitination is required for Twist localization to the nucleus for subsequent epithelial-mesenchymal transition and cancer stem cells functions, thereby conferring chemoresistance.
These results indicate that Twist1 Ser42 phosphorylation contributes to the pathogenesis of bleomycin-induced pulmonary fibrosis through angiopoietin-Tie2 (show TEK Antibodies) signaling.
the mesenchymal properties of the cranial mesoderm are likely to be regulated by a network of TWIST1 targets that influences the extracellular matrix and cell-matrix interactions, and collectively they are required for the morphogenesis of the craniofacial structures.
TWIST1 expression promotes developmental angiogenesis by inducing endothelial cell proliferation and migration.
There was a possible regulatory link between Twist 1 and PPARgamma (show PPARG Antibodies) in 3T3-L1 mature adipocytes. This regulatory link enhanced the regulation of PPARgamma (show PPARG Antibodies) and may be a functional mechanism of Twist 1 regulation of adipocyte physiology and pathology
the ventral migration of Cranial neural crest cells (CNCCs) away from a source of Bmps in the dorsal ectoderm promotes ectomesenchyme development by relieving Id2a-dependent repression of Twist1 function.
twist1a and twist1b control skeletal development and dorsoventral patterning by regulating runx2b in zebrafish
These observations are consistent with a role for twist1 in craniofacial, vertebral, and early renal development.
Basic helix-loop-helix (bHLH) transcription factors have been implicated in cell lineage determination and differentiation. The protein encoded by this gene is a bHLH transcription factor and shares similarity with another bHLH transcription factor, Dermo1. The strongest expression of this mRNA is in placental tissue\; in adults, mesodermally derived tissues express this mRNA preferentially. Mutations in this gene have been found in patients with Saethre-Chotzen syndrome.
twist homolog 1
, twist transcription factor
, twist homolog 1 (acrocephalosyndactyly 3; Saethre-Chotzen syndrome)
, hypothetical protein
, twist-like protein
, twist-related protein 1
, twist-related protein
, B-HLH DNA binding protein
, TWIST homolog of drosophila
, class A basic helix-loop-helix protein 38
, charlie chaplin
, polydactyly EMS
, twist gene homolog 1
, twist 1
, twist homolog 1 (Drosophila)